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The Surprising Role of Carbon Sixty in Reversing Aging and Boosting Mitochondria wit Ken Swartz · Hardiness with Dr Paul Taylor

• 0:09
  Speaker 1: Ken Schwartz, Welcome to the podcast.
• 0:12
  Speaker 2: Oh glad to be on all the way.
• 0:14
  Speaker 1: From Denver, Colorado, which you tell me is is has
• 0:18
  Speaker 1: had pretty nice weather over the last number of months.
• 0:21
  Speaker 3: Oh, yes, it's been. It's really nice here. We hardly
• 0:23
  Speaker 3: had a winter at all, so it's hopefully we won't
• 0:26
  Speaker 3: have a hot summer either. Yeah.
• 0:29
  Speaker 1: Yeah, you do get the extremes over there, don't you.
• 0:32
  Speaker 1: I am so Ken, you are. I think it's fair
• 0:35
  Speaker 1: to say a wellness entrepreneur, but but you haven't always
• 0:40
  Speaker 1: been a wellness entrepreneur. What the hell is a radiation
• 0:43
  Speaker 1: scientist doing as a wellness entrepreneur?
• 0:46
  Speaker 3: Well, actually, I was working on the metal oxygen fusion
• 0:48
  Speaker 3: reactor project. It was I've worked various research labs over
• 0:52
  Speaker 3: the years, and so we were doing this one. And
• 0:54
  Speaker 3: when doing your research, you know, there was a lot
• 0:56
  Speaker 3: of dead PhDs and late professors associated with this kind
• 0:59
  Speaker 3: of search. And I didn't really want my crew myself
• 1:02
  Speaker 3: to end up that way because they have families and things,
• 1:05
  Speaker 3: and and so I went looking for something to help
• 1:08
  Speaker 3: protect from gunst the radiation that you know it's gonna
• 1:11
  Speaker 3: suffusion reactor, right, And I discovered I re found this stuff.
• 1:14
  Speaker 3: Or they did a research called carbon sixty C sixty
• 1:16
  Speaker 3: for short, and it's uh and it's well, it's a
• 1:20
  Speaker 3: little a little molecule of carbon sixty CAR sixty atoms
• 1:24
  Speaker 3: shaped like a little soccer ball, strangely enough, and they
• 1:28
  Speaker 3: they did a research where they gave one center at
• 1:30
  Speaker 3: C sixty. The control group, of course, didn't get any
• 1:33
  Speaker 3: Then they hit them with a fatal dose of radiation.
• 1:35
  Speaker 3: Most of the C sixty routes live where all the
• 1:37
  Speaker 3: control routes, of course died. It's a fatal dose. And
• 1:40
  Speaker 3: so when I learned about that, I got a couple
• 1:42
  Speaker 3: of sources for my crew myself, and we're all still
• 1:44
  Speaker 3: around even though after the experiment was over. But but
• 1:49
  Speaker 3: I kept doing it because I had more energy, better
• 1:52
  Speaker 3: mental focus, just I just feeling better really taking the
• 1:56
  Speaker 3: C sixty. And then seven months after I started taking
• 1:59
  Speaker 3: and I went to see my aptometrist or my druis
• 2:02
  Speaker 3: or dry imaccurate generation had completely disappeared. You'd never actually
• 2:07
  Speaker 3: seen that happen in as many years of study.
• 2:11
  Speaker 1: So can you take a step back. You were working
• 2:14
  Speaker 1: on the nuclear fusion reactor, right, give give our audience
• 2:20
  Speaker 1: a little bit of an overview of what was going
• 2:22
  Speaker 1: on there, because I think it's a fascinating it's a
• 2:26
  Speaker 1: fascinating area for sure.
• 2:28
  Speaker 3: No metal oxygen fusion was discovered and actually discovered before
• 2:31
  Speaker 3: was born in the late nineteen fifties, and there was
• 2:34
  Speaker 3: two guys of Sala and Kushi, and basically they were
• 2:37
  Speaker 3: fusing sodium with oxygen and it's kind of they had
• 2:40
  Speaker 3: the ying yang thing going like sodiums the yenn and
• 2:44
  Speaker 3: oxygen's the yang, and then they used argon as a modifier,
• 2:49
  Speaker 3: and that's where yin and yang come together. I'm not
• 2:50
  Speaker 3: quite sure what the word for that is. In the West,
• 2:53
  Speaker 3: we call electro negativity. But they actually were able to
• 2:56
  Speaker 3: do amazing stuff and I had read about it, but
• 2:59
  Speaker 3: they got some pressed right. It was an occupied country
• 3:02
  Speaker 3: and it was threatening industry. So I was with the
• 3:05
  Speaker 3: right people at the right time, and we kind of
• 3:07
  Speaker 3: redid their experiments. But we used what we use is
• 3:09
  Speaker 3: we used heavy water because hydrogen deturium is as a
• 3:12
  Speaker 3: hydrogen isotope, it's in the fame same family as sodium, sodium,
• 3:16
  Speaker 3: lithium hydrogen. We used deturium and oxygen in an argon atmosphere,
• 3:21
  Speaker 3: and we basically.
• 3:22
  Speaker 2: Have over unity fusion. We've open sourced.
• 3:27
  Speaker 3: It and it's happening all over Taiwan and Japan and
• 3:31
  Speaker 3: China and even in Brazil that people are doing this.
• 3:34
  Speaker 3: It's not like you always heard fusionist. They're like trying
• 3:36
  Speaker 3: to fuse like hydrogen isotopes together, but metal oxygen fusion
• 3:41
  Speaker 3: is like a hundreds tens of thousands of times easier
• 3:45
  Speaker 3: to do and it's actually practical. So pretty much all
• 3:48
  Speaker 3: you need. Basically, it's our that the solution has already done.
• 3:52
  Speaker 3: Other people are taking it the next step. It's basically
• 3:54
  Speaker 3: just take water heavy water from regular water and are
• 3:58
  Speaker 3: gone from the air, and you put them in their
• 3:59
  Speaker 3: actor and you press the button in VAM. You've got
• 4:02
  Speaker 3: lots of heat and with that weild water and make electricity.
• 4:06
  Speaker 2: So essentially the carbon crisis.
• 4:08
  Speaker 3: Is over right, thank you, thank yous, from before I
• 4:13
  Speaker 3: was born.
• 4:15
  Speaker 1: So let's let's bring the conversation back to something that
• 4:20
  Speaker 1: that that that people will have heard of, so oxidative stress.
• 4:24
  Speaker 1: So let's let's go through that. You know, for the uninitiated,
• 4:30
  Speaker 1: explain oxidative stress. Explain the role of electrons in this.
• 4:35
  Speaker 1: You know, you can dive into electron transport chain if
• 4:38
  Speaker 1: you want, but just give our listeners a bit of
• 4:40
  Speaker 1: an overview of of the background around the role of
• 4:46
  Speaker 1: oxidative stress in eg well.
• 4:49
  Speaker 2: The key is it's oxidative.
• 4:50
  Speaker 3: It's it's basically your body is rusty and uh and
• 4:54
  Speaker 3: so and of course you know you've seen what does
• 4:56
  Speaker 3: the metal well do the same things your body. There's
• 4:59
  Speaker 3: very oxidaive radical superoxide that's an oxygen with an extra electron.
• 5:03
  Speaker 3: That's the most rusty thing you could possibly imagine for
• 5:07
  Speaker 3: doing damage. And then there's the hydroxyl radical that's a
• 5:11
  Speaker 3: that's a water molecule. It's missing its proton, so it's
• 5:13
  Speaker 3: got this loose electron and it wants to like, you know,
• 5:15
  Speaker 3: grab on and and and do something to something. And
• 5:19
  Speaker 3: that's another nasty and but there's good ones too. I mean,
• 5:22
  Speaker 3: for instance, nitrogen oxide that's a that's a vasodilator and
• 5:28
  Speaker 3: it's super important for health. You know, if you don't
• 5:30
  Speaker 3: have enough your your your blood pressure goes through the roof.
• 5:33
  Speaker 3: So and it's it's it uses signaling molecules, even hydrogen
• 5:36
  Speaker 3: peroxide though technically it's not an oxydaeve radical, it's still
• 5:39
  Speaker 3: oxidizes thing, but it's also a signaling molecule. There's a
• 5:42
  Speaker 3: bunch based around sulfur and iron and zinc and so,
• 5:45
  Speaker 3: so it's not like a bad thing. There's kind of
• 5:47
  Speaker 3: like a redoct balance. You have to have much. Too
• 5:50
  Speaker 3: much is bad, too little is also not good. Now,
• 5:53
  Speaker 3: there's a few things like superoxide and the hydroxy line
• 5:57
  Speaker 3: that the body doesn't use as signaling molecules. They're just
• 6:00
  Speaker 3: basically bad, but it's it's and most of that's produced
• 6:03
  Speaker 3: in the well. There's a couple of things the mitochondria.
• 6:06
  Speaker 3: They're the furnaces in your body that produce ninety five
• 6:09
  Speaker 3: percent of your cellular energy. And like any furnace, they're
• 6:12
  Speaker 3: going to produce, you know, basically pollution. And one of
• 6:15
  Speaker 3: those is superoxide. But also superoxide is also used by neurophils.
• 6:19
  Speaker 3: They're like little they're uh, your white one type of
• 6:23
  Speaker 3: white blood cell, and they basically use superoxide as machine
• 6:27
  Speaker 3: gun bullets against bacteria and other body invaders. And that's
• 6:31
  Speaker 3: you know, if you get the inflammation, you get an infection,
• 6:35
  Speaker 3: you see that inflammation that's basically being caused by a superoxide.
• 6:38
  Speaker 3: You know, your immune system fighting against what'sever invading your body.
• 6:42
  Speaker 3: So and and then there's other things that cause it, like,
• 6:46
  Speaker 3: for instance, radiation causes it a lot of hydroxy lions.
• 6:49
  Speaker 3: But things like now cell phones for instance. You know,
• 6:51
  Speaker 3: they always told us, oh these don't you know, because
• 6:54
  Speaker 3: they looked at the body, that the amount of heating
• 6:56
  Speaker 3: in the body, and they said, well, that can't possibly
• 6:59
  Speaker 3: cause damage. Well, now they found that the cell phone's
• 7:02
  Speaker 3: signal frequencies open up the the ion gated channel in
• 7:05
  Speaker 3: your body, in your cells, and so this this channel
• 7:09
  Speaker 3: opens up and and calcium can go rushing into your
• 7:12
  Speaker 3: cells and cause a lot of damage because it starts
• 7:15
  Speaker 3: doing all kinds of you know, chemical reactions. And that's
• 7:17
  Speaker 3: another source which you know before they said that wasn't doing.
• 7:20
  Speaker 3: And then of course there's all the other things you
• 7:23
  Speaker 3: know with modern living, like the terrible food people eat,
• 7:26
  Speaker 3: uh and you know, smoking, drinking, not getting enough sleep,
• 7:32
  Speaker 3: and even exercising. I mean, when you exercise, your monochondrias
• 7:35
  Speaker 3: are working really hard. They're producing a lot of superoxide.
• 7:38
  Speaker 3: I mean, and and if and and basically, as we
• 7:42
  Speaker 3: get older, there's a lot of indigenous or antioxidants that
• 7:46
  Speaker 3: our body produces to control these oxidative radicals. But as
• 7:49
  Speaker 3: we get older, they kind of go down and uh
• 7:52
  Speaker 3: and and and and when you do a lot of exercise,
• 7:55
  Speaker 3: I mean, that's why you feel bad two days later, right,
• 7:57
  Speaker 3: because you get a lot of damage. If if you're
• 7:59
  Speaker 3: if you're with your antioxidants, you're probably doing better because
• 8:02
  Speaker 3: what happens is superoxides produced tor mitochondria. Now your mitochondria's
• 8:06
  Speaker 3: make it fifty times more energy than it was when
• 8:07
  Speaker 3: you're just resting. So you get these superoxides like they'll
• 8:11
  Speaker 3: they'll get together with the nitrogen oxide and they'll make
• 8:14
  Speaker 3: peroxy nitrite. And that's kind of a long lived one
• 8:17
  Speaker 3: that's kind of with domes delay onset, muscle soreness. That's
• 8:20
  Speaker 3: like the two and also a lot of superoxides they'll
• 8:23
  Speaker 3: damage that, they'll damage your soul. They'll make aldehydes or
• 8:27
  Speaker 3: maldehydes that that kind of just well from aldehyde. They
• 8:32
  Speaker 3: just do more damage too. So it's just basically your
• 8:35
  Speaker 3: body rusting. And but there's signal molecules. It's a little
• 8:38
  Speaker 3: too little, you know, because like exercise, if you didn't
• 8:41
  Speaker 3: exercise at all, you'd be like, you know, pretty bad health.
• 8:45
  Speaker 3: But if you go too much, then you damage your body.
• 8:48
  Speaker 3: There's like a balance in everything, and that's what we're
• 8:52
  Speaker 3: kind of aiming with good health is to achieve that balance.
• 8:55
  Speaker 1: Think I think that's a really important thing for people
• 8:58
  Speaker 1: to understand, is that it is this this readox balance
• 9:02
  Speaker 1: is really really important. I mean, people here they hear
• 9:05
  Speaker 1: free radicals, and you know, the early thinging was oh, bad, bad, bad,
• 9:10
  Speaker 1: bad bad. We need to neutralize free radicals, all of them.
• 9:13
  Speaker 1: But as you say, they are important signaling molecules that
• 9:18
  Speaker 1: that that stimulate, and particularly the antioxidant story is really interesting,
• 9:26
  Speaker 1: right because I remember when I was doing my master's
• 9:29
  Speaker 1: degree in sports entition, it was just twenty odd years ago,
• 9:33
  Speaker 1: nearly thirty years ago, right, But so the theory was that,
• 9:38
  Speaker 1: you know, exercise produces free radicals, as you said, because
• 9:43
  Speaker 1: of that increased energy production in the mitochondria, and we're
• 9:48
  Speaker 1: we're producing free radicals, and therefore, when you're exercising, you
• 9:52
  Speaker 1: need lots of antioxidants. And you know, there's all this
• 9:55
  Speaker 1: big push on antioxidants, and then they find there's a
• 9:59
  Speaker 1: very famous study that showed that if you take antioxidants
• 10:03
  Speaker 1: around the time of exercise, you actually lose quite a
• 10:07
  Speaker 1: lot of the benefits of exercise, right, because you were
• 10:11
  Speaker 1: neutralizing those free radicals. And I think it's important for
• 10:16
  Speaker 1: people to understand is that balance is actually very very delicate,
• 10:22
  Speaker 1: and the overall health of your body influences that balance.
• 10:28
  Speaker 1: So I think where we've landed by particularly with antioxidants
• 10:32
  Speaker 1: and exercise, is that if you're doing regular amounts of exercise,
• 10:36
  Speaker 1: antioxidants can actually minimize some of the benefits if you've
• 10:41
  Speaker 1: got a healthy body. But if you're unhealthy or you're
• 10:44
  Speaker 1: doing super amounts of exercise like running ultramarthons, I mean
• 10:48
  Speaker 1: that's why we see post ultramarathons. There's oxidative damage. There's
• 10:53
  Speaker 1: also damage to the heart and things like that.
• 10:56
  Speaker 2: Right.
• 10:56
  Speaker 1: So it's like so much in science and the human body,
• 11:00
  Speaker 1: it's the Goldilocks effect, isn't It can't be too little,
• 11:04
  Speaker 1: can't be too much. It's got to be just right
• 11:08
  Speaker 1: because those three radicals they stimulate indgenous antioxidant defense mechanisms, right,
• 11:16
  Speaker 1: Superoxide dismute is includethion, perioxidy is those sorts of things.
• 11:21
  Speaker 1: And the way I often position it to people is
• 11:25
  Speaker 1: that when you take antioxidants, whether it's individual antioxidant or whatever,
• 11:32
  Speaker 1: you're kind of bringing in Dad's army to protect yourselves.
• 11:36
  Speaker 1: Whereas when you're actually producing your own antioxidant defense through
• 11:43
  Speaker 1: a little bit of stress, right, this hormesis, you're actually
• 11:47
  Speaker 1: producing a heap of special forces. Right, So that balance
• 11:52
  Speaker 1: is pretty key. But so why then, so let's let
• 11:56
  Speaker 1: then explore So if oxid of stress, you know, it
• 12:00
  Speaker 1: is obviously important, right, we need a little bit, but
• 12:03
  Speaker 1: too much is clearly quite bad. Why is it then
• 12:06
  Speaker 1: that some people who they sleep well, they eat well,
• 12:10
  Speaker 1: they're exercising, well, how is it that they still are
• 12:14
  Speaker 1: having negative effects or they're feeling in flam their low
• 12:18
  Speaker 1: energy you know what's going on there?
• 12:21
  Speaker 3: Well, I mean there's I don't know how many tens
• 12:23
  Speaker 3: of thousands of chemicals that exist in our our environment
• 12:29
  Speaker 3: now from the industrial age that never existed before.
• 12:32
  Speaker 2: We have those.
• 12:32
  Speaker 3: We have all the radiation, I mean the radio I
• 12:34
  Speaker 3: mean we're talking not just you know, atomic radiation, but
• 12:38
  Speaker 3: you know cell phone radiation, electromagnetic stuff. We've never had
• 12:41
  Speaker 3: that before in our history.
• 12:43
  Speaker 1: Yeah, getting getting on a plane regularly, and and.
• 12:46
  Speaker 3: Yeah, do you get a lot of that and and
• 12:48
  Speaker 3: and just all and then also the social stress of living,
• 12:52
  Speaker 3: you know, in cities with lots of people I mean,
• 12:54
  Speaker 3: there's just so many in our modern world, there are
• 12:57
  Speaker 3: just so many, and you know, and the the worst
• 13:00
  Speaker 3: of course is processed foods, highly processed foods like and
• 13:03
  Speaker 3: especially the seed oils, uh, you know, high.
• 13:06
  Speaker 2: Fructose corn syrup for hours.
• 13:09
  Speaker 3: You know, words that you look at the label. Basically,
• 13:11
  Speaker 3: if you can't read, if you can't pronounce the word
• 13:14
  Speaker 3: on the label, you probably shouldn't be eating it.
• 13:16
  Speaker 1: That's that's right exactly. I mean, my listeners have heard
• 13:19
  Speaker 1: me talk about a low hit Tai diet, low human interference.
• 13:23
  Speaker 1: But I think that that is important for people to understand,
• 13:27
  Speaker 1: is it's the the overall burden of of of not
• 13:32
  Speaker 1: just radiation, but things that are going to stimulate oxidative
• 13:37
  Speaker 1: stress and and other different pathways in the in the body. Right,
• 13:40
  Speaker 1: there's pollution as well. We didn't even talk about pollution, right, Oh.
• 13:43
  Speaker 2: Yeah, yeah, that's yeah, it's just terrible. Yeah.
• 13:45
  Speaker 3: So, and it's just just in today's modern environment, we
• 13:49
  Speaker 3: just have all of these stressorders that we've never had before,
• 13:52
  Speaker 3: and so sometimes you just need to take And the
• 13:54
  Speaker 3: other thing, of course, when we talk about food, I mean,
• 13:56
  Speaker 3: you could eat organic food and you should, but you
• 13:59
  Speaker 3: know that you do it all the time, but a
• 14:01
  Speaker 3: lot of our foods don't have the nutritional value they
• 14:03
  Speaker 3: used to have. Well be literally, you know, measure the
• 14:06
  Speaker 3: broccoli of today versus the broccoli of forty years ago.
• 14:08
  Speaker 3: And it's a lot of this has to do with
• 14:10
  Speaker 3: you know, the mind out soils, but a lot of
• 14:12
  Speaker 3: it's with the breeding. You know, they bred plants that
• 14:14
  Speaker 3: look all beautiful and wonderful, but you know they're not
• 14:17
  Speaker 3: like Grandma's plants that had like that full nutritional you
• 14:21
  Speaker 3: know profile that had the minerals and all the other
• 14:24
  Speaker 3: things that are really what you're eating food for.
• 14:27
  Speaker 1: Yeah, and I think I think a lot of people
• 14:29
  Speaker 1: don't understand is that a lot of the food that
• 14:33
  Speaker 1: we eat, particularly fruits and vegetables, and you know, they
• 14:38
  Speaker 1: have all of these different nutrients in them that that
• 14:41
  Speaker 1: play rules is enzymes and cofactors for the millions of
• 14:46
  Speaker 1: chemical reactions that are happening in your body. But interesting,
• 14:51
  Speaker 1: there's a little part of the nutrients and fruit and vegetables.
• 14:55
  Speaker 1: As we were talking earlier on about the Goldilocks effect
• 14:59
  Speaker 1: right there are I think it was Mark Professor Mark Mattson.
• 15:02
  Speaker 1: I remember reading twenty years ago about hormetic dietary polyphenol.
• 15:07
  Speaker 1: So I'm like, what the hell is this shit? And
• 15:10
  Speaker 1: he was the one that showed. So if we take broccoli,
• 15:12
  Speaker 1: like everybody knows, the procoly is great for you. Everybody
• 15:14
  Speaker 1: knows the broccoli sprouts are amazing. That's so furifreeing, and
• 15:18
  Speaker 1: is this potent compound that can fight cancer and things
• 15:22
  Speaker 1: like that, and people are talking about it as an antioxidant.
• 15:24
  Speaker 1: It's not an antioxidant. It's a bloody prooxidant.
• 15:27
  Speaker 2: Right.
• 15:28
  Speaker 1: It's a little dose of poison that in insects is
• 15:32
  Speaker 1: enough to stimulate the insect not to eat it, but
• 15:35
  Speaker 1: in humans, because we are much much bigger, it just
• 15:39
  Speaker 1: creates that very mild metabolic stress and then we upregulate
• 15:44
  Speaker 1: our stress our stress response mechanisms. Right, So we're again
• 15:48
  Speaker 1: we're back to this balance. But to your point, if
• 15:52
  Speaker 1: we are you know, eating those things, they could be
• 15:56
  Speaker 1: great in one thing, but if you have all of
• 15:58
  Speaker 1: this load, you're not well. Are generally other than that,
• 16:02
  Speaker 1: you're not sleeping well, you know, you're exposed to lots
• 16:05
  Speaker 1: of pollutions. The bodies defenses then get overwhelmed, right.
• 16:09
  Speaker 2: Yes, and and so and especially and also as we
• 16:12
  Speaker 2: get older.
• 16:13
  Speaker 3: I mean, there's monogenous antioxidants that our bodies produce, and
• 16:17
  Speaker 3: our ability, you know, creatine and all that you know, well,
• 16:19
  Speaker 3: you know, just various glycine and all that our body
• 16:22
  Speaker 3: you know, has biochemistry, and so to me to stay healthy,
• 16:25
  Speaker 3: I mean, you don't have to, but to optimize your
• 16:28
  Speaker 3: health as.
• 16:29
  Speaker 2: You get older than one of those bills that.
• 16:31
  Speaker 3: You kind of need to take. And uh, and it's
• 16:34
  Speaker 3: different for each person. I mean, some people, like I'm
• 16:37
  Speaker 3: really I mean, I'm not in the business, but getting
• 16:39
  Speaker 3: a genetic test is really important because you can tell
• 16:42
  Speaker 3: where you have weaknesses. And I've done mine, and you know,
• 16:45
  Speaker 3: there's I have, like I have really strong vitamin D
• 16:47
  Speaker 3: production and you know processing, but my vitamin b's you know,
• 16:51
  Speaker 3: I need extra supplementation on that. And some people have
• 16:55
  Speaker 3: like alleles that make so OD drop off faster than
• 16:58
  Speaker 3: other people.
• 16:59
  Speaker 1: Yeah, and so so sorry just for our listeners. That's
• 17:02
  Speaker 1: superoxide dismutias. Yeah, and and then other people who have
• 17:06
  Speaker 1: got alleles like the mt h f R, which is
• 17:10
  Speaker 1: called the motherfucker gene and hey, oh no, it's not
• 17:16
  Speaker 1: good if you've got certain allegles of that. So yeah, look,
• 17:19
  Speaker 1: I think you're right. It's this. It's this interplay between
• 17:22
  Speaker 1: your genetics, the inputs into your body, the good inputs
• 17:27
  Speaker 1: and the bad inputs. There's all of the psychology, but
• 17:30
  Speaker 1: then there's the aging process which then changes things. So
• 17:34
  Speaker 1: do your point. As we age a lot of these
• 17:37
  Speaker 1: defense mechanisms they start to drop off, or you know,
• 17:41
  Speaker 1: the electron transport g and just gets a bit tired, right,
• 17:44
  Speaker 1: and our mitochondria don't work particularly well. And I think
• 17:50
  Speaker 1: that's important for people to understand. So, for instance's stick
• 17:53
  Speaker 1: a molecule like glinac, which is glycine plus an acetyl
• 17:56
  Speaker 1: systing has got heapes of benefits in terms of oxidative
• 18:01
  Speaker 1: stress and anti inflate inflammation and things like that. But
• 18:06
  Speaker 1: there's absolutely no point in taking this if you're in
• 18:09
  Speaker 1: your twenties and thirties and forties, right, because.
• 18:13
  Speaker 2: Anythink yeah about that stuff when you're younger, That's right.
• 18:17
  Speaker 1: And I think that's important for people to understand that
• 18:20
  Speaker 1: that stuff that may be beneficial for somebody like me
• 18:25
  Speaker 1: who's in my fifties, would just be expensive piss for
• 18:30
  Speaker 1: somebody who's in their twenties.
• 18:32
  Speaker 2: Right, Yes, it is.
• 18:33
  Speaker 3: There's just yeah, you remember our twenties, and yeah, changes
• 18:37
  Speaker 3: as you get older, just little things here, a little
• 18:39
  Speaker 3: bit there. It's kind of a gradual thing. But when
• 18:41
  Speaker 3: you get in your thirties and forties you begin to
• 18:43
  Speaker 3: notice it.
• 18:45
  Speaker 1: So let's talk about carbon sixty then and were this
• 18:51
  Speaker 1: were this theoretically plays a role, right, and then we'll
• 18:56
  Speaker 1: dive a little bit into the mechanisms.
• 18:59
  Speaker 2: Yeah, well see sixty.
• 19:00
  Speaker 3: You know, it's basically what they've discovered. Now I go
• 19:04
  Speaker 3: over maybe the story a little bit, but it's really
• 19:06
  Speaker 3: what is It's an S O D catalyase mimic.
• 19:09
  Speaker 2: So it actually.
• 19:10
  Speaker 1: Explain that in layman's terms.
• 19:13
  Speaker 3: Yeah, there's a well, there's a superoxide disputase that I
• 19:18
  Speaker 3: talked about earlier. About that superoxide, you know, an oxygen
• 19:21
  Speaker 3: with an extra electron, the most damaging oxyde of radical
• 19:23
  Speaker 3: in your body. Well, your body makes superoxide disputase to
• 19:27
  Speaker 3: neutralize that.
• 19:28
  Speaker 2: Yeah, and that's that's the way.
• 19:29
  Speaker 1: And why why is this important? Like like like so
• 19:34
  Speaker 1: tell people so so when an electrons lake, right and
• 19:38
  Speaker 1: they attached to oxygen, they the kind of way I
• 19:42
  Speaker 1: describe it is they just become like a wrecking ball
• 19:45
  Speaker 1: inside your body and give people a sense of what's
• 19:49
  Speaker 1: kind of happening at a cellular level.
• 19:51
  Speaker 3: YEA, well, just it's just, yeah, it is a wrecking
• 19:53
  Speaker 3: ball because it's gonna that electron is gonna go off
• 19:55
  Speaker 3: and it's going to grab on to some molecule that
• 19:58
  Speaker 3: you need to have work, and it's going to take
• 20:00
  Speaker 3: out you know, it'll take uh you know, maybe it'll
• 20:02
  Speaker 3: tear a hydrogen out of it, or break a bond
• 20:05
  Speaker 3: between two oxygens or a nitrogen, right, we don't, you.
• 20:08
  Speaker 2: Know, it's are like a lot of times.
• 20:09
  Speaker 3: What it does is damages your cell walls, which are
• 20:13
  Speaker 3: basically fatty acids, and so it'll break the double bonds,
• 20:17
  Speaker 3: it'll it'll cause you know, your your cells will leak out.
• 20:20
  Speaker 3: I mean, it's just they're just all kinds of things
• 20:23
  Speaker 3: that this It just basically breaks down the molecular structure
• 20:26
  Speaker 3: of proteins, fats, you name it, of DNA especially and
• 20:30
  Speaker 3: you're your mitochondria they have their own little DNA. They're
• 20:33
  Speaker 3: actually organelles in your body.
• 20:36
  Speaker 1: Is not fascinating that your mitochondria and have your own DNA, right,
• 20:41
  Speaker 1: So these are inside the vast majority of our sales.
• 20:44
  Speaker 1: And yes, my understanding that you get your mitochondrial DNA
• 20:48
  Speaker 1: comes from your mum.
• 20:50
  Speaker 3: Yes, yeah, in the egg all the mitochondria or in
• 20:53
  Speaker 3: the egg, and yeah, you just get the the DNA
• 20:55
  Speaker 3: for your dad. But the yeah, the problem with mitochondria
• 20:59
  Speaker 3: though is they have their with that O DNA if
• 21:02
  Speaker 3: and they want to survive and continue and so like
• 21:05
  Speaker 3: if you're not getting enough antioxidants like sod or catalyse
• 21:08
  Speaker 3: or there's glued tothion, there's coke ten like in the
• 21:11
  Speaker 3: CREB cycle, that's you know, makes that ATP energy.
• 21:14
  Speaker 2: The model chondria.
• 21:15
  Speaker 3: Will turn down their thermost or their production rate, their thermostats,
• 21:19
  Speaker 3: whatever you want to call it. So now you're not
• 21:21
  Speaker 3: getting as much ATP as you did before. You don't
• 21:23
  Speaker 3: have that energy because that's just their way that you know,
• 21:26
  Speaker 3: that's the way they want to survive, so they turn
• 21:29
  Speaker 3: it down, and that's the you get the energy. As
• 21:30
  Speaker 3: you get older, you don't have as much energy primarily
• 21:33
  Speaker 3: because your model chondria aren't functioning the way they used to.
• 21:35
  Speaker 3: So you're not getting as much energy. So you feel
• 21:38
  Speaker 3: like you don't have as much energy because frankly, you don't.
• 21:41
  Speaker 1: You don't, you don't, And that's exactly right, and I
• 21:44
  Speaker 1: think you know, if we look at the there was
• 21:48
  Speaker 1: a very famous research paper produced I think it was
• 21:50
  Speaker 1: twenty thirteen called the Hallmarks of Aging, and they identified
• 21:56
  Speaker 1: nine different processes by which we aage. And then in
• 22:00
  Speaker 1: twenty twenty three, I think they did an update and
• 22:04
  Speaker 1: added another couple of different hallmarks. So there's now twelve
• 22:07
  Speaker 1: recognized hallmarks of aging, such as DNA damage and telling
• 22:13
  Speaker 1: me are shortening the su But for me, and I
• 22:15
  Speaker 1: actually presented on this last just this week at a conference,
• 22:20
  Speaker 1: and mitochondrial dysfunction is one of the twelve hallmarks of aging,
• 22:26
  Speaker 1: but it is the hallmark that affects more of the
• 22:30
  Speaker 1: other hallmarks. So for me, it's kind of central into
• 22:34
  Speaker 1: the equation because I think most people will have heard
• 22:37
  Speaker 1: of the mitochondria and they know that it produces energy
• 22:41
  Speaker 1: within the sale produces a TT, but Jesus, it does
• 22:43
  Speaker 1: so much more right way way more.
• 22:48
  Speaker 2: Yeah.
• 22:48
  Speaker 3: It makes like, for instance, pregntolone that's the precursor molecule
• 22:52
  Speaker 3: for all your hormones, so that's another thing. And also
• 22:55
  Speaker 3: it makes like precursors for RNA and DNA synthesis, and
• 22:59
  Speaker 3: you and the mitochondria in your different cell types like
• 23:02
  Speaker 3: the motochondria in your liver are going to be making
• 23:05
  Speaker 3: stuff just for your liver, versus say the motochondria in
• 23:09
  Speaker 3: your kidneys, which will be making additional stuff for your kidneys.
• 23:12
  Speaker 3: And so it's it's it's quite amazing all the different
• 23:16
  Speaker 3: things and we don't even know the half of it yet,
• 23:19
  Speaker 3: new things every month, and I mean it's hard to
• 23:21
  Speaker 3: keep up on a lot of that stuff these days.
• 23:24
  Speaker 1: Yeah, But I think the key thing, and this is
• 23:27
  Speaker 1: presented the conference, like the vast majority of human chronic
• 23:32
  Speaker 1: diseases have got mitochondrial dysfunction in there somewhere and often
• 23:37
  Speaker 1: driving it. And there's a a famous Harvard psychiatrist, doctor
• 23:41
  Speaker 1: Chris Palmer. He's written a brilliant book, Code Brein Energy,
• 23:45
  Speaker 1: and has basically shown that every single psychiatric condition has
• 23:49
  Speaker 1: got mitochondrial dysfunction in it.
• 23:52
  Speaker 3: Right, So yeah, that would make sense because if you're yeah,
• 23:55
  Speaker 3: and if your cells aren't working correctly in your brain,
• 23:59
  Speaker 3: then you know what's the you know, because if that's all,
• 24:01
  Speaker 3: you don't have.
• 24:01
  Speaker 2: The at P. You can't make those neurotransmitters. Yeah, yeah,
• 24:05
  Speaker 2: I mean you can't.
• 24:06
  Speaker 3: Have the at P, can't pull them back in and
• 24:09
  Speaker 3: and yeah, I mean if without ATP you're just and
• 24:12
  Speaker 3: it drops off. That's why you lot to see like
• 24:14
  Speaker 3: a lot of those conditions start appearing as people get older. Yeah,
• 24:18
  Speaker 3: one things were working better, but then they have a
• 24:21
  Speaker 3: weakness here or there, and then you get to a
• 24:23
  Speaker 3: certain age and bam, it starts showing up and uh,
• 24:26
  Speaker 3: and that's pretty common. And and that's pretty much the
• 24:30
  Speaker 3: degenerative diseases that people see as the age.
• 24:33
  Speaker 1: Yeah, and and and look at the risk can of
• 24:36
  Speaker 1: us to us to getting to geek so geeky that
• 24:39
  Speaker 1: that the eyes roll back in the head of our listeners.
• 24:44
  Speaker 1: And you know, there are there are different complexes in
• 24:48
  Speaker 1: the electron transport chain. They you know, they need new chains,
• 24:51
  Speaker 1: iron plays of roll b Vinamin's player role and co
• 24:55
  Speaker 1: q ten or you picked for them in that place
• 24:57
  Speaker 1: of roll in different field. Yeah. Yeah, so all of
• 25:00
  Speaker 1: these things. So we're let's come back to carbon sixty.
• 25:04
  Speaker 1: Where is this enter the conversation.
• 25:08
  Speaker 2: Is it.
• 25:10
  Speaker 1: Purported effects particularly in the electron transport chain or does
• 25:15
  Speaker 1: it have effects outside of that as well?
• 25:17
  Speaker 3: And it's just yeah, it doesn't really mess with the
• 25:21
  Speaker 3: electron transport chain.
• 25:22
  Speaker 2: What it does. It's a it's.
• 25:24
  Speaker 3: A SOD catalyase mimic. Now, usually what your body does
• 25:29
  Speaker 3: is that you get the superoxide that's an oxygen with
• 25:32
  Speaker 3: an extra electron, right, and then what happens is SOOD
• 25:35
  Speaker 3: changes that into hydrogen peroxide, and then catalyse comes by
• 25:39
  Speaker 3: and turns that hydrogen peroxide back into an oxygen in
• 25:42
  Speaker 3: the water's.
• 25:44
  Speaker 1: And buying neutralizes it all right.
• 25:46
  Speaker 3: R exactly, And you need a little ATP with every
• 25:48
  Speaker 3: one of those steps, right, and the enzymes that go
• 25:51
  Speaker 3: do that. What C sixty does is it basically just
• 25:53
  Speaker 3: pulls positive hydrogen ions from the outer environment and then
• 25:58
  Speaker 3: uses those to neutralize the super oxide. Basically, the superoxide
• 26:01
  Speaker 3: it makes itself positively charged. The superoxide is a negative charge,
• 26:04
  Speaker 3: it will stick to it. The C sixty basically just
• 26:07
  Speaker 3: steals the electron off of the superoxide, gives it to
• 26:10
  Speaker 3: one of the positive hydrogen ions, which now turns into
• 26:12
  Speaker 3: hydrogen gas, which is also an antioxidant and a signaling molecule,
• 26:16
  Speaker 3: and then the oxygen can just go back into the
• 26:19
  Speaker 3: CREB cycle, and C sixty also takes care of the
• 26:22
  Speaker 3: hydroxyl ion, and that's basically a water molecule. Missing its
• 26:26
  Speaker 3: proton and uh and what's really interesting is neither of
• 26:30
  Speaker 3: and the usually glue to thion does that job. And
• 26:32
  Speaker 3: it actually so good.
• 26:33
  Speaker 1: So just for this so I can't have to jump
• 26:36
  Speaker 1: in every nine and to pull the balloon back dance
• 26:39
  Speaker 1: of people understand. So glutas ion is the kind of
• 26:42
  Speaker 1: master antioxidant within the body, right, So the hydroxyl ion,
• 26:47
  Speaker 1: you know, why why is that? Why is that a problem?
• 26:50
  Speaker 3: Just because the it's it's when I mean, the the
• 26:54
  Speaker 3: it wants the electric electron. It wants to like balance
• 26:58
  Speaker 3: out water. I mean, it's really fundamental. It takes a
• 27:00
  Speaker 3: lot of energy to bust water off, so that water
• 27:03
  Speaker 3: now has a bunch of energy, and it will tear
• 27:05
  Speaker 3: stuff off. It'll tear a proton off, you know, a protein,
• 27:09
  Speaker 3: or damage or so all because it's it's hungry, it
• 27:12
  Speaker 3: wants you know, it wants to balance out those electron shells, right,
• 27:16
  Speaker 3: and so it just causes all kinds of damage just
• 27:18
  Speaker 3: the primarily it's the primary.
• 27:20
  Speaker 1: It's another.
• 27:22
  Speaker 3: Another nasty And what's interesting, the body doesn't use those
• 27:25
  Speaker 3: either of those antioxidants as a as a things. One
• 27:29
  Speaker 3: of the things about C sixty and C sixty is
• 27:31
  Speaker 3: you know, it was discovered nineteen eighty five, got a
• 27:33
  Speaker 3: Nobel Prize in nineteen ninety six in chemistry, and uh,
• 27:37
  Speaker 3: but what it But it's it's actually in the early Earth,
• 27:41
  Speaker 3: there was actually geologic deposits of this and if you
• 27:43
  Speaker 3: were in a really cold environment, C sixty would fit
• 27:46
  Speaker 3: exactly into the groove of the RNA and the side
• 27:49
  Speaker 3: groove of the DNA. And it's theorized in the ancient
• 27:52
  Speaker 3: pass C sixty was the original antioxidant of life because
• 27:57
  Speaker 3: it was it's hydrophobic, so it floats, but it's lipophilic,
• 28:00
  Speaker 3: so it attracts fats and amino acids and hydrogen gas
• 28:04
  Speaker 3: and so it was kind of like the and and
• 28:06
  Speaker 3: later life evolved s O D and glued thion and
• 28:10
  Speaker 3: catalyts to take care of some of the jobs that.
• 28:12
  Speaker 2: This thing used to do.
• 28:14
  Speaker 3: And of course, you know it disappeared, and but now
• 28:17
  Speaker 3: now you can get it.
• 28:18
  Speaker 2: Now you can. It's it's kind of back.
• 28:20
  Speaker 3: And it was also I could get more on that,
• 28:22
  Speaker 3: but that's that's kind of its key that people use.
• 28:24
  Speaker 3: We have a lot of doctors, dozens or hundreds now
• 28:27
  Speaker 3: that use it, and it's it's for your basically for
• 28:30
  Speaker 3: your listeners to help them kind of understand. There's basically
• 28:33
  Speaker 3: four key what we can call in doogenous antioxidants in
• 28:37
  Speaker 3: our body that are key to like general health, but
• 28:40
  Speaker 3: also the creb cycle.
• 28:41
  Speaker 2: Right, that's that atp stuff. Okay, One is co q ten.
• 28:45
  Speaker 3: Yeah, and if you've got gray hair, you should be
• 28:47
  Speaker 3: taking that. That's a super important one. And that's also
• 28:50
  Speaker 3: what statin is destroyed.
• 28:55
  Speaker 2: Yeah.
• 28:55
  Speaker 3: I can give medical advice, so but I I am
• 28:58
  Speaker 3: a research scientist, and I'll probably make comments which some
• 29:01
  Speaker 3: people might not like.
• 29:03
  Speaker 1: Well, I'm happy. I'm happy to say if you're on
• 29:06
  Speaker 1: a statin, you'd be fucking bunkers not to be taking coq.
• 29:10
  Speaker 3: Ten and exactly, in fact, if you're probably just bonkers
• 29:13
  Speaker 3: to be taking statins in the first place. Inside of research, iientist.
• 29:17
  Speaker 3: I can look at the papers and I can't see
• 29:19
  Speaker 3: any effic seat instead.
• 29:21
  Speaker 1: Yeah, but but the early staatens they did in the
• 29:24
  Speaker 1: early research, they had q coq ten in it because.
• 29:27
  Speaker 3: They know, yeah, because yeah, it just sucks coke ten out.
• 29:31
  Speaker 3: And I mean we're kind of getting off track here,
• 29:32
  Speaker 3: but that is something that people should know if they're
• 29:35
  Speaker 3: in coq ten is just super important.
• 29:37
  Speaker 2: Yeah, and.
• 29:40
  Speaker 1: One hundred percent and it plays a significant role in
• 29:42
  Speaker 1: the electron transport chain. Right, So so okay, so we've
• 29:45
  Speaker 1: got so sorry I interrupted fourth Yeah.
• 29:49
  Speaker 3: Number two, the glued to thion, and we mentioned that before.
• 29:52
  Speaker 3: That is like the master does like dozens of jobs.
• 29:55
  Speaker 3: It's it's super it's not it's like before it was
• 29:58
  Speaker 3: really hard to step. It won't make it past your gut,
• 30:01
  Speaker 3: your digestive system. So now they've got like transdermals and liposomals,
• 30:06
  Speaker 3: and so you can supplement for for glutathion, or you
• 30:10
  Speaker 3: can take the precursor amino acids and that's probably even
• 30:13
  Speaker 3: a better way to do it.
• 30:15
  Speaker 1: But that's where that's where knac plays a role.
• 30:17
  Speaker 2: Can exactly.
• 30:18
  Speaker 1: Sorry well, and and particularly glynac the glycine plus.
• 30:21
  Speaker 2: NCA is yeah, exactly.
• 30:23
  Speaker 1: And I said to people, don't take gludothione supplements and
• 30:26
  Speaker 1: it's just expense Well, it's not quite expensive pests because
• 30:28
  Speaker 1: it doesn't even survive your digestive system.
• 30:31
  Speaker 2: It just breaks it down in the amino acids.
• 30:32
  Speaker 3: Then your body can take those amino acids and reassemble it, right,
• 30:35
  Speaker 3: or you can just take the plane only amino acids
• 30:37
  Speaker 3: and let the body do it. Yeah, and there's other
• 30:40
  Speaker 3: there's other ways, but at least you can They have
• 30:43
  Speaker 3: ways now that you can supplement for it or get
• 30:45
  Speaker 3: the precursors in so your body can do it, and
• 30:47
  Speaker 3: that's really important. But there's really was never anything for
• 30:50
  Speaker 3: SOD and catalys because you know C sixty, you will
• 30:53
  Speaker 3: pass the gut barrier, pass the blood brain bearer, go
• 30:56
  Speaker 3: in your cells by endocytosis, that into the mitochondria by endocytosis,
• 31:00
  Speaker 3: where it acts as that so OD catalyse mimic. Now
• 31:03
  Speaker 3: you know the body your cells will excrete SOD into
• 31:07
  Speaker 3: the environment around them and catalyse into the environment around them,
• 31:10
  Speaker 3: but there's no uptake path, there's no channel that can
• 31:13
  Speaker 3: get it in there.
• 31:14
  Speaker 2: There is you can there is a way that they can.
• 31:16
  Speaker 3: The cells can make exosomes, little packages of exosomes, but
• 31:20
  Speaker 3: that's extremely Yeah, they don't really have that technology yet
• 31:24
  Speaker 3: to do for exosomes right for anything, hardly if they
• 31:28
  Speaker 3: sell them, but that's not very good. So basically, if
• 31:30
  Speaker 3: you had a problem with your so OD or catalyse
• 31:33
  Speaker 3: in the mitochondria system, there is nothing you can do
• 31:36
  Speaker 3: until C sixty came along and CCO is a mimic
• 31:39
  Speaker 3: that does the job of both of those in one shot.
• 31:42
  Speaker 3: And so and that's that's what like, we have a
• 31:44
  Speaker 3: lot of doctors and and this it's basically part of
• 31:46
  Speaker 3: a standard protocol that a lot of like I don't know,
• 31:50
  Speaker 3: functional medicine, doctors, natural paths, the other kind of the
• 31:53
  Speaker 3: people out there that they do is, you know, you
• 31:55
  Speaker 3: basically want to get your patient healthy, get healthy cells.
• 31:58
  Speaker 3: So any treatment you do on top of that, including
• 32:00
  Speaker 3: if you are a patient out there people listening, is
• 32:03
  Speaker 3: you know, you get the KOKE ten, you get the glutathion,
• 32:08
  Speaker 3: and then you get the C sixty which covers the
• 32:10
  Speaker 3: SOD and catalytes.
• 32:11
  Speaker 1: Now, sorry, Ken, we didn't We didn't actually finish that.
• 32:14
  Speaker 1: You didn't you didn't make the just to make the
• 32:17
  Speaker 1: connection for people, they are the other two antioptions.
• 32:20
  Speaker 3: With SOD and caddis those the other two are the four,
• 32:23
  Speaker 3: but you can't supplement for them. But see sixty is
• 32:26
  Speaker 3: a mimic that does the job of both and uh
• 32:28
  Speaker 3: and and that's really that's that's where it kind of
• 32:31
  Speaker 3: fits in. It's i mean, it's one toolkit in the
• 32:34
  Speaker 3: toolkit for good health. But it's certainly not the only thing. Yeah,
• 32:37
  Speaker 3: I should make them now, I mean, like vitamin D
• 32:39
  Speaker 3: and vitamin K. I mean that's that's a chronic problem.
• 32:42
  Speaker 2: Okay.
• 32:43
  Speaker 3: I mean here magnesium, right, you probably talked them. Proper sleep,
• 32:47
  Speaker 3: clean water. I mean, all of those things are necessary
• 32:50
  Speaker 3: because if you want to get your look as you
• 32:52
  Speaker 3: get older, it's gonna get harder and harder to keep
• 32:54
  Speaker 3: your health and you have to put energy into it.
• 32:57
  Speaker 3: It's not gonna happen, and you have to be the
• 32:58
  Speaker 3: advocate for your own health. That's kind of our attitude here. Look,
• 33:01
  Speaker 3: we can give you the tools, but really, if you
• 33:04
  Speaker 3: want to fix it, you've got to do it yourself,
• 33:06
  Speaker 3: and you've got to do the exercise you've got to
• 33:08
  Speaker 3: put in.
• 33:08
  Speaker 2: You know, you've got to do your own research. You
• 33:10
  Speaker 2: have to.
• 33:11
  Speaker 3: I mean, these are all the things because really your
• 33:13
  Speaker 3: health is your own responsibility. Yeah, there's nobody for you,
• 33:17
  Speaker 3: and there is no magic pill that's gonna it's going
• 33:20
  Speaker 3: to bring it back.
• 33:21
  Speaker 2: Sorry.
• 33:22
  Speaker 1: And the vitamin D one is really interesting because it's
• 33:26
  Speaker 1: you know, I come from a place where the sun
• 33:30
  Speaker 1: is a kind of mythical creature right in Northern Ireland,
• 33:33
  Speaker 1: and so everybody's always out any time there's any sort
• 33:37
  Speaker 1: of a sun And then I moved to Australia, where
• 33:39
  Speaker 1: we've got a big issue with with it, right and so,
• 33:42
  Speaker 1: and people are very sun aware in Australia. And to
• 33:48
  Speaker 1: touch a point that we now have vitamin D deficiency
• 33:52
  Speaker 1: and insufficiency in Australia at the same levels that you
• 33:55
  Speaker 1: get in northern Europe, which is it's yeah, yeah, a.
• 34:00
  Speaker 3: Lot of people don't know that the actual office workers
• 34:02
  Speaker 3: in Australia have a higher screen cancer rate than like
• 34:05
  Speaker 3: the surfer dudes.
• 34:07
  Speaker 1: And I've never heard that one, but I do know
• 34:09
  Speaker 1: that low vitamin D can increase your risk of many
• 34:14
  Speaker 1: different cancers, right, So we're trying to prevent one cancer.
• 34:18
  Speaker 1: And it's these unintended consequences, right if you're not getting
• 34:24
  Speaker 1: enough sun exposure, because good luck getting enough vitamin D
• 34:28
  Speaker 1: from your diet, right, But if you're not getting enough
• 34:31
  Speaker 1: sun exposure, you know, whether that's not being in the
• 34:34
  Speaker 1: sun or sunscreen, and having that low vitamin D can
• 34:39
  Speaker 1: be really problematic because it affects almost every one of
• 34:43
  Speaker 1: the major hallmarks of aching. So there's these unintended consequences
• 34:48
  Speaker 1: that happen, right.
• 34:50
  Speaker 3: Oh yeah, And all the office workers. It's actually from
• 34:52
  Speaker 3: the fluorescent lights because what they do is they have
• 34:55
  Speaker 3: mercury vapor in those fluorescent lights and they have phosphorus
• 34:58
  Speaker 3: around the outside. And then what happens that mercury vapor
• 35:01
  Speaker 3: produces a lot of ultra violet A and B as
• 35:06
  Speaker 3: well as E and CO. Of it will tunnel out
• 35:09
  Speaker 3: through that phosphorus and so that's the nasty stuff that
• 35:13
  Speaker 3: that U V A and that u V B and stuff.
• 35:16
  Speaker 3: And so you're sitting in like a fluorescent light bulb
• 35:19
  Speaker 3: is actually emitting a lot of A and u V
• 35:21
  Speaker 3: A and B and damaging people. And that's why these people,
• 35:24
  Speaker 3: at least with under I don't know what with you know,
• 35:28
  Speaker 3: phosphorescent those lights, you know, they they just they they
• 35:32
  Speaker 3: leak you know, UV stuff, and most people didn't even
• 35:34
  Speaker 3: know that. And that's why those office workers were having
• 35:37
  Speaker 3: all kinds of cancers because they're getting like really nasty
• 35:40
  Speaker 3: doses of of UV radiation that.
• 35:43
  Speaker 1: Ah, well, I didn't know that. I wasn't aware of
• 35:45
  Speaker 1: that at all. And let's come back to the today,
• 35:48
  Speaker 1: the carbon C sixty. So we've we've established here that
• 35:53
  Speaker 1: you know, our mitochondria need to function well that that
• 35:57
  Speaker 1: we are just from daily living, just from existing, and
• 36:02
  Speaker 1: we are producing oxidative stress. And then we have these
• 36:06
  Speaker 1: antioxidants that go in and mop that up. And that's
• 36:08
  Speaker 1: a that's an important balance, a redox balance that can
• 36:13
  Speaker 1: change over time as you eage and and the effectiveness
• 36:16
  Speaker 1: of those antioxidant systems tends to drop off. And then
• 36:21
  Speaker 1: so we can we can supplement co Q ten, we
• 36:24
  Speaker 1: can take precursors for glut to thione. But then you're
• 36:27
  Speaker 1: saying the SOD and the catally is there's nothing that
• 36:29
  Speaker 1: we can take that will will stimulate that. And that's
• 36:32
  Speaker 1: where carbon C sixty comes.
• 36:34
  Speaker 3: Now, that's that's where it fills it up. So it'll
• 36:36
  Speaker 3: and that's what basically we're talking to the gray hair
• 36:38
  Speaker 3: croucher folks. Yeah, as you get older, our SOD and
• 36:41
  Speaker 3: catalase production goes down along with coke ten and glued
• 36:43
  Speaker 3: to thion and and I mean, to stay good health,
• 36:46
  Speaker 3: you need to supplement to a certain extent. And uh,
• 36:49
  Speaker 3: and that's it's just one of those things that does it.
• 36:53
  Speaker 3: There's a lot of other things too that that we've
• 36:55
  Speaker 3: discovered over time. And because it's it has some interesting
• 36:59
  Speaker 3: uh other effects. Uh, one of the one of the
• 37:03
  Speaker 3: I guess I could talk some of the studies the scientific.
• 37:06
  Speaker 1: Yeah, yeah, yeah, Well I was just about to get
• 37:07
  Speaker 1: into this. So so there is throwing out the devil's advocate, right,
• 37:11
  Speaker 1: So there is a and there's been lots of molecules
• 37:15
  Speaker 1: that are out there that have this plausible biological mechanism.
• 37:19
  Speaker 1: But you've got to be able to take that molecule.
• 37:21
  Speaker 1: It's got to be digested, it's got to get to
• 37:24
  Speaker 1: where it's supposed to you in the body, and then
• 37:26
  Speaker 1: it's actually got to have the effect. And sometimes that
• 37:29
  Speaker 1: doesn't play out in in real stuff. Sometimes it happens
• 37:33
  Speaker 1: in animals, but it doesn't happen in humans. So this
• 37:37
  Speaker 1: was my question, where are where is this deed of
• 37:39
  Speaker 1: the science in carbon three sixty? I'm aware of that
• 37:42
  Speaker 1: rat study, right, but but humans humans aren't big rats, right,
• 37:46
  Speaker 1: So it's over there there.
• 37:48
  Speaker 3: There's lots of studies and you know, some studies you
• 37:51
  Speaker 3: can't do on humans, right, but studies where you put
• 37:55
  Speaker 3: human cells in test tubes.
• 37:57
  Speaker 2: Yes, So that's there's a lot of those studies.
• 38:01
  Speaker 3: Basically, as I said, discovered ninet eighty five got a
• 38:03
  Speaker 3: Nobel Prize in ninety six. And then when they went
• 38:06
  Speaker 3: for testing for like toxicity, well, first they used they
• 38:09
  Speaker 3: used a type of the famous botry study is they
• 38:12
  Speaker 3: use wister rats. And wister rats are really wimpy rats,
• 38:17
  Speaker 3: you know, because if you're going to study for toxicity,
• 38:19
  Speaker 3: you don't want a street rat, right, you want a
• 38:20
  Speaker 3: wimpy rat. And so usually they'd have cancers or other
• 38:23
  Speaker 3: degenerative diseases associated with aging. But when they gave wister
• 38:27
  Speaker 3: rats C sixty, they increased their lifespan by ninety percent,
• 38:33
  Speaker 3: and narily by preventing cancers, which I'll probably get into
• 38:37
  Speaker 3: a little bit later, though not directly that way, but
• 38:40
  Speaker 3: really one of the other studies that you know, they
• 38:42
  Speaker 3: it took five and a half years for that study,
• 38:45
  Speaker 3: and it was it was, you know, they were dragging out.
• 38:48
  Speaker 3: In fact, they did another study during that study. They
• 38:50
  Speaker 3: decided because they had so much time because the C
• 38:52
  Speaker 3: sixty rats, i mean all the control rats that died
• 38:54
  Speaker 3: in the C sixty rats were refusing to die, right
• 38:56
  Speaker 3: the wister rats. And so they gave one where they
• 38:58
  Speaker 3: had two sets of rats. One was they exposed them
• 39:01
  Speaker 3: to carbon tetrachloride and not only your your audience out there.
• 39:05
  Speaker 2: Carbon tetrichloid is what carboride.
• 39:09
  Speaker 3: It's a carbon added with four chlorines on it, and
• 39:12
  Speaker 3: it's extremely toxic. They used to use it in dry cleaning, okay,
• 39:18
  Speaker 3: and it was terrible effects on the dry cleaners and
• 39:20
  Speaker 3: then it would leak out in the environment. It was
• 39:22
  Speaker 3: like one of those Really, it's banned now pretty much.
• 39:24
  Speaker 3: It's one of those things so bad that you know, well,
• 39:26
  Speaker 3: you know, like asbestos, right, they didn't.
• 39:28
  Speaker 2: Know about it until oops, and so it's one of
• 39:31
  Speaker 2: those things.
• 39:31
  Speaker 3: So they basically with the C sixty rats, the rats
• 39:33
  Speaker 3: have got C sixty didn't have any liver damage, whereas
• 39:37
  Speaker 3: the control rats had like big lesions in their livers.
• 39:40
  Speaker 3: And there's there's a website what is C sixty dot org.
• 39:43
  Speaker 3: It's like a five to one P three that our
• 39:46
  Speaker 3: the company I work with and other companies support because
• 39:50
  Speaker 3: on our websites and stuff, we can we can show
• 39:52
  Speaker 3: you human studies, but we can't talk about animal studies.
• 39:55
  Speaker 3: We can't mention you know, what's this or that, the
• 39:57
  Speaker 3: bad and the ugly. But what is C sixty dot
• 40:00
  Speaker 3: calm is? What is C sixty dot com? Is a
• 40:02
  Speaker 3: five O one C can show you the good, bad,
• 40:04
  Speaker 3: and the ugly, so that you can kind of know
• 40:06
  Speaker 3: what's going on.
• 40:07
  Speaker 1: And so so why why are you not allowed to
• 40:09
  Speaker 1: talk about the animal studies and.
• 40:13
  Speaker 2: The FDA and stuff.
• 40:14
  Speaker 3: It's very tight here in the United States, and so
• 40:17
  Speaker 3: like we we have studies like, for instance, the very
• 40:19
  Speaker 3: first study of C sixty, they put in a cream
• 40:21
  Speaker 3: for Japanese women and they put it on ja and
• 40:24
  Speaker 3: it was found to decrease lines and wrinkles in Japanese women.
• 40:27
  Speaker 3: We now know that's because C sixty increases stem cellars.
• 40:31
  Speaker 1: Right, But you're allowed to put but you'd be allowed
• 40:33
  Speaker 1: to put human studies like that on your website.
• 40:36
  Speaker 3: Oh yeah, absolutely any human study or human cell type
• 40:38
  Speaker 3: in but any animal studies because you can't say what
• 40:41
  Speaker 3: happens to an animal can be applicable to a human being.
• 40:44
  Speaker 2: So that's not allowed to be stad So to.
• 40:47
  Speaker 1: My point earlier on right, yes exactly, But again that
• 40:50
  Speaker 1: that doesn't mean that that everything that happens in an
• 40:52
  Speaker 1: animal doesn't happen any human.
• 40:54
  Speaker 2: No, No, it's.
• 40:55
  Speaker 1: Got to have and this is where the science starts
• 40:57
  Speaker 1: to stack up, right, So.
• 40:59
  Speaker 3: You have, yeah, C sixty happens at the cellular level
• 41:02
  Speaker 3: down there in the mitochondria and all that stuff, which
• 41:05
  Speaker 3: is almost the same for all animals, right, especially mammal
• 41:08
  Speaker 3: I mean, the difference between a rat and a human
• 41:10
  Speaker 3: in the cellular inner functions are very little because we
• 41:15
  Speaker 3: have you know, we're very very identical.
• 41:17
  Speaker 2: But there's other studies, Like there's the other study. It's
• 41:20
  Speaker 2: basically when they.
• 41:22
  Speaker 3: A carboxy fullering SOD mimic improves cognition and extends the
• 41:26
  Speaker 3: life span of mice. And this one basically they kind
• 41:29
  Speaker 3: of learned from the first study. And what they did
• 41:32
  Speaker 3: is they took wild type mice, right, which is more
• 41:35
  Speaker 3: like what people would be, yeah, and they found that
• 41:38
  Speaker 3: it increased the life span by eleven percent and completely
• 41:41
  Speaker 3: prevented cognitive decline during agent.
• 41:44
  Speaker 1: Now I would just step in here and then we'll
• 41:47
  Speaker 1: come back to this. Why wild type is really important
• 41:50
  Speaker 1: is that a lot of the studies that we see
• 41:53
  Speaker 1: in mice, and you know, there's people promoting stuff and
• 41:56
  Speaker 1: you look at the study and the studies and mice
• 41:58
  Speaker 1: they tend to be these bread mice, right, And although
• 42:02
  Speaker 1: there's a ship heap of them in the study, you're
• 42:04
  Speaker 1: essentially doing a study on one one person because they
• 42:09
  Speaker 1: are they're in bread and all the same, whereas the
• 42:11
  Speaker 1: wild mice they have different genes just like me and
• 42:13
  Speaker 1: you have different changes are just gene variants, right, And
• 42:17
  Speaker 1: so that's why any study you gotta look at it,
• 42:22
  Speaker 1: whether it's a and and and in bread mouse, which
• 42:25
  Speaker 1: is the ones that they use in most of those experiments,
• 42:28
  Speaker 1: or a wild type mouse. Right, And then then we
• 42:30
  Speaker 1: look at is there a plausible biological mechanism? Does that
• 42:35
  Speaker 1: biological mechanism happen in humans as well?
• 42:38
  Speaker 2: Right?
• 42:38
  Speaker 1: And there's where we started get increasing evidence. So I
• 42:41
  Speaker 1: just wanted to kind of close that loop for people
• 42:44
  Speaker 1: back to you.
• 42:44
  Speaker 2: Yeah, and so basically what it is, because you know
• 42:46
  Speaker 2: what is good?
• 42:47
  Speaker 3: What good is getting old if you're just going to
• 42:49
  Speaker 3: end up have a long life span, if you're gonna end.
• 42:51
  Speaker 2: Up drooling in a wheelchair.
• 42:53
  Speaker 3: What you're really looking for as a health span health
• 42:55
  Speaker 3: spent the time in your life when you're you know,
• 42:57
  Speaker 3: you're healthy and functional, like you can live life. Right,
• 43:01
  Speaker 3: You're we're like us, right, we're living a life, you know,
• 43:04
  Speaker 3: having you know, experiences, and that's health span is what
• 43:07
  Speaker 3: you're looking for. Because lifespan doesn't mean anything if you're
• 43:11
  Speaker 3: just der crepit in a wheelchair.
• 43:13
  Speaker 1: Yeah, yeah, so we're where are we? Where are we?
• 43:16
  Speaker 1: So let me take let let let me do do
• 43:19
  Speaker 1: a comparator here. So we got something like glinac, right,
• 43:23
  Speaker 1: which has been shown in animal studies to reduce oxiedative stress,
• 43:26
  Speaker 1: reduce inflammation, improve cognition, and improve lifespan. Right now. You
• 43:31
  Speaker 1: can't do that same study in humans, right because we
• 43:34
  Speaker 1: can have me and you you know, for for thirty
• 43:38
  Speaker 1: forty fifty years. But in humans, Glenac has been shown
• 43:42
  Speaker 1: to reduce oxeative stress, reduce inflammation, right, and works on
• 43:46
  Speaker 1: the same pathways that you see in the mice. But
• 43:48
  Speaker 1: then in the mice it ends up with enhanced lifespan
• 43:51
  Speaker 1: and enhanced cognition. So where are we in using that
• 43:57
  Speaker 1: as a comparative? Where are we in the C sixty
• 44:00
  Speaker 1: research right now?
• 44:02
  Speaker 3: Okay, Well, they like one of the studies they did,
• 44:04
  Speaker 3: They found that C sixty helped cells resist damage from
• 44:07
  Speaker 3: UV radiation. So they took human skin cells, they put
• 44:11
  Speaker 3: them in the test tubes. Right, one set got C sixty,
• 44:14
  Speaker 3: the other set didn't. They hit them with UV radiation.
• 44:17
  Speaker 3: The C sixty cells resisted damage from the UV radiation.
• 44:20
  Speaker 3: That's why they did things with like human brain cells,
• 44:23
  Speaker 3: like you know, they put them in a test tube,
• 44:25
  Speaker 3: you know, the cells. The C sixty was given to
• 44:28
  Speaker 3: the nerve cells. You know, controls didn't get it. They
• 44:31
  Speaker 3: exposed the nerve cells to a variety of toxins. The
• 44:34
  Speaker 3: nerve cells that had the C sixty resisted damage versus
• 44:38
  Speaker 3: the controls. And it's basically because usually when things break down,
• 44:43
  Speaker 3: they produce a lot of things like superoxide and hydroxyl radicals.
• 44:47
  Speaker 3: Those are high energy, super damaging ull oxy radicals and
• 44:51
  Speaker 3: and and those you know, when those those complicated things
• 44:54
  Speaker 3: break down in the body, that's what they're producing a
• 44:56
  Speaker 3: lot of those as they break down, and C sixty
• 44:59
  Speaker 3: just gets rid of those before they caused damage and
• 45:02
  Speaker 3: that and that's pretty much the reason why they that
• 45:05
  Speaker 3: you know, you get these increase in health thing like, well,
• 45:08
  Speaker 3: there's a study. This is a study they kind of
• 45:10
  Speaker 3: led to one. They have a rabbits. They had this
• 45:12
  Speaker 3: study in Japan by rabbits. They gave rabbits arthritis, okay,
• 45:16
  Speaker 3: which is you know, inflammation in the joints. Yeah, and
• 45:19
  Speaker 3: then they injected C sixty into those joints and they
• 45:23
  Speaker 3: found that it reduced inflammation, which was expected, right, the
• 45:27
  Speaker 3: neurophyll that do you like it out? But when they
• 45:29
  Speaker 3: sacrificed the rabbits, because that's what they do in science,
• 45:31
  Speaker 3: and they looked at the and they looked at the
• 45:34
  Speaker 3: at the actual joints, they found that C sixty had
• 45:38
  Speaker 3: the C sixty rabbits had regrown the cartilage in their joints,
• 45:43
  Speaker 3: and that was totally unexpected. And then, you know, because
• 45:46
  Speaker 3: this is a story, it's still ongoing, but a story,
• 45:48
  Speaker 3: what they found out is C sixty. Basically, there's certain
• 45:52
  Speaker 3: kinds of cells in your body that called sinesseen cells.
• 45:55
  Speaker 3: They're kind of like zombie cells used damage.
• 45:58
  Speaker 1: Yeah, it's can I used the for anybody who has
• 46:02
  Speaker 1: seen Game of Thrones and sinessen cells are like the
• 46:05
  Speaker 1: white Walkers.
• 46:06
  Speaker 2: Yes, yes, exactly, yes exactly.
• 46:09
  Speaker 1: That's those shit aren't good because they they leak out
• 46:12
  Speaker 1: inflammatory side of kinds and they infect nearby sales and
• 46:16
  Speaker 1: stuff like that.
• 46:17
  Speaker 2: Right, yes, so throw it back to you.
• 46:19
  Speaker 3: Oh yeah, that's exactly right. They're just they're poisoning all
• 46:21
  Speaker 3: their neighbors, right, And usually it's because there's usually ways
• 46:24
  Speaker 3: that like when cells get to a certain way, they
• 46:26
  Speaker 3: have apoptosis or program tell death, but a lot of
• 46:29
  Speaker 3: these ones don't. And what happens is the cell wants
• 46:32
  Speaker 3: to live, and so it goes back to a more
• 46:34
  Speaker 3: primitive state. It basically goes back into the fermentation method,
• 46:38
  Speaker 3: which like we burn oxygen and the cit These Sinessen
• 46:40
  Speaker 3: cells mostly go to the fermentation mode where they're you know, fermenting.
• 46:44
  Speaker 3: They're like ancient primitive cells and that's where they're making
• 46:46
  Speaker 3: all this nasty toxins. Well, what happens when you take
• 46:49
  Speaker 3: C sixty. C sixty goes into those Sinessen cells and
• 46:52
  Speaker 3: it restarts the creb cycle in the mitochondria. And what
• 46:56
  Speaker 3: happens is those mitolchondria will then give messages the nucle
• 47:00
  Speaker 3: do your name, and they won't get the right message
• 47:02
  Speaker 3: back and that causes that Then the mitochondria themselves.
• 47:06
  Speaker 2: Will like blow up.
• 47:07
  Speaker 3: They'll cause apoptosis or programed cell death of those Sinessen cells,
• 47:12
  Speaker 3: and so your body gets wiped out of sines and cells.
• 47:15
  Speaker 1: So is this is this a mechanism through autophogy or
• 47:20
  Speaker 1: or is it similar sophogy?
• 47:24
  Speaker 2: Yeah?
• 47:24
  Speaker 1: Right, okay, so.
• 47:27
  Speaker 2: It's not inflammatory.
• 47:28
  Speaker 3: What happens is these cells break down into apo tophic bodies,
• 47:31
  Speaker 3: which then the phagocytes come by and just clean up.
• 47:34
  Speaker 3: It's not an inflammatory process.
• 47:37
  Speaker 1: And ken Ken sorry, just on that, Sorry to interrupt.
• 47:40
  Speaker 1: I think it's important for people to understand that that
• 47:44
  Speaker 1: this autophogy is a constant process. It happens within the celle,
• 47:49
  Speaker 1: it happens within the mitochondria. We call it mitophogy. And
• 47:53
  Speaker 1: but then there is there's a more broader autophogy which
• 47:58
  Speaker 1: is just cleaning up those those This is why long
• 48:01
  Speaker 1: term fasting like three four day five day fast, once
• 48:05
  Speaker 1: you hit a bite day three, your body starts to
• 48:08
  Speaker 1: it just becomes a massive recycling machine and it recycles
• 48:12
  Speaker 1: those sinesse and sales.
• 48:13
  Speaker 2: Right.
• 48:14
  Speaker 1: So I just want people to understand exactly.
• 48:16
  Speaker 3: Yeah, when we talk about that apoptosis apops, it's basically
• 48:20
  Speaker 3: like the janitors, right and the garbagemen, right, they go
• 48:22
  Speaker 3: because if you even take out your garbage, pretty soon
• 48:25
  Speaker 3: you know you know how the health yep.
• 48:27
  Speaker 2: So it's the same way with cells.
• 48:28
  Speaker 3: You need to you need to clean those suckers out,
• 48:30
  Speaker 3: which which is actually I'm an intermittent faster myself and
• 48:33
  Speaker 3: I've been doing that and then every now and then
• 48:35
  Speaker 3: you do like the forty eight hour fast for the
• 48:37
  Speaker 3: twenty four hours or thirty six are usually forty eight seven.
• 48:40
  Speaker 3: Those are you know, those are those are important things too,
• 48:42
  Speaker 3: because you know, I'm just talking a little bit about
• 48:44
  Speaker 3: C sixty, but remember we're C sixty is one little tool,
• 48:47
  Speaker 3: right all the rest of the things you need.
• 48:49
  Speaker 2: To do to stay good health.
• 48:51
  Speaker 3: And and what happens is basically causes the wife's out
• 48:54
  Speaker 3: those sinessen cells and now your organ or tissue doesn't
• 48:58
  Speaker 3: have enough cells to do the job. So that stimulates
• 49:02
  Speaker 3: stem cells to make more stem cells. And stem cells
• 49:04
  Speaker 3: are like baby cells. They haven't decided what they want
• 49:06
  Speaker 3: to do, and so then your body makes a bunch
• 49:08
  Speaker 3: of these baby cells and then like if your skin
• 49:11
  Speaker 3: cells need more, you know, if your skin needs more cells,
• 49:13
  Speaker 3: then the stem cells turn into your skin cells. That's
• 49:16
  Speaker 3: why the Japanese women had had more you know, their faces,
• 49:20
  Speaker 3: because they had more cells to turn over, so they
• 49:22
  Speaker 3: made more cartilage and or you know, all the cells
• 49:26
  Speaker 3: that didn't have the wrinkles, and then the rabbits they
• 49:29
  Speaker 3: had damaged cartilage, right, so the stem cells then converted
• 49:33
  Speaker 3: themselves into cartilage cells and help regrew the joints. And
• 49:37
  Speaker 3: then like in my case, the retina, the retina well
• 49:40
  Speaker 3: to a certain extent, probably had got retina stem cells
• 49:43
  Speaker 3: to going in my retina.
• 49:44
  Speaker 2: Of course.
• 49:44
  Speaker 3: The other thing is brain cells are like half mitochondria
• 49:47
  Speaker 3: by weight, and that's where C sixty really does job
• 49:50
  Speaker 3: anything that has a lot of mitochondria in it, like
• 49:53
  Speaker 3: stem cells, I mean, I mean, well, like brain cells, cells,
• 49:57
  Speaker 3: the endricn system, and of course muscles, especially the heart.
• 50:01
  Speaker 1: Yeah, they're they're they chase that that mitochondria are. So
• 50:06
  Speaker 1: I think it's about forty of the heart volume is
• 50:09
  Speaker 1: made up from mitochondre, right, Yeah, crazy.
• 50:12
  Speaker 3: It is crazy, And like in this in the brain
• 50:14
  Speaker 3: cells it's behalf. But actually I learned that long ago.
• 50:18
  Speaker 3: It actually sells the ender consistem even more mitochondria than
• 50:23
  Speaker 3: nerve cells.
• 50:24
  Speaker 2: I didn't know that they're working.
• 50:26
  Speaker 3: So hard, right, And so that's the key when you
• 50:29
  Speaker 3: take C sixty, Its just basically you know it's gonna
• 50:32
  Speaker 3: any cell type that's got a lot of mitochondria, you're
• 50:34
  Speaker 3: gonna get those mitol kundry, especially as you're older, right,
• 50:36
  Speaker 3: if you're in your twenties or eighteens, you know it's
• 50:39
  Speaker 3: not gonna be a thing. As you get older, you know,
• 50:40
  Speaker 3: you don't have as much SOD, so it'll turn those
• 50:42
  Speaker 3: mityl chondria back on and then you make more atp
• 50:46
  Speaker 3: you make more pregnant alone the precursor molecule. And like
• 50:50
  Speaker 3: our CEO, Jess, I start kind of helped start the company,
• 50:53
  Speaker 3: just came on, helped she runs it because she's much
• 50:56
  Speaker 3: better at that. But she just had a baby at
• 50:58
  Speaker 3: forty nine, her first baby.
• 51:00
  Speaker 2: Wow. Yeah, yeah, so's Yeah, it's a C sixty. There's
• 51:04
  Speaker 2: lots and lots of C sixty babies. So that is
• 51:07
  Speaker 2: a question. So you be walked out.
• 51:10
  Speaker 3: There if you're I should say that is I don't
• 51:12
  Speaker 3: know what you call those side effects or something.
• 51:14
  Speaker 2: Is that yeah, that's.
• 51:15
  Speaker 1: Because an unintended consequence.
• 51:18
  Speaker 3: Yes, and it increases libido in both men and women
• 51:21
  Speaker 3: a huge amount, because what's happening is it's just you're
• 51:24
  Speaker 3: stimulating the cells of your endercan system. So now your
• 51:27
  Speaker 3: pineal gland will make more melatonin. You know, your hypothomos
• 51:31
  Speaker 3: complex there will make uh, you know, more T and
• 51:34
  Speaker 3: T three and T four, and then you got your
• 51:37
  Speaker 3: h what is it the other thing?
• 51:38
  Speaker 2: Anyway, your human growth home.
• 51:39
  Speaker 3: I mean, all the cells you're adrenal glands, they're going
• 51:42
  Speaker 3: to be making more mineral corder coids. Oh, which is
• 51:44
  Speaker 3: by the way, if you're on UH, if you're on
• 51:46
  Speaker 3: blood thinners, you probably need to want to talk to
• 51:48
  Speaker 3: your doctor and get more testing. And because that's usually
• 51:51
  Speaker 3: determined by your mineral corder coids produced to your adrenam glands.
• 51:54
  Speaker 3: And so when you take C sixty, underlying problem can
• 51:57
  Speaker 3: go away. And so you know, as mintal cord acord
• 52:01
  Speaker 3: production goes down, and if you're taking a blood thinter,
• 52:03
  Speaker 3: now your blood might become too thin. But the dreams
• 52:07
  Speaker 3: also produce uh the androgens and that turns into testosterone
• 52:11
  Speaker 3: progester and you know, uh estrogen, and so that's kind
• 52:15
  Speaker 3: of the other thing that you have to go out there.
• 52:17
  Speaker 3: So yeah, so you'll definitely, I mean you'll you'll you'll
• 52:20
  Speaker 3: get a huge increase in so you get better sleep,
• 52:24
  Speaker 3: more energy. It's it's all across the board. It's not
• 52:26
  Speaker 3: like hormone therapy replacement where you just get one hormone.
• 52:30
  Speaker 3: It's just like everything will increase.
• 52:32
  Speaker 1: So where are we sitting just as we finish up,
• 52:35
  Speaker 1: where are we sitting on the human research right? Now,
• 52:38
  Speaker 1: are there are there any randomized control trials going on
• 52:42
  Speaker 1: to your knowledge of C sixty in humans at the minute, Yes,
• 52:46
  Speaker 1: Are there any been published? Uh?
• 52:48
  Speaker 2: Well, we everyone. I think we've been publishing art.
• 52:51
  Speaker 3: We did one with people with vision because people were
• 52:54
  Speaker 3: telling us, you know, their cataracts were going away, and
• 52:57
  Speaker 3: we actually did a study and we found out what
• 52:59
  Speaker 3: the cat exu for not going away, what was happening.
• 53:01
  Speaker 3: Improvement in the in the retina, in the nerve cells
• 53:05
  Speaker 3: was really the reason.
• 53:06
  Speaker 2: It was like a research PhD. You know.
• 53:08
  Speaker 3: Uh, Optometrists State Board approve the whole thing. I think
• 53:12
  Speaker 3: we're trying if that's published or not yet. They're working
• 53:14
  Speaker 3: on getting that published. But there's some there's also a
• 53:17
  Speaker 3: study and it's in South Carolina but they haven't done
• 53:19
  Speaker 3: it yet. And they found C sixty is really great
• 53:22
  Speaker 3: with post traumatic stress syndrome soldiers. You know a lot
• 53:25
  Speaker 3: of the guys broken from the Gulf Wars and stuff.
• 53:28
  Speaker 3: They come back with a lot of damage and uh,
• 53:31
  Speaker 3: they just a lot of treatments don't work. But they
• 53:33
  Speaker 3: found that when they give them C sixty and they
• 53:35
  Speaker 3: get you know, the brain functioning, that any any any
• 53:40
  Speaker 3: treatment they give, especially with the post traumatic stress syndrome
• 53:43
  Speaker 3: is way more effective.
• 53:45
  Speaker 1: We've helped that has been published that I haven't.
• 53:48
  Speaker 3: Been published yet. I mean, we've helped. I've actually helped
• 53:51
  Speaker 3: some soldier guys do that. I taught them how to
• 53:53
  Speaker 3: make C sixty preparations because they were just new started
• 53:56
  Speaker 3: doing it on their own because they he was he was,
• 53:59
  Speaker 3: he was an ex.
• 53:59
  Speaker 1: Soldier making their own C sixty.
• 54:02
  Speaker 3: Well, no, not to C sixty, but making preparations of it.
• 54:05
  Speaker 3: So I talked, because it's pretty it's it's actually it's
• 54:07
  Speaker 3: it's C sixty costs more by weight than gold and
• 54:10
  Speaker 3: everything else, and so yeah, it's pretty expensive.
• 54:14
  Speaker 2: A way to make it good?
• 54:16
  Speaker 3: Yeah, there you go, And yeah, we I was working
• 54:18
  Speaker 3: with them so that they could, you know, do it
• 54:20
  Speaker 3: themselves because you know, the VA here doesn't really it's
• 54:25
  Speaker 3: wounded warriors very well.
• 54:27
  Speaker 1: So so ken. So so we give that's a really
• 54:31
  Speaker 1: good background to round that all of the malecia mechanisms
• 54:35
  Speaker 1: and and and C sixty and where it's purported to work.
• 54:38
  Speaker 1: Is there are there any people that that should be
• 54:43
  Speaker 1: cautious about C sixty and and you know, what about
• 54:48
  Speaker 1: what about the age any point in younger people taking
• 54:52
  Speaker 1: C sixty.
• 54:53
  Speaker 2: Well, we did have a one of our here, one
• 54:55
  Speaker 2: of she works with us. She gave it.
• 54:58
  Speaker 3: Well, it's we can't say, you know, for children because
• 55:00
  Speaker 3: it's not an FDA proof thing, but her son, it's
• 55:03
  Speaker 3: like for athletics. C sixty is we've had like mid
• 55:07
  Speaker 3: packers in you know, in their sports come to be
• 55:11
  Speaker 3: champions their field using C sixty and with horses, but
• 55:14
  Speaker 3: we weren't selling them to the horses. That's somebody ok
• 55:17
  Speaker 3: in Island by the way, some Irish horses taking C sixty.
• 55:20
  Speaker 3: So yeah, it definitely, no, it will definitely increase athletic performance. Facts,
• 55:24
  Speaker 3: it's the only antioxidant there will because C sixty only
• 55:27
  Speaker 3: interacts with superoxide and the hydroxy lion, which the body
• 55:31
  Speaker 3: does not use as signal molecules, right because he takes
• 55:34
  Speaker 3: vitamin D or vitamin E.
• 55:35
  Speaker 2: And so they actually took C sixty to ten thousand.
• 55:37
  Speaker 3: Times the health beneficial dose, which if you use vitamin
• 55:41
  Speaker 3: E or vitamin D you would have killed the organ.
• 55:45
  Speaker 3: There was no toxicity at all.
• 55:47
  Speaker 2: None.
• 55:48
  Speaker 3: Obviously you're not getting any more benefit from taking more
• 55:50
  Speaker 3: than a certain amount, but there's no toxicity because because
• 55:53
  Speaker 3: C sixty only interacts with superoxide and the hydroxy lion
• 55:57
  Speaker 3: which the body doesn't use as signea molecules and all
• 56:00
  Speaker 3: it interacts with it doesn't interact with it.
• 56:02
  Speaker 1: So it doesn't have any other effects in metabolism.
• 56:05
  Speaker 2: No, no, it doesn't.
• 56:06
  Speaker 3: It doesn't become part of the metabolism because it's not
• 56:09
  Speaker 3: it's not like a nutrient. It's it's it's an allotropic carbon.
• 56:14
  Speaker 3: And it probably in the ancient past, as I discussed before,
• 56:16
  Speaker 3: it like filled a role, and of course they went
• 56:19
  Speaker 3: away and then the body came up with new ways
• 56:21
  Speaker 3: of producing something, you know, like sad and catalyzed to
• 56:24
  Speaker 3: do the job that C sixty was doing on superoxide.
• 56:26
  Speaker 3: But you know C sixty can still do the job
• 56:29
  Speaker 3: that it used to do in cells if you supplement
• 56:31
  Speaker 3: with it.
• 56:32
  Speaker 2: And it's just it's it's.
• 56:33
  Speaker 3: Just a really I mean, we I mean, I can
• 56:36
  Speaker 3: just tell you a story after a story of just
• 56:38
  Speaker 3: amazing things that because and I think like we had
• 56:41
  Speaker 3: a like a lady she had she got she went
• 56:44
  Speaker 3: to enough for all of this. She was having problems
• 56:46
  Speaker 3: and he and she had thirty percent kidney function loss right,
• 56:50
  Speaker 3: so thirty percent was lost and her nephrologist told her,
• 56:53
  Speaker 3: it's just gonna get worse till you know, you have
• 56:55
  Speaker 3: to need a kidney transplant. She took C sixty for
• 56:57
  Speaker 3: several months and she went back one hundred percent kidney
• 56:59
  Speaker 3: function because it wasn't a kidney problem. It was a
• 57:02
  Speaker 3: mitylchondria problem and probably sod or catalyse related. And when
• 57:07
  Speaker 3: you got C sixty in there, and there's like hundreds
• 57:10
  Speaker 3: of mitochondria quote diseases, but it's really just you know,
• 57:13
  Speaker 3: and if if we're gonna have an analogy for your listeners,
• 57:16
  Speaker 3: C sixty is like the radiator fluid. If if your
• 57:20
  Speaker 3: body wears a car, C sixty is the radiator fluid.
• 57:23
  Speaker 3: So if you've got a problem with the radiator, it's
• 57:25
  Speaker 3: going to fix that. Now, there's other things, you know,
• 57:27
  Speaker 3: with the engine oil and the fuels, like key tones
• 57:30
  Speaker 3: and all that stuff that you've probably talked about that
• 57:32
  Speaker 3: those are important things, but kind of C sixty is,
• 57:35
  Speaker 3: you know, if you're gonna get your cars gonna run,
• 57:37
  Speaker 3: you want to make sure that the radiar fluid is
• 57:38
  Speaker 3: topped off and working great. And so that's that's what
• 57:41
  Speaker 3: C sixty places. You know, you want to get the
• 57:44
  Speaker 3: engine oil and the windshell washer stuff and the fuel.
• 57:47
  Speaker 3: That's another that's the whole other stories and other things,
• 57:50
  Speaker 3: but it's just one of those important things that's kind
• 57:52
  Speaker 3: of critical to a cellular function and good health.
• 57:56
  Speaker 1: That's a really cool summary and a nice way to
• 57:59
  Speaker 1: end it. So can thank you very much. This has
• 58:02
  Speaker 1: been a great tour of cellular cellular metabolism and function.
• 58:07
  Speaker 1: Or can people go to find out more? Where do
• 58:10
  Speaker 1: we send all of the listeners?
• 58:11
  Speaker 2: Oh?
• 58:12
  Speaker 3: There, you can go to shop C sixty dot com
• 58:15
  Speaker 3: and don't forget you go to what is C sixty
• 58:17
  Speaker 3: dot org to learn a little bit more about it.
• 58:18
  Speaker 3: There's a lot more to learn about C sixty, but
• 58:20
  Speaker 3: it's it's you know, it's just a short term. But
• 58:22
  Speaker 3: shop C sixty dot com and that's our website.
• 58:25
  Speaker 1: You can go to Chase that that was a good
• 58:27
  Speaker 1: r L to get shop.
• 58:28
  Speaker 3: C And also we're the only company that does third
• 58:32
  Speaker 3: party testing. Okay, And another thing is we there's not
• 58:36
  Speaker 3: we're not the only company, but uh we we also
• 58:39
  Speaker 3: use sublimated C sixty sublimations like evaporation or distillation.
• 58:44
  Speaker 2: That's when a solid turns into a gas the back
• 58:46
  Speaker 2: into a solid. Uh. There's also an industrial production method of.
• 58:49
  Speaker 3: Making C sixty and and they use methyl benzene.
• 58:55
  Speaker 1: Which I don't know what it is, but it doesn't.
• 59:00
  Speaker 2: Bad. It's like a No.
• 59:00
  Speaker 3: One carcinogen and then they bake it off, right, But
• 59:02
  Speaker 3: it's there's lots of industrial uses for C sixty, but
• 59:05
  Speaker 3: if you want to use human consumption C sixty, you
• 59:08
  Speaker 3: want to make sure it's sublimated, and you want to
• 59:11
  Speaker 3: make sure it's third party tested, because we we actually
• 59:13
  Speaker 3: test all our competitors because we're the only ones that
• 59:15
  Speaker 3: does it, and like forty percent of the guys out there,
• 59:17
  Speaker 3: Zippo C sixty thirty percent of the guys out there
• 59:22
  Speaker 3: have like insufficient amounts to be effected. So you're only
• 59:25
  Speaker 3: talking about a third of the market. And there are
• 59:27
  Speaker 3: other good C sixty companies out there besides prop C
• 59:30
  Speaker 3: sixty dot Com or technically C sixty Power, but that's
• 59:33
  Speaker 3: our company name.
• 59:34
  Speaker 2: There's others, but.
• 59:34
  Speaker 3: You just have to be careful because there's a you know,
• 59:37
  Speaker 3: it's it's like in the supplement business. You already know
• 59:39
  Speaker 3: about that, yeah, yeah, yeah.
• 59:40
  Speaker 1: There's so there's so many supplements that just that do
• 59:43
  Speaker 1: not have what they say they have, or are you
• 59:47
  Speaker 1: know a Mega three fatty acids. There was a ship
• 59:49
  Speaker 1: heap of supplements shown to the being the ones that.
• 59:52
  Speaker 2: Oh god, that's yeah.
• 59:53
  Speaker 3: I always buy you Omega threes out of the refrigerating
• 59:55
  Speaker 3: section of your health food storks. Never buy fatty acid
• 1:00:00
  Speaker 3: us off the shelf. Please don't do that.
• 1:00:03
  Speaker 1: I completely agree. So keay, this is this has been awesome.
• 1:00:07
  Speaker 1: So shop three sixty dot com and what is C
• 1:00:10
  Speaker 1: sixty dot org that's where listeners can go. So thank
• 1:00:14
  Speaker 1: you very much all the way from Denver, Colorado.
• 1:00:17
  Speaker 2: Have a great text