Latest / Ageless and Timeless Podcast® / Amy Saphic, Dr. Sam

Transcript

Amy Saphic, Dr. Sam · Ageless and Timeless Podcast®

0:08
Good morning everyone. This is Michelle Hughes from Ageless and Timeless. Today,
0:13
I have two guests that I've met recently in December, and actually I met
0:19
one of the guests and the other one I'm meeting today for the first time
0:22
with you. My first guest is Amy Amy. Do you say your name
0:27
spay Hick? Is that it's spoh? That is that Hungarian? Or it's
0:33
Bosnian? Bosnia? Oh wow? And then my other guests, who I
0:38
have not met until this morning is well, I should say, I'm meeting
0:43
him right now with you. And he is the medical director with Amy for
0:47
Heartcare. And his name is doctor Sam and doctor Sam Fillingame. That's great,
0:54
Okay. What nationality is that? English? English? So we have
1:00
the United Nations represented here today. Well, thank you very much for joining
1:04
us. This is really a pleasure, and let me just give a little
1:07
background to who you are, why we are gathered together to talk about everything
1:15
to do with the heart today, which the name of the company will tell
1:19
you that that's our main focus. It's called, as I mentioned, Heartcare
1:23
Corporation. Um So, so what happened is that the show in December at
1:30
the American Academy of anti aging. I was walking, just walking along the
1:36
show, and I saw this one booze and I don't know what happened,
1:40
but there was an intuition. My my intuition said you need to stop here,
1:46
and so I stopped and I did the test and Amy was there and
1:49
she did actually did the test. I mean the test that they do is
1:53
called ms MCG and it means multi function Cardiograph correct right ult cardiogram Crediograham.
2:04
I'm sorry, and so what I want to tell everybody that's viewing us today
2:09
this is probably the most efficacious and important and compelling test for you to do
2:17
for your heart. And if your doctor doesn't know about it, then after
2:22
today's podcast, please please let your doctor know about them. You can look
2:27
them up on their website and also on my website because we all have some
2:34
information there. But so so let me just let them tell you what the
2:38
test is. But before we start that, because I wanted to just let
2:42
you know who who doctor Sam and Amy are. So. Amy has a
2:46
really fascinating background. When she was in her teens, her father had a
2:51
huge heart attack and that kind of turned Amy's life around because she decided at
2:55
that point that she was going to focus her energy and education and all her
3:04
efforts on heartcare. Is that Is that a good way to describe it?
3:07
Amy? It was like life changing for you, right, Yes, it
3:10
is. When I was fifteen, he had a quadruple bypass, and you
3:15
know, I was mad because his heart grew another artery of branch out of
3:20
the outside on the side of his right heart. And we were taught that,
3:23
you know, the heart can't reverse itself, and we're finding that,
3:27
yes, your heart is a muscle and it does reverse. So I was
3:30
mad. You know, we don't have anything for early detection or early prevention
3:34
to stop that from or catch up before it even happens for a lot of
3:38
families. Doctor Shen Joseph T. Shen from Premiere Heart. He's the developer
3:43
of the MCG and he actually was cracking the code cardiovascular micro cardiovascular disease with
3:51
the MCG the same time that I was drawing the machine. So we have
3:54
a really good connection with our mission to save lives. Isn't that amazing though,
4:00
how life works? I mean truly that you know, what has meant,
4:04
what is supposed to happen will happen. I mean, the fact that
4:08
you and he were aligned at a comfortable time in life and that you came
4:15
together. That's the amazing part that you It's like miraculous that you would meet
4:19
each other. And doctor Sam, how did you get involved with Amy?
4:25
Well, I've been spending the better part of my thirty four years in practice
4:30
working with cardiovascular risk reduction. I had three patients that passed away back to
4:34
back in my earlier days in practice from heart attacks, and they died slowly,
4:40
and it completely devastated me. So I went out and got all the
4:45
training I could possibly find to become better at reducing events, and shortly thereafter
4:53
I got pretty good at it. It wasn't long after that I started teachings.
4:58
I've spent the better part of twenty five years teaching across the nation these
5:02
tools that I've learned to not only stop the progression of the disease, but
5:06
to reverse the disease. And meanwhile used multiple devices to help me in my
5:14
detection disease, but I've never seen the device that could do with this one
5:18
did. This is the biggest breakthrough we've had in thirty years in cardiovascua disease.
5:23
This is going to be a game changer when he gets in the hands
5:27
of physicians and they learn how to use it and then learn how to do
5:30
disease reversal. That's that's amazing. So you know how and then how did
5:35
you any amy find each other through a mutual connection with doctor chen I genuinely
5:45
and you'n amy, And he put us together because he thought we'd make a
5:46
good fit. Next chality, we make a wonderful hit. And you so
5:51
you are the chief medical officer, correct, that is correct? And I
5:57
love what I do. So so okay, So and you're you're a chiropractor
6:01
or he says he oh, umtastopath, which is you know a little more.
6:10
It's a lot more senior than than than chiropractor. Right, so you
6:15
I'm a physician. The ultra physicians m deser positions. We we prescribe to
6:19
everything else. Same Doctor Shen is a medical doctor, right, Amy,
6:26
Yes, he's an electro physiologist. Background. He's the one I met at
6:30
the show. He was standing there chatting away at everybody. Oh no,
6:34
not doctor Shen. Oh okay, who was the one I met? It
6:38
was he was a vice president then or um that that is Yuel Glenn.
6:43
He is the CEO of Heartcare Corps. Okay, he's the CEO. Okay,
6:47
so what is your exact titled? And amy founder? I'm the founder
6:51
of Heartcare Corp. And the chief clinical officer okay. And and and doctor
6:57
Sam he's also a co founder, is a founder in chief medical usher.
7:01
Very good, Okay, So let's dive in. This is really important information.
7:05
So tell us first the science behind MCG. How does it qualify for
7:13
FDA approvals? I know you've been working, you've worked on so give us
7:17
a little bit more of the history of why this We're going to talk more
7:23
about what it is, but why it is scientifically sound. Let sell this
7:29
machine span development for over twenty four years now, and it's worked through a
7:33
large database of seriously ill cardiovascur patients. It's been tested on over two million
7:39
patients, and it received FDA approval years ago. It won the top award
7:46
in the Innovation of the Year in American Heart Association in twenty seventeen. Would
7:51
have gone on the market in two and twenty, but COVID hitn't that stifled
7:57
the ability to go with this out on the market, so it was delayed
8:01
a couple of years and we just started introduced this to physicians and it's it's
8:05
it was slow starting to begin with, because it's a new technology. It
8:09
takes doctors a few months to kind of the cipher and see for themselves what
8:15
this does. But it's begin to take off in class four or five months.
8:20
And so we've been incredibly busy teaching doctor group, actor doctor group how
8:24
this works. And the schedule is is is almost beyond me. Now,
8:33
well, you started during COVID. I'm starting to go to you know,
8:37
promote this in the time when doctors were focused on other things. So it
8:43
must have been a bit challenging, right to try to present when doctors were
8:48
distracted by COVID patients that needed more immediate care. And even though heart disease
8:54
was one of the conditions in COVID that caused people to get COVID re exists.
9:00
So it is kind of a catch twenty two that you would have to
9:03
have faced those challenges when actually your technology would have helped to prevent some COVID
9:11
pieces correct, it would have helped tremendously. In fact, with the disease
9:18
itself, it caused a vasculitis, it also caused a lot of card eumopathies
9:22
and myocarditis. This machine sees all of that, and it sees it in
9:28
early stages and so we could have seen it from the beginnings of when it
9:31
started to hurt the patient tool it was resolving with proper treatment. So you
9:37
can use this device to see if your medical management's working and we don't have
9:41
any other tools that do that. Today's podcast is brought to you by Gigenics.
9:46
From stem cell therapy to lifestyle coaching, Gigenics is the perfect place to
9:52
go for all your health and wellness needs. This health clinic is amazing at
9:58
getting you personalized medicine and wellness that is unique to you. Use the code
10:03
ageless and you'll get twenty percent off your first service with them. Go to
10:09
www dot g gen x q I g E n i x dot com to
10:16
find out more information about these powerful methods. So let's talk about the machine
10:22
itself. So I'm one of your victims and knows that you know, I
10:28
was hesitant. I didn't know what it was. They're putting all these probes
10:31
on the sensors on you, and really it's painless and it's only what fifteen
10:39
or twenty minutes, and you're reading a person's entire heart energy and any blockages.
10:48
So tell me, let's let's talk more with everybody here today too,
10:52
so they understand what the MCG does. Whatever we're entering your world, do
10:58
you wouldn't do it in I'll just say the patient party what they see when
11:03
they come in. So the MCG is a ten minute scan. We do
11:07
five scans of eighty two seconds each. There's a clamp that goes on each
11:11
wrist, each ankle, and a lead that goes on V five which is
11:15
under the left chest area outer part of the chest. And you lay there
11:20
comfortable, you try not to fall asleep. We don't have any cell phones
11:24
near you or any metals that can interfere with the machine. And it's six
11:28
minutes twenty two seconds. We get the whole total of your microvascular hearth help
11:35
and once we upload that scans to the internet, we get your report immediate.
11:39
It's all based on AI. And then doctor Sam, you can continue.
11:45
Yes, I think artificial intelligence isn't an end to all parts of life
11:48
now, and it's certainly not a stranger to medicine. It's never entered into
11:52
this world before though, and this artificial intelligence allows us to use signal processing
11:58
techniques basically look at a frequency domain. It's it's not like an ordinary KG
12:03
where you go on a time based mechanisment. Long story short, we're seeing
12:07
things that we could have never seen Elsewhy is the all of the technologies that
12:13
are out there today will only detect the disease and it gets into later stages
12:18
when a person is in quite a bit of trouble. And the problem with
12:22
that is two thirds of heart attacks occur before they get to that stage,
12:24
because two thirds of heart attacks occur due to plaque rupture, a smaller exclusive
12:30
disease. So you could have a vessel that's thirty to fifty percent blocked and
12:33
it ruptures, and that's the number one cause of heart attacks that we have
12:35
today. So there was no technology that would catch these people, and we
12:39
now have something they'll do that. Now this machine goes way past just detecting
12:45
with the blockage of arteries is seen. It actually sees when a heart becomes
12:50
stiffened. And so there are several different pathways that a heart becomes stiffened in
12:56
the way that it leads toward heart failure. So we can see the two
13:00
different pathways and we can begin to do something about it and then plan our
13:03
therapy around that and see if it's working. It sees congenital defects. So
13:09
many people are born with birth defects in the heart and had no idea,
13:13
but this actually picks up on that and it gives us some clues whether we
13:16
need to work on that. You can basically look at the information here and
13:20
listen to the heart two different value of the areas that actually will be pronounced
13:24
with murmurs if you actually have these problems, and so it gives a doctor
13:30
some cluesive how to address that. And then also this device picks up on
13:33
global asynchrony, which has to do with one side of the heart tracking before
13:37
the other side because of blood supply issues. It picks up on mitochondrial dysfunction
13:43
which has to do with the energy and the cells. And it sees myocardidas,
13:48
which is a big thing in many different ways of going disease state going
13:54
on on it. And it also tells us when we have myocard or remodeling,
14:00
which is to do with the heart stiffening and scar tissue related to zarious
14:03
disease states. And so when you start adding all this information together, there's
14:09
so much we can do. There's almost nine hundred thousand people a year that
14:13
have heart disease to the point of heart attacks, and many of those succumb
14:18
to that, and we can basically identify them earlier, we can begin stopping
14:22
the great majority of those three hundred thousand people a year die from heart failure,
14:28
and five million people in United States alone have to deal with it.
14:31
Wouldn't it be nice if we had six or eight years warning so we could
14:35
prevent that before ever occurred. Possibly these heart failures would have never had to
14:39
occur if we knew that they were coming and we did something about it.
14:43
And so rhythm disturbances, it actually sees a rhythm disturbance before it happens.
14:48
You always hear about that patient that went to sleep and didn't wake up,
14:52
and you just assume that they succumbed to heart something. But some of those
14:58
patients succome to venture defibrillation, which is basically the heart rhythm disturbance that occurs
15:03
for to six in the morning when you're adrenaline levels are at the lowest part
15:07
of the day, so they just go to sleep and never wake up.
15:09
That we actually can see a person that's potentially going in that direction that we
15:13
can put a defibrillator in before it actually happens, if that's necessary. So
15:18
the tools are endless here, and that's why I said this is the biggest
15:20
development cardiovast disease in over thirty years. So would you say the uniqueness of
15:28
your MCG is the AI or is there something further than that that makes you
15:35
different differentiates you from whatever else is out there. You can never do what
15:41
this machine did without artificial intelligence, and so yes, that's what makes it
15:45
unique. I'm sure that there are going to be many aspects of medicine that
15:48
we use artificial intelligence to break through barriers that we had. But this took
15:54
twenty three years, twenty four years in the development to get to the stage.
15:58
It's quite common implicated to figure out what each of these peaks stand for
16:03
and then quantify them and then get them sensitive enough to pass through the muster
16:07
of testing. But there's eleven different tests now that are peer reviewed that show
16:14
how well this machine works. They were done in the United States, but
16:18
they were also done a number of other countries on four different continents, and
16:22
so this device has been incredibly well studied and that artificial intelligence is a deal
16:30
breakers or in this case, a dealmaker, right, So what else is
16:36
technologically is potentially going to happen to it continued to improve your technology, well,
16:45
artificial intelligence just gets smarter as the date goes on. So the more
16:48
uses it has, the smarter it yets. It's already if you compare this
16:55
to the present technologies, it's already more sensitive and more specific than every technology
17:00
out there. So it's already superior to anything on the market. In fact,
17:04
there's nothing even close. But it's only had two million studies. If
17:10
you let this thing go out on the market like it's doing now, it
17:11
will be no time at all. Of this will have ten million studies and
17:15
thirty million studies and fifty million studies, and the amount of learning that takes
17:21
place with our efficient tels, it's just going to get better. It will
17:22
get more and more specific to the point it will be ridiculously good at picking
17:27
up details, and it's going to help save countless lives. Anticipate in the
17:33
approximately five years, every year in America will have one of these because you
17:37
can actually detect who's having a heart attack and who's not within ten minutes at
17:41
the time they get to the station to the hospital. We're actually looking at
17:45
some emergency management systems with ambulances, putting the machines on the ambulances so you
17:52
actually have them test available when you get to the hospital because transit times for
17:56
some places is over twenty minutes. Being said, every clinic in America should
18:03
have one of these in ten years. Once you start getting this available to
18:07
the levels I'm talking about, that's about five to six hundred thousand people a
18:11
year that are dying today that won't be dying in six to eight to ten
18:15
years, depending on how long it takes us to get out here. But
18:18
when you go to your doctor's office now, you almost expect them to have
18:21
an EKG because that's just standard. But I suspect eight or ten years this
18:26
will be replacing every EKG that's out there, So everywhere you would use an
18:30
EKG would use this. I'm very excited about what it does for women's heart
18:34
disease because women have been in a precarious situation for years because forty nine percent
18:40
of women have small vessel disease before they get large vessel disease, and they
18:44
will carry that small vessel disease on for six to eight years, and it
18:48
makes them incredibly fatigued and they get some shortness of breath with exertion with that,
18:52
but it's not detectable by any device on the market today, and so
18:56
these patients, these women will have hardcasts and be told that there's nothing that
19:00
can be found, there's zero percent blockage, and six months later they'll be
19:04
in the er with one hundred percent blockage. Because what happens when they get
19:07
to critical stage disease. Something flips a switch in the women and they go
19:11
from zero percent blockage to one hundred percent blockage in two to three vessels in
19:17
six months time. Why why is that, doctor Sam, It's a million
19:22
dollar question. I wish I had the answer for nobody knows what flips the
19:26
switch, but we know what happens in the advanced stages. So it's usually
19:29
when a person gets to that extreme fatigue. But it's one of the reasons
19:33
that women go undetected more often than men because you have to catch them in
19:37
that six month window when they go from zero percent to one hundred percent,
19:41
and they tend to get studied in that high fatigue level when they don't have
19:45
any blockage and so it has been throwing off our system. But now we
19:48
actually have the ability to see it with this device because this machine see small
19:52
vessels ease extremely well. In fact, you just can't miss it. So
19:57
now that we've got some way of detecting this, we can tell a female
20:02
patient that they're having trouble and this is what's doing it. Now that same
20:04
process happens in fifteen percent of men, so it's not exclusive to women.
20:08
Fifteen percent of men do the same thing, but it's just much more common,
20:12
so we're three times more common in women. So it's been a real
20:17
bothersome problem for years that we finally have a solution to. You think it
20:22
has something with women to do with hormones. Could it could, But that
20:29
wouldn't explain the fifteen percent of men that do it. But there's probably some
20:33
co variant factors and some genetics that play into it. But it's something that's
20:40
going to need to be study in the future. We didn't actually know that
20:44
this was for sure occurred until two thousand and seven and it was published in
20:47
circulation and they finished the study in twenty thirteen and it continued to show the
20:52
same parameters, so there was periods of times there that doctors argued over the
20:57
fact where the small vessels is easy existed, but that arguments it's going now
21:02
it's called microvascular disease or small vessel disease. And maybe what about diet and
21:08
nutrition? What influence does that have not only on the small vessel disease but
21:15
just overall on Harthouse? So what do you what is your composal to your
21:22
clinics about teaching their patient the doctor's patients about better better diet. Diet's going
21:30
to play a huge factor. Now you have to funderstand I spent the better
21:33
part of my last twenty five years, almost twenty eight taking care of nothing
21:38
about high risk cardiovascular patients, and I would get ninety and ninety five percent
21:42
blocked arteries on a regular basis, and they would almost every single one reverse
21:48
and go back to essentially no blockage. In the way that was done was
21:52
knowing that there were ten disease states that create inflammation. It's all about inflammation,
21:56
and the patient that controls the inflammation wins. Inflammation gets within the artery,
22:02
and once it gets within the artery, it causes a signal to go
22:06
out. It is called a chemokinans the chemokind that brings the fat into the
22:10
cell. So if fat never enters a blood vessel until it gets an invitation,
22:15
to invitation is from inflammation that secretes a chemical called a chemokinda. That
22:19
being said, when a person has dietary issues taken with this, it's usually
22:23
related to insulin resistance that gets Listen trouble because when you're born and you're normal
22:30
and you have no instular resistance, you can eat up to ninety grams of
22:33
carbs on a four hour basis and you can get away with it. But
22:36
once you become insulent resistant, that carbon tolerance goes down from ninety to twenty
22:41
five grams every four hours. And so when a person's trying to eat under
22:45
twenty five grams of carbs are I have to really concentrate on it because that's
22:48
not easy. And so most of the patients that are instant resistant don't know
22:52
they're resistance. There's a large growing number of patients with obesi and obest Central
22:59
obestia comes from eating excess carbohydrates beyond your tolerance. So your carbohydrate tolers was
23:04
it ninety grams when you started, it went to fort went down to twenty
23:07
five grams, and then you start flipping that switch over and over. Every
23:11
time you flip that switch, your liver is making fat and inflammatory factors for
23:15
four days. So if we missed today, it would be essentially Monday before
23:21
the liver stopped making fat and inflammation. And what we just said, it's
23:25
inflammation that brings the fat in. So the reason insular existence and diabetes is
23:30
such a big deal is because it's the perfect storm. Every time you over
23:33
eat carbs, you make both. If you're making a lot of inflammation and
23:37
you're making a lot of bad fats natural safety, you're going to have cardiovasturs
23:41
he is occurring from it. So we have to teach the populations that carbohydrates
23:45
are essentially a poison. As you get older and you commence resistance, Now
23:51
you say, well, how many people are into resistant America or there's some
23:53
considerations that approximately seventy percent of people over the age of twenty have some degree
23:57
of insular resistance. There's over nine hundred genes in a body that relate to
24:03
that. How do you tell I mean, how do you measure insulin resistance?
24:07
I mean, obviously glucose and insulin our blood test that you can take.
24:12
Look, is there anything I know? The HC what's it called?
24:17
HC two? That test? And then there's something else I heard h O
24:29
M A. But go ahead, I'm answering the question for you. Go
24:32
ahead and tell us what are the ways that you determine because look, sixty
24:36
eight percent our population is obese. That's that's that's where we're starting with a
24:41
huge problem. The majority of doctors are doing it in an old school fashion
24:47
or picking up on its resist and that's why we're picking it up late.
24:49
If you use the hema glowing A ONEC is your main mean of detecting who's
24:53
pretty diabetic and not you, you're picking on patients that are lost or forty
24:59
percent of their beta cell function. We need to be doing things that pick
25:03
it up earlier. Now HDL goes down earlier, triglyshrides goes up earlier,
25:07
fasting insulin levels go up earlier. But truthfully, the number one test to
25:11
see if a person has pre diabetes and INNS resistance is called an alpha hydroxybuturate
25:18
test. And when you check an alpha hydroxybuturate you could just be an instant
25:22
resistant. A few weeks and it's already clicked in. So if that level's
25:25
elevated, you have instant resistance. But the nice thing about it is once
25:27
you see that and you get them on a low carb diet and maybe some
25:32
intermittent fasting, you can recheck that and you can see if the resistance has
25:36
gone down or gone away. And so it's a major improvement in how you
25:41
can look at this disease. But at the very least, we should be
25:42
checking things like fasting insulin levels and looking at triglyceride levels that go above ninety
25:49
and looking at HDO levels that fall below fifty, and that gives us so
25:55
much better idea of who is developed inser restance, rather than waiting for the
26:00
resistance to show up in the human goo when they and see, nobody would
26:03
play a football game and give up forty points before they ever start the game.
26:07
That would be ludicrous. But waiting for a patient at lose forty percent
26:11
or beta cell function before you do anything about the disease, it makes no
26:15
sense to me. You know, that's so interesting because a very few doctors
26:18
ever test you for alpha hydroxy buttery, right, I mean, you go
26:22
to a normal blood work and you're doing your panels and it's not even included.
26:27
So why why is that? And how can you accomplish a very mainstream
26:33
testing protocol to become mainstream to the doctor. It just takes longer. Years
26:41
ago, I used to sell stereos in my dad's store before I again,
26:45
in medical school, we learned about devices very quickly. When the new stereo
26:51
had a new widget, everybody in the country that sold these stereos and knew
26:53
about the widget three months. But medicine, the same thing takes ten to
26:59
fifteen years. Physicians have tremendous amount of regulations over how they discuss things and
27:03
what they do to disseminate information. So I think some of those regulations slow
27:08
us down in our ability to exchange information. But eventually the job does get
27:14
done. It just moves a lot slower than I would like it too.
27:17
But yes, it would be wonderful for every doctor to know about alphydroxribut rates
27:23
instantly once that became common knowledge. So here's another question that comes up all
27:30
the time. It's cholesterol. So let's take a moment because I'm sure you've
27:33
heard this before, but why could you just debump the cholesterol myth? For
27:38
all of us today, and tell us what is the real culprit for the
27:45
measurement when you're doing your blood lipid panel for measurement triglycerides though you just said
27:52
is obviously the fast insulin and glucose tolerance, but what about just absolutely choless.
28:00
Sure, why is it that so many doctors want to prescribe stating drugs
28:04
for people who have cholesterol over two hundred when actually cholesterol is protective. And
28:11
yesterday I was watching an internet show podcast and some guys that he was being
28:18
interviewed by his doctor after doing some kind of change in his diet, and
28:23
the doctor said, oh, your cholesterol is one fifty. And the doctor
28:29
didn't even go into anything else besides you know, the particle size or the
28:34
ratios. So just help us out because I think too many people are on
28:38
statins that don't even need to be on statons. So you can help us
28:44
there. Go back to what I just said earlier, cholesterol never enters a
28:48
blood vessel without an invitation. The one who controls the inflammation wins. If
28:52
a person had familial hyper libel propanemia, which I had thirty of those prices
28:56
in my practice when I had to close down your several years ago when my
29:02
wife guide him. Long story short, those patients had such bad cholesterol levels
29:06
you couldn't measure it with the machines. When you actually ran their panels,
29:10
it would say too high to read. So long, these patients had horrible
29:14
chlesterral. But as long as I control the inflammation, I could control their
29:17
disease. And the way you control inflammation, number one way is exercise aerobically.
29:22
When you aerobically exercise a patient for fifty minutes non stop, and you
29:26
get the heart rate up to the seventy percent level, that removes ninety three
29:29
percent of the inflammation for a day. It will come back tomorrow, and
29:33
then you exercise again tomorrow, but it's always coming back the next day and
29:37
the next day and the next day. So what do you do. You
29:37
exercise every day of the week. As long as I had those FH patients
29:42
exercise in seven days a week, they didn't have worsening of their disease because
29:47
basically there was no inflammation to invite the cholesterol in. Now that being said,
29:52
it's not that cholesterol doesn't play a part in PARTI of extra is ease.
29:56
If you have inflammation in the blood vessels. And you have bad fat
30:00
DLC circulating in the bud stream, it will enter the vessel and when it
30:03
enters the vessel, that is what causes plaque to be made. So there
30:07
is a place for statins in certain patients, but the emphasis has to be
30:12
shifted from just putting a person on a statin too controlling that inflammation, and
30:15
doctors need to be measuring things that measure inflammation hscrps, fibrinogen levels, plaque
30:23
two levels, milo peroxidase. These are all things that tell us about acute
30:27
and chronic stages of the inflammatory process. When we look at those inflammatory responses,
30:33
then we know a little bit more about what to do with cholesterol.
30:36
And if you just look at cholesterol itself, you were talking about particle size
30:40
and or thing that does make a difference when you actually have the majority of
30:42
your cholesterol coming from your liver, that cholesterol that LDLC contains a lot more
30:48
triglyceride than it does cholesterol. The C on the LDLC stands for cholesterol or
30:53
measuring the cholesterol within the molecule. But the problem is that molecule is shifting
30:59
in quantity. It is much much less cholesterol than it ever did before.
31:02
It has much more triclyst ride than it ever did before. So the person's
31:07
LDLC could be going down and it could be much much worse simply because you're
31:12
not paying attention to the right part of the molecule. Now, looking at
31:15
a total cholesterral is useless. You could have a high total chlesterral and be
31:19
safe. You can have a low one and be in total danger. Right,
31:22
Well, that's what I was. I was like shocked yesterday when I
31:26
heard the one fifty and I said, well, that doesn't tell you anything.
31:30
I mean, actually, maybe he was two sixty he got down to
31:33
one fifty, But what happens to the protective aspect of all that cholesterol.
31:37
It may have been that he was actually hurting himself more than helping if he
31:42
didn't have all the other measurements that you've just described. So Amy, when
31:47
you go on the road to these doctors, what is the most resistance you
31:52
get from the doctors that you know? Obviously they're not all educated to your
31:56
technology. So what do you find is the most challenging. Um The most
32:02
challenging is the device is not recovered by insurance as as of right now.
32:08
It was given a CPT code back in twenty twelve UM and it ordered for
32:15
it to be covered. Physicians cannot do any kind of cardiovascular testing for six
32:20
months after having this scan done. That's how accurate the scan is UM the
32:24
MCG device. So that's that's the main focus on a lot of practices who
32:30
just are insurance based. UM. But you know there there are physicians right
32:36
now who feel that you know, insurance is not the best way to go
32:39
and patients need to take control of their health. The test is inexpensive,
32:44
it's cost efficient for patients to have done, and UM, you know,
32:47
the physicians can do a super bill and have the patients get reimbursed through their
32:52
insurance as an outpatient diagnostic test. So that's probably the biggest, biggest problem
33:00
that I've been facing. Well. And also you could have your target market
33:04
to go to concierge medicine clinics first or doctor individual practitioners first, because you
33:12
know that they already expect the patient that you know, to pay out of
33:16
pocket. So maybe that's I mean, I know you have a huge huge
33:22
challenge because doctor Samu said it, I mean, the heart disease is the
33:28
number one killer, and why hasn't it changed? You know, why hadn't
33:32
technology? We can go to the move, but we can't cure the number
33:37
one killer of human beings in our and our very technologically given society. They
33:44
hadn't changed because we're not We don't have any devices on the market until this
33:47
device that picked up disease early. So we're always playing last minute ketchup and
33:52
that doesn't work, and especially if you're in an environment where a doctor doesn't
33:55
know how to reverse disease. Now, very few doctors in medical clinics today
34:00
are reversing disease. They're doing things to slow down progression of disease, and
34:05
so we've got to teach that model on how to reverse disease. You can
34:07
make ninety five percent blocked dirters go to eighty percent blocked dirties in a year,
34:12
and you can make that eighty percent go down to sixty percent in another
34:15
year, but you've got to know how to do that. So we have
34:16
to dissimulate that information. But getting back to what they just said, this
34:21
test cost on average about four hundred dollars per test to do. When you
34:24
do a nuclear stress test, it costs about thirty five hundred dollars just to
34:29
copay a loan of a nuclear stress test costs more than this test, and
34:32
our device actually gives you a lot more information if you would be paying on
34:37
average twenty percent for copay on that other test, which is around six hundred
34:40
dollars versus the four hundred dollars this cost on a cash basis. So I
34:45
don't really think cost is the factor, but I think it's a mindset.
34:49
I think doctors hadn't thought through this completely because they're used to running their patients
34:52
through with things that are covered on insurance and just a matter of time,
34:57
we have a CPT code that actually covers this. But Carti Baccher these skills
35:01
more than the next four causes of death put together. It takes if you
35:06
took every cancer in the world put together, and then you added the next
35:08
three causes of death with that, Carti Baccher these skills more than all those
35:13
diseases. So it's it's definitely got to be target number one. So the
35:16
fact you don't have a CPT coating you would even hesitate for one billy second
35:22
makes no sense to me, because this particular product needs to get out on
35:24
the market. It needs to be used. It's have you have you ancient?
35:30
Are you know doing work for the government as the military, And of
35:36
course now with all the myocardidis has come after the COVID pandemic, it would
35:42
seem to me that this would be a place to get your feet your foot
35:47
in the door, so to speak. How has that approach done gone?
35:52
If you've made that approach, We're talking to all those people and we're basically
35:58
talking to anyone that wants to listen. And at first it went slow because
36:02
it's new technology. It was unusual. A lot of doctors like to wait
36:07
till someone down the street hasn't tested before they do. But there's earlier doctors
36:10
out there. We're finding them. And once the earlier doctors get to use
36:15
this and they have the amount of success I know they will, it's just
36:17
going to be a matter of time this thing snowballs into major and massive use.
36:25
I've watched what the other technology that I use, the other technologies for
36:30
the most of my career, and the hands down this is up Now.
36:32
It's a disruptive technology. It basically replaces things in other markets, and that's
36:37
going to cause a little bit of a clamor, and it's it doesn't involve
36:44
radiation, it doesn't involve contrast, I could do this on a pregnant patient.
36:49
I could do this on a new doorn baby and not feel bad at
36:52
all. So it's such a safe test, it doesn't cost a lot of
36:55
money, it gives us more information. It's just a matter of where this
37:00
is going to disrupt the audience the way we've been doing things. You know,
37:05
I you know, I sorry to you know. The good thing is
37:10
that I did to test, and so I can be a very good spokesperson
37:15
for you as your host today. And one thing I want to say is
37:20
well, not only is it painless and quick, it is so thorough and
37:23
and Amy knows it. You know, my test was very very good.
37:30
And I had no idea that you know, I didn't have any hardening of
37:34
the arteries or any kind of flaque or and the elasticity and all the things.
37:38
In fact, I have to test right in front of me. And
37:40
there were things that I went over with Amy because I had no idea that
37:44
these even were things that you would want to test. So, as a
37:47
patient who is depending on a doctor, you know, when you don't have
37:53
any symptoms that you're just doing it preventatively as a part of anti a uh
38:00
that that's a whole other cohort, right, people like me who have no
38:06
history of heart disease but want to know what their condition is. And so
38:10
Amy knows that one thing it did is it gave me a big smile on
38:15
my face and a lot of sense of real relief, and that in itself
38:22
just less of stress. I mean, stress is one of the great you
38:25
know, causes of heart disease. So the fact that you could walk away
38:30
from a chest like that and not have anything to worry about at least it
38:34
was part of that you know test result to Amy, I think you told
38:37
me I was one of the top results other than your you're a CEO.
38:44
That in itself gave me such a boost that I'm sure that you know that
38:50
loss of stress, of worry and concern is something else to you know,
38:55
factor into the results. So just you know, things that I didn't know
39:01
anything about, like what is your schemia? You know, that was one
39:06
of the questions I asked Amy. And then there were all these pathological conditions
39:09
and physio pathological conditions that we're tested. So can you just give us a
39:16
little breakdown of that so people know what to expect when they do this test.
39:22
The beauty of it. It's just kind of a scoring system that goes
39:25
from zero to twenty two, and anyone can understand that zero being good in
39:30
twenty two being the worst it could possibly be. If your number is going
39:32
up from year to year, you're getting worse. If just number is going
39:37
down, it's getting better. Once your number starts crossing over four, you're
39:40
starting to get some significant disease. I anticipate this being part of a yearly
39:45
physical exam for people, and so once you get to the age of thirty,
39:50
you'll be going to get your blood drowning to being you're checked out with
39:52
your doctor and you'll get your MCG test, and when you get that multifunctional
39:55
cartogram, you'll get your score. And if your score is two point two
40:00
this year and it's two point nine next year, that means we're doing something
40:04
wrong and we can begin doing something about it before it gets bad. And
40:07
if it come back the next year, now it's one point seven, you
40:10
got better, and so apparently the adjustments you made accounted for something, and
40:15
we can avoid an awful lot of heartache down the road just by keeping an
40:19
eye on this in their early stages and fixing it. Preventative medicine is where
40:23
we need to be going if we want to actually help save lives and improve
40:27
healthcare costs and improve quality of life. It all starts with prevention, preventing
40:34
heart failure, preventing microvascular disease, and women front developing catching it early.
40:39
There's no way to catch early small vessels ease and women because they don't have
40:44
symptoms, but we can catch it with this device seeing when a person has
40:47
rhythm disturbance issues, atrial fibrillation and tricker fibrillation. The value of this is
40:57
immense, but I like that number system. I think this is going to
41:00
actually not only go into the family practice and internal medicine officers. I anticipate
41:05
this to go into ganecology offices too, because so much women health takes kicks
41:08
there and they can pick up on women as they're developing disease and begin addressing
41:13
it. So Amy, I don't when you did to test, where is
41:17
that score on my I'm looking at my test right now just so I understand
41:22
this. Where would I see that score that doctor Sam just mentioned it would
41:27
be on your third page. So you have your first page as your name,
41:30
second contents, and the third pages where your report starts. You'll see
41:36
you'll see the scan um chronological order, yeah, date, and then if
41:42
you go over it, I'll say ECG tracings which is good or marginal are
41:45
which are acceptable? And then one more over is your schemic results. And
41:50
then your score would be right there right next to it. Yeah, And
41:53
I copied this with you that, David, I think maybe I don't have
41:58
to complete I POSI you know what it says here. I'm just noticing this
42:00
on the bottom. It's just one of eight and two of eight and that's
42:05
all I have. So I'm going to come back to you next week and
42:09
try to get the full full report. I'm so sorry to take time on
42:13
our very valuable podcast, but I want to know for sure. And then
42:17
the question is do I should I be going once a year to get this
42:21
test? Yeah? Absolutely, ye. With your scores which we're very good,
42:29
you would do it once a year and so and then and you have
42:34
a list on your website of doctors who are using your protocol using the machine,
42:38
and we have testing locations. What's our authorized testing locations? You can
42:45
go to heartcarecorp dot com and find out more information. There is an info
42:51
at heartcarecorp dot com that if you have any questions or if you don't see
42:54
in a testing location there in your area, just give us an email and
42:59
we'll be happy to help you. M So, I know our guests always
43:04
get some kind of an incentive from our hope, I mean from our guess
43:08
our our viewers sometimes get incentives from our guests. And I know we didn't
43:15
really chat about this, but I'd love to know if you would give any
43:17
kind of incentive to the first time users of your protocol and what you how
43:25
people could take advantage of that. Is there anything you would like to do
43:30
with that in that respect? Yeah, we can do ten percent off of
43:34
if they use uh, you know your name with the website of this podcast,
43:38
and we can definitely take care of them. Right. So, we
43:42
have an icon on our website called Michelle Recommends. So what I'll do is
43:49
we'll just add that and say, you know, for anyone that's going to
43:52
for their first time right for their first time user to use the ageless code,
43:58
and that would they contact the company's website or is there a telephone number,
44:04
and you think is an easier way to get the benefit. The easiest
44:08
way is to go to the website heartcarecrep dot com and fill out the info
44:14
at heartcareccorp dot com and just put in their you know, the code.
44:20
Okay, the ageless code will work if they just say ageless and then they'll
44:23
okay, very good. Well, and then ten percent off of the four
44:27
hundred is that correct? Yes, so they would get forty dollars. Forty
44:31
dollars, so it's pretty darn good. Yeah, And so then they get
44:36
a chance to try it and then they but they do they have to go
44:39
through their doctor or they go to that clinic that you're certifying uses your machine,
44:45
and then that clinic has to then offer them the discount. Um.
44:51
Yeah, we would, we would set that up for them. Okay,
44:53
all right, okay, So just before we run out of time, tell
44:58
me what you see in the few What is your vision for continued expansion,
45:04
continued innovation. Where are you, like in two years, where do you
45:09
see yourselves being Hopefully it will be in at least a twenty percent of the
45:15
hospitals in the nation and I hope within twelve months will have at least one
45:21
of these devices in every state and the nation in the bigger states multiple And
45:29
for me, you know, the biggest, biggest challenge for me is to
45:32
you know, we want to take patients off of the hard transplant list and
45:37
knowing if they have metabolic, toxin or environmental diseases that we can reverse it
45:42
and you know, save lives. So in the next two years, you
45:46
know, we want to make cardiovascular disease not the number one killer for Americans
45:52
for everyone, but make it all the way at the bottom and the rest
45:55
of the diseases will go away. So we want to try to try to
46:00
focus on that saving lives a one like that at a time. I love
46:04
that. But the only thing I think it's such an integrated effort and challenge.
46:10
You know, you can do all that you're doing with these this test
46:14
and educating people to what their challenges are. Then it's up to them,
46:19
with hopefully some guidance from their physician, to then implement some of those changes
46:25
that doctor Sam and you Amy have talked about, which you know, integrating
46:30
mean diet, exercise, and stress reducing stress. And we didn't really talk
46:37
about supplements. So let's take a moment just to chat about that. Is,
46:39
are there any supplements that in addition to all these other protocols that we
46:45
just talked about that people have to comply with, which is to change their
46:50
lifestyle. If they have a heart condition, then what supplements could they take
46:54
because most allopathic doctors are not going to tell them about supplements that I know
47:00
you would. So is there anything that you know a very proactive patient could
47:07
start to do to reduce their risk of heart attack? Well, probably the
47:13
number one supplementation issue is dealing with the MTHF or genetic mutation issue, and
47:19
that would be a process where folic acid doesn't break down the metal foliate because
47:22
as you age, you lack an enzyme to make it happen. And this
47:28
makes up about fifty percent of American population, so one out of two people
47:30
have this genetic effect. And as they get to twenty five, they start
47:35
having some fatigue and they don't sleep as well, and things start getting on
47:37
the nerves, and by the age fifty they have chemical depression. When you
47:42
have that chemical depression, you search late high levels of adrenaline. If you
47:45
supplement these patients in the six seventy seven mutations in the twelve ninety mutation.
47:49
With the proper dose of el metal foliate, which is a product called the
47:52
Novo for Speed twelve that I use, it makes a huge difference. They
47:55
basically get their brain chemistry back. They start making nactical side and it stops.
48:00
Nicrotoxy is the number one chemical in the body that removes inflammation from the
48:05
blood vessels. And I keep using that word inflammation over again because the one
48:07
who controls it wins. There's a supplement that called our tarasol that it seemed
48:14
to be helpful as well, and Amy. Once you go over that one,
48:17
you like that one? Now what is that? Who makes it?
48:21
Amy? So it is from Calvary Health Science, doctor Chen He is the
48:27
scientist developer behind that our tarasol. It actually repairs the endothelial lining and it
48:32
helps a lot of the small vessel disease, which we're seeing a lot of
48:37
repair with the MCG and especially on myself. So I tried it on myself
48:43
first. I have small vessel disease and I see a huge improvement. And
48:46
not only you know the way my scores have gone down, but also they
48:52
have another vaskanofs, which is a nitric oxide supplement. You take two capsules.
48:58
It works within an hour and stays in your system for twenty four hours,
49:01
so it's constantly giving you nitric oxide. So the two are tyrosol and
49:06
basquinox is. Uh, you know, pretty strong supplements that are not harmful
49:10
to you but actually help you. And you know, if you go under
49:14
our website, we do have discounts that we give for patients as well in
49:17
individuals. So those where do those products originate? Those two Calroy Health Science,
49:25
right Roy? Are they affiliated with you in a joint venture or are
49:30
they just an independent company that you happen to like and respect. Yeah,
49:36
they're independent company which we are now venturing as a collaboration. Okay, all
49:43
right, that's really important. And what about coca can or ubiquitol because so
49:47
many of your patients, I'm sure are already on statins and most doctors don't
49:52
ever tell their patients that they need to supplement with ubiquitol or coca can if
49:58
they're taking a stat And so, do you have a ubiquinol product that you
50:01
like, not particular on the ubiquino, but on the coquten and vitamin D.
50:08
You've got to have both adequate too for a good subset of the population
50:14
to actually get away with statins. So the statins themselves are not terrible drugs.
50:20
I don't want you to think that they're horrible. They actually help save
50:23
a number of people from some disturbance. But that's right. They just need
50:28
to be used correctly, and then then the patients that struggle with them and
50:30
get my aside to see the Usually it's a coqutenish or vitamin D, and
50:35
approximately seventy percent of the United States has low or deficient vitamin D levels,
50:39
even though sunshines out there. So just because it tags in with skin and
50:44
it goes to makes the exchange environment D, it doesn't seem to be effective
50:47
in a number of patients, even when they're sun exposed. So I would
50:51
suggest that both of those things be handled. And if you're taking vitamin D
50:54
three, you need to take K two because vitamin D three can calcilum.
51:00
So K two and D three are like a kissing cousins, right, So
51:05
it's important to do together. And there are supplements that combine them, but
51:08
you can buy them. I buy them individually and just make sure that I
51:12
always take them together. So yeah, so I think that well, obviously
51:19
stress, we haven't we haven't really focused on that. You kind of touched
51:22
on it, doctor Sam. But give us a few moments about stress and
51:27
how people can reduce stress and even with the supplements that like to lower court
51:31
is all levels. If there's anything that can help people that are chronic stress.
51:37
When I realized that one out of two people have that mthf oargenic mutation,
51:42
which means one out of two people as they get over twenty five years
51:45
of age or starting to begin to feel the effects of stress in a negative
51:49
way, and by the time they're fifty, they're actually being incredibly affected by
51:52
another that's uh convinced them from making poor chemicals in their brain, including serotonin,
52:01
melotonin, dopamine, and nor open deefference of these products play central roles
52:07
in how you feel, in function and have your memory, your emotions,
52:10
and so things that used to not get on your nerves start getting on your
52:14
nerves. So just by replacing that methyl fola will help. Now, there's
52:19
about eight percent of the country that has sleep disorders, and so things like
52:22
sleep apnea taking care of that would help. There are more narcoleptics out there
52:27
than we ever diagnosed. But these are sleep disorders that need be fixed.
52:30
But if you add the MTHFR plus the sleep apnea, plus the narcilepsy and
52:35
the old hodgepris, that's about sixty percent of the country that struggles with sleep.
52:39
And the whole purpose of sleep is to make chemicals. You're tired at
52:43
the end of the day because you have used up your chemicals and that olympic
52:45
system your brain. You you're supposed to manufacture those chemicals in deep sleep and
52:51
then wake up the next day refreshment. So many people go to bed tired
52:53
and they wake up tired because they don't have the ability to make the chemicals.
52:58
You have five stages to sleep, one, two, three, four
53:00
rim you make your brain chemicals, and stage three and four will If you
53:05
don't spend any time in stage three or four, or if you don't break
53:07
down polic acid into metal folate, you just can't make those chemicals. Things
53:13
like vibin D effects serotonin, So there's some other contributory factors. But if
53:17
a person was taking care of their vibin D level and getting it between fifty
53:21
and one hundred and they were taking care of their metal folate by checking their
53:23
mthfur status. Everyone in America should have an MTHF genetic mutation. Since one
53:30
out of two people have it. It just makes no sense not to get
53:31
the test. It's not that expensive. Yeah, addresses it. Well,
53:37
that's a new finding. It's not a mainstream test though, you know.
53:40
You mean when you go to your lab requisition. If you look at most
53:45
people's lab lab requisitions, mthfire is not on there. So the doctor right
53:57
sometimes. This was only discovered in two thousand and five, and we really
54:01
didn't know how to treat it till twenty eleven. It was the first effectively
54:07
treated the psychiatric worldness, which is why psychiatrists would be more likely to order
54:10
this test than primary care doctors. But eventually we'll get this across the primary
54:15
care doctors. It also affects coagulations, so it will be doctors are familiar
54:20
with it in the coagulation since it affects the ability of a woman to get
54:22
pregnant because miscarriages are recurrently occurring because they're MTGF far and they have age with
54:29
them by the time you get past the age of twenty five and on end
54:31
to thirty. If you have this MTHF for our genetic mutation, you can
54:36
have recurrent miscarriages because you don't make the spinal then one of my trains the
54:45
neural tube properly without the properties we always knew about folic acid. But if
54:50
a person can't break folic acid down, you could take a boatload and pulic
54:52
gas, it won't work. So these female patients that don't have methyl folate
54:57
in their system because that mutation have struggles with however, So there's so many
55:00
reasons to be ticking right. Thank you for this because honestly, this is
55:05
not a mainstream awareness and you created something or stated something today that I think
55:10
we'll open the eyes of a lot of people to you know, basically,
55:15
you have to be your own doctor and you have to educate your doctor,
55:17
two things that you want. And I think we do this if we're all
55:22
proactive and research and look at you know, different situations than uh and today
55:29
with the internet, it's very well not always accurate, but it's certainly a
55:32
method to go start finding some statements that of with scientific research behind it that
55:43
would help to or to meet people like you and Amy. Right, but
55:47
you know. I just before we say goodbye, I want to just mention
55:51
melotonin because you did touch on it, and I think melotonin was underrated of
55:57
all supplements. So would you agree that melotonin is a supplement to add to
56:02
your protocol most completely, especially for the fifty percent They have empty j far
56:08
issues and they struggle with manufacturing that product. They can't turn their brain off
56:14
at night and sleep. So lower doses tend to be safe and effect.
56:17
If you get too high, you can actually have some rebound effects, so
56:21
just have to be careful how you dose it, all right, So it's
56:23
three milligrams, Okay, two and three milligrams are proper doses, right,
56:29
That's what I use. Yeah, Well, I just want to say thank
56:32
you so much. This has been amazingly helpful, and you're both so knowledgeable
56:37
and and just so credible and everything that you're doing. It's it's really a
56:44
great, great effort to save humanity and from the number one killer of humanity.
56:52
And I just want you to know that all of us are cheering for
56:55
you to continue your journey and really the best of luck in this and I
57:02
will be talking to you next week. Gave me about getting this UM test
57:07
result in a more complete way, so I know to tell my doctor next
57:14
next time we do our test that I want to see if she has that
57:17
accreditation with you. So thank you again for taking the time for us today
57:24
and for all of you. Goodbye, have a beautiful weekend, and we'll
57:30
see you again in Angels and Timeless in two weeks. Thank you, bye bye.