Latest / Wake Up with Miya / Episode 200. Cancer, Fever & Chromosomal Imbalance | David Rasnick

Transcript

Episode 200. Cancer, Fever & Chromosomal Imbalance | David Rasnick · Wake Up with Miya

0:00
Speaker 1: So this is decades after Theodore Bovari's theory, and then
0:03
Speaker 1: everybody got all excited worked up the genes DNA. Now
0:07
Speaker 1: we can make synthetic molecules and we can go after
0:10
Speaker 1: the gene. Then the gene mutation theory of cancer was
0:14
Speaker 1: all the rage that mutations of a certain genes caused cancer. Well,
0:18
Speaker 1: it turns out that's completely wrong. There are no such
0:20
Speaker 1: thing as cancer causing genes, not to this day. Even
0:23
Speaker 1: many of the leading cancer researchers have acknowledged it over
0:26
Speaker 1: the past kingdom, but they don't cite our work at
0:29
Speaker 1: all anymore. All right, So, anyway, at this coffee shop
0:33
Speaker 1: right before Peter, days before he went on his journey
0:36
Speaker 1: to Germany, I said, Peter, what are you going to
0:38
Speaker 1: be doing? He said, I'm going to see if Bovari's
0:41
Speaker 1: theory of cancer was wrong. And I worked with about
0:43
Speaker 1: twenty different enzymes in my career. Tissue destroying insis making
0:47
Speaker 1: inhibitors for them, you know, and we have about twenty
0:50
Speaker 1: thousand genes. So I was trying to get in my head, Okay,
0:53
Speaker 1: twenty thousand, how do you deal with that? And I said, Peter,
0:55
Speaker 1: is it just numbers?
0:56
Speaker 2: And he said yes, I had a fascinating conversation with
1:05
Speaker 2: David Resnik, and we covered some ideas that challenge much
1:10
Speaker 2: of what we've been told about cancer and cancer research.
1:14
Speaker 2: But before we get started, don't forget to subscribe Hit
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1:29
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1:33
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1:39
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1:47
Speaker 1: Now.
1:47
Speaker 2: If you want to be a guest on the show,
1:49
Speaker 2: or you want to suggest a guest on the show,
1:51
Speaker 2: I'm always looking for interesting guests who can speak on
1:54
Speaker 2: topics such as human trafficking, the paranormal, occult symbolism, hidden history,
2:00
Speaker 2: spiritual warfare. If that sounds like you, or if you'd
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Speaker 2: like to recommend someone for the show, please email me.
2:06
Speaker 2: I do have my email down below in the description. Aloha,
2:10
Speaker 2: and welcome to another show, guys. Today I'm joined by
2:13
Speaker 2: David William Resnik, a chemist, researcher, and author of The
2:17
Speaker 2: Outsider's Advantage, A Personal Odyssey into the Essence of Cancer.
2:23
Speaker 2: In this conversation, we discussed his decades of research into
2:26
Speaker 2: cancer and why he believes cancer may be fundamentally a
2:30
Speaker 2: disease of chromosomal imbalance rather than a gene mutation. We
2:35
Speaker 2: talk about the history of cancer research, spontaneous remission, the
2:38
Speaker 2: role of fevers, and the challenging questions along held of
2:44
Speaker 2: scientific assumptions. Now, I hope you enjoy this. Let's bring
2:51
Speaker 2: David Resnik into this conversation. Andre the show. Welcome to
2:56
Speaker 2: the show. Now, you're a chemist, a researcher, and author
3:00
Speaker 2: of The Outsider's Advantage, A Personal Odyssey into the Essence
3:04
Speaker 2: of Cancer. In your book, you challenge some of the
3:08
Speaker 2: most widely accepted ideas about cancer. So my question to
3:13
Speaker 2: you is what led you to question the conventional understanding
3:17
Speaker 2: of cancer in the first place.
3:20
Speaker 1: Well, when I joined up with Professor Peter Dewsburg in
3:24
Speaker 1: nineteen ninety six and UC Berkeley, California, I left the
3:28
Speaker 1: pharmaceutical industry that year diod I set up with it
3:33
Speaker 1: and I went across the bay to support Peter Dusburg
3:38
Speaker 1: and his battle against Anthony Fauci and the aide stuff
3:44
Speaker 1: about the DHIV stuff. Peter Dewsburg, the world authority on retrovirus,
3:48
Speaker 1: has published an article about Robert Gallows so called HIV
3:54
Speaker 1: virus that he actually stole from Montaigner back in nineteen
3:59
Speaker 1: four eighty four. And Peter is the word of authority
4:03
Speaker 1: on those kind of viruses, and he published an invited
4:06
Speaker 1: article in nineteen eighty six in Cancer Research was like
4:11
Speaker 1: a legal document, point for point for points for point,
4:13
Speaker 1: how retroviruses in general and HIV in particular cannot cause
4:18
Speaker 1: any disease whatever, and that nobody has to worry about
4:21
Speaker 1: any of that stuff. And peters a number of the
4:24
Speaker 1: National Animal Sciences. He had the outstanding investigators grant that
4:29
Speaker 1: Robert Anthony Schouse he killed right after Peter's article came out.
4:34
Speaker 1: And you know, they couldn't fire him because he was
4:38
Speaker 1: a full professor, but they wouldn't lead him teach graduate
4:41
Speaker 1: students anymore, and they made his life miserable for this
4:45
Speaker 1: all the time he was still there. But anyway, Peter
4:49
Speaker 1: had extraordinaries sense of humor. I never saw the guy
4:52
Speaker 1: do anything. He just went with the float. And it
4:56
Speaker 1: was a blessing in disguise about what happened to him,
4:59
Speaker 1: for him and me both because now he was free
5:03
Speaker 1: to do what he wanted to. And he went on
5:05
Speaker 1: his first sabbatical leave in the fall of nineteen ninety six.
5:10
Speaker 1: He went to Germany to We met at a coffee
5:14
Speaker 1: shop a couple of days right before he left to
5:16
Speaker 1: go to Europe, and I asked, Peter, what are you
5:19
Speaker 1: going to do while you're there, and he said, I'm
5:22
Speaker 1: going to looked into Theodore Bovari's chromosomal Imbalanced Theory of
5:28
Speaker 1: Cancer all right, which was published in nineteen fourteen. That's
5:31
Speaker 1: a long time ago, yes, And I got Bovari's book
5:35
Speaker 1: on my website under the cancer tap in fifty nine pages.
5:39
Speaker 1: People click on it, and it's written in England. Anybody
5:42
Speaker 1: can understand it, understands England. And unfortunately Bovari died in
5:46
Speaker 1: nineteen fifteen, and of course World War One started in
5:50
Speaker 1: nineteen fourteen and he was in Germany, so his death
5:54
Speaker 1: two World wars basically stopped his theory from progressing about
6:01
Speaker 1: how unbounced crimosomes caused cancer because of that. But there's
6:05
Speaker 1: several other people who independently came up with the same
6:08
Speaker 1: thing over the years, including George Paperennicolau of pap spear thing.
6:13
Speaker 1: He didn't come up with a theory, but he came
6:15
Speaker 1: up with a way to diagnose cancer, and even an
6:19
Speaker 1: automated machine, but that was never allowed to take off.
6:23
Speaker 1: And so anyway, and then in nineteen fifty three, Watson
6:27
Speaker 1: and Criek came up with the structure of DNA double
6:30
Speaker 1: helical structure of DNA. So this is decades after Theodore
6:34
Speaker 1: Bovari's theory. And then everybody got all excited worked up.
6:38
Speaker 1: Oh the genes DNA. Now we can make synthetic molecules. Ever,
6:42
Speaker 1: we can go after the genes. Then the gene mutation
6:45
Speaker 1: theory of cancer was all the rage that mutations of
6:49
Speaker 1: the certain genes caused cancer. Well, it turns out that's
6:52
Speaker 1: completely wrong. There are no such thing as cancer causing genes,
6:55
Speaker 1: not to this day. Even many of the leading cancer
6:58
Speaker 1: researchers have acknowledged it over the past Kingdom, but they
7:02
Speaker 1: don't cite our work at all anymore. All right, So anyway,
7:08
Speaker 1: at this coffee shop right before Peter, days before he
7:11
Speaker 1: went on his journey to Germany. I said, Peter, what
7:15
Speaker 1: are you going to be doing? He said, I'm going
7:16
Speaker 1: to see if a Very's theory of cancer was right.
7:20
Speaker 1: And I worked with about twenty different enzymes in my career,
7:24
Speaker 1: tissue to strying ends. I was making inhibitors for them,
7:27
Speaker 1: you know, and we have about twenty thousand genes. So
7:31
Speaker 1: I was trying to get in my head, Okay, twenty thousand,
7:33
Speaker 1: how do you deal with that? And I said, Peter,
7:34
Speaker 1: is it just numbers? And he said yes. So he
7:38
Speaker 1: went to Germany and did his started doing his experimental
7:42
Speaker 1: work to look into the chromosome of balance theory. And
7:47
Speaker 1: I went to the library and I studied, studied and studied,
7:51
Speaker 1: and I learned a lot about chromosomes, about chromosome of diseases,
7:55
Speaker 1: think about like down syndrome and things like that, and
7:57
Speaker 1: a various work. I looked all it, all this this
8:00
Speaker 1: is in my book, by the way, and then I
8:04
Speaker 1: started looking at this and then I got it, and
8:07
Speaker 1: then I published a paper. I think it was in
8:11
Speaker 1: nineteen ninety seven, something like that. Nineteen ninety eight, my
8:15
Speaker 1: first paper on chromosomal and balanced theory of cancer. Anybody
8:18
Speaker 1: all right? Published three of them total, published two books
8:22
Speaker 1: on it. Of Peter and I published a number of
8:26
Speaker 1: scientific articles papers on the chromosomal a balanced theorio of cancer,
8:31
Speaker 1: and we had three very successful international conferences on the
8:35
Speaker 1: chromosomal a bounced serio of cancer. But it went completely
8:39
Speaker 1: against the mainstream view that key mutations are caused cancer.
8:45
Speaker 1: So we have been shut down in silenced for the
8:48
Speaker 1: past twenty years at least. But COVID was another blessing,
8:54
Speaker 1: Like having Peter having his funds cut off was a
8:57
Speaker 1: blessing for cancer research because he and I I got
9:00
Speaker 1: to figure out and prove Theudobo area's theory of cancer.
9:03
Speaker 1: We proved it, and we've written about it, published it
9:06
Speaker 1: and had these conferences and everything. And now the mainstream
9:10
Speaker 1: guys I have known now, like I say, for a
9:13
Speaker 1: decade that they could not. They've looked at so many
9:15
Speaker 1: gene gene theories and hypotheses and stuff, and thousands, tens
9:20
Speaker 1: of twenty thousands of these molecules that they've synthesized, some
9:24
Speaker 1: of them they used put in animals whatever, see if
9:26
Speaker 1: they work. None were and they admit to this day
9:29
Speaker 1: that they do not have any even a single gene
9:32
Speaker 1: mutation that causes cancer, you know, but they stay totally
9:36
Speaker 1: silent about it. But now we are starting, like with you,
9:42
Speaker 1: with other people around the world that I've been talking
9:44
Speaker 1: to because of the COVID thing. We are out in
9:47
Speaker 1: the world now, people, we are sharing stuff that all
9:52
Speaker 1: kinds of things that people coming from all walks of
9:55
Speaker 1: lives that they're experts in and now we're getting to
9:57
Speaker 1: share it, you know, and then like YouTube and stuff
10:01
Speaker 1: like that, you know, shut it down. I'll give you
10:04
Speaker 1: a chance to give me a breather.
10:08
Speaker 2: So, chromosomal imbalance, that's the driving force behind cancer. If
10:15
Speaker 2: you can explain a little bit about what that is
10:17
Speaker 2: and why do you think gene mutation theory became the
10:22
Speaker 2: dominant explanation.
10:25
Speaker 1: That's the easy part listening there. It's because it was good.
10:29
Speaker 1: You know. It's a trillion dollar industry. Cancer is a
10:31
Speaker 1: trillion dollar industry, all right, And it's molecules, synthetic molecules.
10:36
Speaker 1: I'm organic chemists. They're like tinker toys. You can make
10:40
Speaker 1: these things thousands, you know, and that's the pharmacy Company's
10:44
Speaker 1: been doing that as long as they've been in existence,
10:47
Speaker 1: and then they can market them, sell them out there,
10:50
Speaker 1: do it really really quick and make a lot of money,
10:53
Speaker 1: and they are uh, it's but unfortunately a lot of
10:58
Speaker 1: people don't know this. They're drugs, therapy for cancer, a
11:01
Speaker 1: lot of these things. So that is now the sixth
11:04
Speaker 1: leading cause of cancer. The therapies are. And the thing, yes,
11:11
Speaker 1: is that the chrominosombial balance theory it is correct. It
11:16
Speaker 1: means their whole trillion dollar approach to cancer is wrong.
11:19
Speaker 1: He chose it overnight like that. If it was made public,
11:21
Speaker 1: it's finished. It's done all right, and a lot. And
11:25
Speaker 1: the other thing is the cancer can be treated and cured.
11:28
Speaker 1: And this has been known for over a century. And
11:31
Speaker 1: in my book, William Coley medical doctor in New York
11:36
Speaker 1: in the late nineteenth century early twentieth century, he came
11:40
Speaker 1: up with what's called now Coley's toxins or mixed bacterial toxins.
11:44
Speaker 1: And I tell this story in my book. There was
11:48
Speaker 1: a fellow hand of sarcomo on his cheek and they
11:52
Speaker 1: did numerous surgeries on it but could never cure it,
11:55
Speaker 1: make it go away. And one day this guy had
11:58
Speaker 1: two ballots of very back material infections, very high spiking
12:01
Speaker 1: fevers and his tenser went away in hours and it
12:07
Speaker 1: did not return and he left that Bavari didn't see it.
12:11
Speaker 1: Then he just heard about this story. So then he
12:14
Speaker 1: spent a few years, several years, I think this might
12:17
Speaker 1: have been as five to seven years searching for this guy.
12:20
Speaker 1: And he finally found it and he was still alive,
12:23
Speaker 1: and he looked absolutely normal. He had a scarleless cheek
12:27
Speaker 1: from where they did surgery to try to remove it
12:30
Speaker 1: the timber, but he was totally healthy. And then then
12:34
Speaker 1: Bavaria went back and then he started treating cancer patients
12:38
Speaker 1: with these bacterial toxins and he came up with two
12:43
Speaker 1: of them. Eventually that led to what they call Col's
12:45
Speaker 1: toxins and his To this day, they're using this method
12:49
Speaker 1: mixed bacterial toxins in Europe and in Mexico and other places.
12:55
Speaker 1: Not in the USA though, unless it's in combination with
12:58
Speaker 1: chemos air, which then it's like putting your foot on
13:01
Speaker 1: the gas and the brakes at the same time. And
13:05
Speaker 1: his daughter went around the world and collected the data
13:09
Speaker 1: of all the people who've been using his talks, and
13:11
Speaker 1: she published a book on it. And the guess what
13:15
Speaker 1: the five year survival rate of chemotherapy is of cancer
13:20
Speaker 1: in the United States two two to three percent below
13:27
Speaker 1: five percent. Coley's tocsin is fifty percent fifty percent, And
13:34
Speaker 1: there are other ones too. There are other ways of
13:38
Speaker 1: going after this. There's just been cheap, inexpensive ways that work,
13:43
Speaker 1: but the trillion dollar industry shuts it down. In fact,
13:49
Speaker 1: you know, I don't know if you're your interview with
13:51
Speaker 1: me is he didn't going to bill out on the
13:52
Speaker 1: air from what I'm telling you, because this is what
13:56
Speaker 1: I was talking about. So, I mean, this is the story.
14:03
Speaker 1: Everything else is just glitter. So I will let you
14:06
Speaker 1: ask me something else, or tell me to shut up.
14:09
Speaker 2: No, never tell you to shut up. No, I want
14:11
Speaker 2: to hear this. What is this coly toxin? How do
14:14
Speaker 2: you get it?
14:15
Speaker 1: They're they're bacterial toxics like the ones I remember Serrationian
14:20
Speaker 1: Mark Sessions, I remember when I was in college. That's
14:23
Speaker 1: an orange like bacterium. And then there's uh I forget
14:27
Speaker 1: something back, Silla's pyogens or something for I fever. These
14:30
Speaker 1: are just two there and they're killed, they're not alive,
14:34
Speaker 1: and they inject them in people and it causes a
14:36
Speaker 1: high spiking beef. You see, very very high spiking fever
14:41
Speaker 1: has been linked to curing cancer for god who knows
14:46
Speaker 1: how many centuries. This has been known for over a
14:50
Speaker 1: thousand years. It's been documented, all right, And some places
14:55
Speaker 1: I was wondering, you know, they have places like the
14:57
Speaker 1: American Indians and the Scandinavians, they have these sweat lodges,
15:02
Speaker 1: you know, they do on a regular basis, you know.
15:05
Speaker 1: And my guess is that those are therapeutic things that
15:09
Speaker 1: also prevented things, but not just for cancer, probably a
15:14
Speaker 1: lot of things, you know, And I don't know that
15:17
Speaker 1: that's that's my hypothesis that those things are probably effective,
15:22
Speaker 1: but there are other things out there. People. This is
15:24
Speaker 1: ivermictan thing. Let me tell you something that's this has
15:30
Speaker 1: been known since the early since nineteen thirty six. It
15:34
Speaker 1: was auto Warburg had discovered what's now called Warburg effect
15:39
Speaker 1: that cancers, all cancers, no exception, all cancers have an
15:43
Speaker 1: absolute dependence requirement on what's called glycolosis. Glycolosis is a
15:49
Speaker 1: method of like making the fuel for the cell to
15:54
Speaker 1: live and divide. It turns glucose into what's called ATP
16:00
Speaker 1: and it does it two hundred times faster than the
16:03
Speaker 1: normal cells mitochondria that are in the cell, although they're
16:08
Speaker 1: much more efficient. But I go into the book and
16:11
Speaker 1: explain all that detail. There's too technical to get into
16:14
Speaker 1: you right now. But the main thing is is that
16:22
Speaker 1: cancer has an absolute dependency on glycolysts. It's called it anaerobic.
16:27
Speaker 1: In other words, it means in the absence of oxygen,
16:31
Speaker 1: and cancers that solid cancers, especially have what's called hypoxia
16:35
Speaker 1: low oxygen content, and right in the heart of really
16:39
Speaker 1: the really meat of the cancer oxygen levels go wait wait,
16:43
Speaker 1: wait down, but the cancers really thrive in there. And
16:49
Speaker 1: the other thing that the like ivermectin and some of
16:52
Speaker 1: these other parasitic drugs that people use, and people think, oh,
16:56
Speaker 1: because they kill parasites, cancer must be parasites must cause cancer,
17:02
Speaker 1: you know, because the same drugs will cure both. But
17:05
Speaker 1: they didn't really look into it deep enough. It really
17:09
Speaker 1: does cure killed parasites, but the parasites live also on glycolysis,
17:15
Speaker 1: turning glucose into ATP. The tumors turn glucose into ATP,
17:22
Speaker 1: and then that's why the same drugs molecules kill the
17:28
Speaker 1: parasites and also kill tumors because the tumors have an
17:33
Speaker 1: absolute requirement for glycolosis, just like the parasites. Is it
17:38
Speaker 1: the sugar glycolysis is the turning sugar into energy. That's
17:45
Speaker 1: what's the process of turning glucose, specifically glue close close
17:51
Speaker 1: into ATP. It's molecular reactions that go down there. You know,
17:56
Speaker 1: it's not very efficient, but it's very very fast two
18:00
Speaker 1: D about two hundred times faster than the normal mitochondrial
18:05
Speaker 1: production in normal healthy cells.
18:09
Speaker 2: Wow.
18:10
Speaker 1: Yeah, it's amazing story.
18:12
Speaker 2: It is absolutely One of your themes in your book
18:15
Speaker 2: is that cancer develops over many years, sometimes decades.
18:20
Speaker 1: So what is.
18:21
Speaker 2: Happening inside the body during this process?
18:25
Speaker 1: Well, now we're getting into what I really put in
18:28
Speaker 1: my pepitis. All this details are in my book. I
18:32
Speaker 1: mentioned chromosomes and the David Hasman back in the late
18:38
Speaker 1: eighteen hundreds, he looked at about ten thousand or more
18:42
Speaker 1: cancer cells. They had good microscopes in those days. He
18:46
Speaker 1: took pictures of what he saw. He also drew a
18:48
Speaker 1: lot of little pictures by hand drawing and normal cells.
18:54
Speaker 1: We humans have twenty three chromosomes coming from our mother,
18:58
Speaker 1: twenty three chromosomes coming from Finally, for a total of
19:01
Speaker 1: forty six. We have about twenty to forty trillion cells
19:06
Speaker 1: in our body, all right, and they all contain the
19:10
Speaker 1: balance set twenty three plus twenty three, you say, for
19:13
Speaker 1: forty stings, all right, and our body, our body undergoes
19:17
Speaker 1: about twenty thousand trillion cell divisions in a lifetime. Normal
19:22
Speaker 1: cells keep dividing and they still keep that balanced. That's
19:27
Speaker 1: called euploidy, balanced set of forty six chromosomes, all right.
19:31
Speaker 1: No cancer cell has a balanced set of these chromosomes.
19:35
Speaker 1: None does. Not one does. And that's called an uploit,
19:40
Speaker 1: meaning it's not balance, all right. And that's what David
19:44
Speaker 1: Hanseman saw with his microscope. Every one of the cancer
19:49
Speaker 1: cells over ten thousand that he looked at, No, not
19:52
Speaker 1: two of them were the same. Not two of them
19:54
Speaker 1: have the same combinations. Everyone was different. Remember I said,
19:57
Speaker 1: the normal cells, all those trillions, thousands of I mean
20:01
Speaker 1: thousands of trillions of cell divisions, they stay normal. You know, percentages? People,
20:06
Speaker 1: where's anybody come from? All right? The unbalanced chromosomes caurse, entergy,
20:10
Speaker 1: cigarette smoke, asbestos, anything that goes in there and disrupts
20:14
Speaker 1: that balance separation of chromosomes, like asbestos. It's a mechanical,
20:19
Speaker 1: a physical disruptor of chromosome separation. The little fibers that
20:24
Speaker 1: pull your chromosomes apart. When the cells divide, they stick
20:27
Speaker 1: into the fibers, and then you have one or too
20:30
Speaker 1: many two chromosomes go into that cell and the too
20:33
Speaker 1: few going into the other way. Now you started unbalanced cells,
20:37
Speaker 1: unbalanced chrimosomes in your cells, and they're damaged as they
20:43
Speaker 1: try to divide. They try to put these things back
20:46
Speaker 1: in balance, and usually they always go away, almost all
20:49
Speaker 1: almost all these go away, you know. But the more
20:53
Speaker 1: for example of cigarette smoking. You know, if you most
20:58
Speaker 1: of the people had lung cancer, throat and neck cancer
21:03
Speaker 1: had smoked, you know, the chronic users cigarettes, but only
21:08
Speaker 1: uh fewer than ten percent of the let me say, yes,
21:14
Speaker 1: about ninety percent of all those were cigarette smokings. They
21:17
Speaker 1: got that, okay, But the public didn't. The people who
21:22
Speaker 1: did not smoke did not get those stuff. And uh,
21:26
Speaker 1: you know, so only ninety percent of the people smoked
21:30
Speaker 1: did not get the cancer, right, but ninety percent of
21:33
Speaker 1: the people had the cancer smoked. To make that to clear,
21:37
Speaker 1: make that clear, all right, So there was a link there.
21:40
Speaker 1: So even using the carcinogens of cigarette smoking. You know,
21:44
Speaker 1: the vast majority of you are going to win that
21:46
Speaker 1: lottery and not get cancer. But still the people that
21:49
Speaker 1: got the most of the people got the head, neck,
21:51
Speaker 1: throat cancer, and lung cancer was cigarette smoking. The same
21:55
Speaker 1: thing was asbestos with glyph of sad. If you're working
21:58
Speaker 1: out in the fields, I mean, if the more doses
22:02
Speaker 1: of carcinergen, it's like the lottery are playing pokers up.
22:06
Speaker 1: The more bets you're taking that you're going to lose.
22:09
Speaker 1: You know, at some point you're going to generate enough
22:12
Speaker 1: cancer cells in your body and damage your own health
22:16
Speaker 1: in the process. You know. Such now it gives the
22:19
Speaker 1: cancer cells an opportunity to thrive, to survive and thrive.
22:24
Speaker 1: And normally before COVID, before these injections came out, normally
22:30
Speaker 1: it took years for blood cancers to develop and decades
22:34
Speaker 1: for solid tumors to develop. And I've got those numbers
22:37
Speaker 1: in the graphs that show this in my book, so
22:40
Speaker 1: you can see the time course in there. And that's
22:43
Speaker 1: because these anuploid cells are damaged. They're there. They you
22:48
Speaker 1: have to just keep putting your gas your foot on
22:51
Speaker 1: the gas pellet to keep it going to some point
22:54
Speaker 1: where you'll get some that will survive. Some of these annuploids,
22:59
Speaker 1: the cells will even the cancer cells. The majority of
23:04
Speaker 1: the cancer cells in a solid tumor that is thriving
23:07
Speaker 1: and expanding. Most of the cells are dying still because
23:10
Speaker 1: they're damaged cells, but they keep new populations that keep
23:14
Speaker 1: dividing new populations. They have enough there to keep fueling
23:18
Speaker 1: the cancer and spread and everything. It takes decades, unfortunately.
23:25
Speaker 1: You know, before COVID, cancer was the third leading cause
23:29
Speaker 1: of death in the United States, third before COVID. Since
23:33
Speaker 1: the injection started sarscobe injections in twenty twenty one, now
23:38
Speaker 1: it has moved up to the second leading cause of
23:41
Speaker 1: cancer or death is cancer second leading cause of death.
23:47
Speaker 1: And then there was this phenomenon that you know, as
23:51
Speaker 1: a cancer researcher and oncologists like Macus and William Macis
23:56
Speaker 1: in Canada and other places, you know, the air of
23:58
Speaker 1: this expression promoke answer. None of us tensor researchers or
24:02
Speaker 1: physicians ever heard of this phrase before. You know, it
24:06
Speaker 1: was what the public was seeing with their own eyes,
24:08
Speaker 1: in their own family and their own neighborhood. It was
24:12
Speaker 1: that obvious of what was going on. So, being a
24:16
Speaker 1: Kisser researcher before my book got published, I said, I
24:19
Speaker 1: got to write a little sub sub chapter on this
24:23
Speaker 1: turbo cancer business, you know. And I looked it up,
24:27
Speaker 1: and of course it's not a technical term. You don't
24:29
Speaker 1: find it in the scientific literature. So I was looking
24:33
Speaker 1: for rapid progression. And finally I saw this phrase hyper
24:37
Speaker 1: progressive disease, hyper progressive disease. So then I looked that
24:42
Speaker 1: up and I found just under three hundred papers that
24:46
Speaker 1: mention hyper progressive disease in the scientific literature, in the
24:52
Speaker 1: title or in the abstract. So then I go back
24:55
Speaker 1: and see what the earliest one was. It was twenty seventeen.
24:58
Speaker 1: That was the first mintion of hyper progressive disease in
25:02
Speaker 1: the scientific literature. And the it happened it was in
25:07
Speaker 1: these reports, these cancer study, cancer treatment studies. I don't
25:11
Speaker 1: know if you've heard of immune checkpoint in hippiters. That's
25:14
Speaker 1: all the rage in cancer research right now. In therapies,
25:17
Speaker 1: immune checkpoint and nippers. They were treating these people who
25:21
Speaker 1: had cancer, they already had cancer, they were cancer patients,
25:25
Speaker 1: and they were treating them with these new drugs called
25:27
Speaker 1: immune checkpoint killers and in some papers reported maybe two percent.
25:34
Speaker 1: Then there'd be like ten percent, seventy percent of the
25:38
Speaker 1: patients that received these immune checkpoint hippters got these really
25:43
Speaker 1: rapidly developing cancers and die and they call them hyper
25:47
Speaker 1: progressive disease. All right, And just so you see the
25:52
Speaker 1: story here. As a researcher, you know, I keep looking
25:56
Speaker 1: and seeing looking up. This hyper progressive disease is almost
26:00
Speaker 1: exclusively reports of other positions mentioning it. You know, like
26:06
Speaker 1: I had so many patients that there's so many patients
26:08
Speaker 1: of that, and you don't find the study of people
26:11
Speaker 1: who are trying to find out what's causing it. There
26:14
Speaker 1: are a few people, fortunately that did look into it,
26:18
Speaker 1: not a whole lot that were least allowed to get published.
26:23
Speaker 1: And it turns out that they found this. Our inning
26:27
Speaker 1: system we have, our imine system has the brakes and
26:30
Speaker 1: the accelerator. You know, if you get an infection or
26:33
Speaker 1: if you need to treat something, you put on the accelerator,
26:36
Speaker 1: you know, to take care of it. Then you've got
26:38
Speaker 1: to put on the brakes to slow it down. So
26:40
Speaker 1: does it cause damage in your body? You have to
26:43
Speaker 1: really control it. Does your inning system is very powerful,
26:47
Speaker 1: very destructive if it gets out of whack. All right,
26:51
Speaker 1: And there's these the cells of these T cells, they
26:56
Speaker 1: have ig G one, i g T two, i GQ
26:58
Speaker 1: three and IgG. Don't worry about all that stuff. The
27:02
Speaker 1: thing is that the the IGT four is part of
27:05
Speaker 1: the breaking system I g E four and it has
27:08
Speaker 1: the lowest concentration of those cells in your body zero
27:13
Speaker 1: two zero point zero five percent or something like that,
27:15
Speaker 1: in your normal, normal body. All right. The people that
27:18
Speaker 1: got those I mean checkpoint hipters, their levels of IgG
27:23
Speaker 1: four went up the highest five hundred fold, five hundred fold, right,
27:28
Speaker 1: the IGT four, the braking system. Those are the ones
27:32
Speaker 1: that died real quick, all right, from the therapies. And
27:36
Speaker 1: then I looked at the people who were giving the
27:39
Speaker 1: sars Kobe two injections that came up with these hyper
27:43
Speaker 1: they call them turbo cancers. Well, there were not too
27:47
Speaker 1: many papers, but I also found some that looked into it.
27:51
Speaker 1: Guess what they had the same high, you know, extraordinary
27:55
Speaker 1: two hundred three hundred, five hundred fold elevation at IgG four.
28:00
Speaker 1: In other words, those injections are stopping the breaking system.
28:04
Speaker 1: You know that. And as allows you're you know, the
28:08
Speaker 1: one that shuts that shuts it down. I mean it's
28:12
Speaker 1: allowing it to accelerate. The breaking system is accelerating, So
28:16
Speaker 1: it's your immune system shuts down and now the cancer
28:19
Speaker 1: can take over the same thing is happening. And the
28:22
Speaker 1: people cancer patients taking the immune checkpoint inhibitors and the
28:26
Speaker 1: non cancer patients who had cancer the noah, But the
28:30
Speaker 1: cancers were already there and they gave them those injections,
28:33
Speaker 1: and then the cascer just multiplied very very fast and
28:36
Speaker 1: then and then killed these people. So that's that one.
28:40
Speaker 1: Nothing controversial there.
28:49
Speaker 2: Why why do so many cancer treatment appears to be
28:54
Speaker 2: successful at first but then stops later?
28:58
Speaker 1: Good question. Also not my book. Anuploid cells, the ones
29:04
Speaker 1: that have unbalanced chriminosomes, they are never healthy your normal
29:09
Speaker 1: cells until you poison yourself. Your normal cells are much
29:12
Speaker 1: shelterier and have a huge advantage over the cancer cells.
29:16
Speaker 1: That's why, you know, most of us have cancer don't
29:19
Speaker 1: even know it. Immune system takes care of it. Gets river. Yeah,
29:23
Speaker 1: I forgot what was I lost the track? What's thet Yeah?
29:28
Speaker 2: It So the cancer treatment seems to work at first.
29:31
Speaker 1: And then it stops right, Okay, at first, I already.
29:36
Speaker 1: The anuploid cells are damaged cells. They die on their own.
29:40
Speaker 1: They don't take much work to kill cancer cells. And
29:44
Speaker 1: so if you put in poisons in the body, it's
29:48
Speaker 1: going to kill put your cells. The chemotherapy, all the
29:52
Speaker 1: all the treatments that they use for cancer also affect
29:57
Speaker 1: your normal cells. So you have your health healthy cells
30:00
Speaker 1: up here, and you have your cancer cells here, and
30:03
Speaker 1: then you put in these poison. Doesn't matter what the
30:05
Speaker 1: poison is. The cancer cells go away really fast, but
30:09
Speaker 1: your your health is lowering too, but just much slower,
30:12
Speaker 1: but it's lower. Then they have to stop the chemo
30:15
Speaker 1: and the radiation before they kill the patient. But they
30:18
Speaker 1: can't detect any cancer cells, even though there's billions of
30:21
Speaker 1: them left, there's not enough of them to really see easily.
30:24
Speaker 1: All right, now you're down here, and then you know
30:28
Speaker 1: you're sick and everything, and you're trying to get better
30:30
Speaker 1: than you. Take you off the drugs whatever they're doing,
30:32
Speaker 1: and then munth to a year or something like that.
30:36
Speaker 1: Certainly you typically well before a year, the cancer cells
30:39
Speaker 1: start coming back because you can't kill them all. No
30:43
Speaker 1: two cancer cells are alike. There's this you know, bell
30:47
Speaker 1: shaped curve. Of you know of what, this collection of
30:51
Speaker 1: cells will be resistant of this, this collection of cells
30:54
Speaker 1: be resisted of that, and the vast majority of them
30:56
Speaker 1: will be in the middle. You can't kill all of them,
30:59
Speaker 1: and then you'll always leave a residue of the camp
31:03
Speaker 1: of cells that are resistant to just about anything. And unfortunately,
31:07
Speaker 1: now the difference of health. Here's the cancer and here's you.
31:10
Speaker 1: It's no longer like this before you were treated. That's
31:13
Speaker 1: like this, all right, And these things come back and
31:16
Speaker 1: they're resistant to any poison therapy that they give. They
31:21
Speaker 1: selected for it. They killed off all the all the
31:25
Speaker 1: answer cells that were susceptible, and they're all wimpy to
31:28
Speaker 1: begin left. But there's always a subpopulation that's resisted to
31:32
Speaker 1: whatever you want to throw at, and then it comes
31:35
Speaker 1: back with a vengeance and that you can't do anything about.
31:39
Speaker 1: Pretty much. At that point, you're ask me good questions.
31:42
Speaker 1: These are in my book, by the way, all these stories. Yes,
31:45
Speaker 1: I tried.
31:45
Speaker 2: I read your book and I try to answer. I
31:48
Speaker 2: try to ask the questions, so it ties back to
31:50
Speaker 2: your book.
31:52
Speaker 1: You appreciate it.
31:55
Speaker 2: In your book, you spend time discussing spontaneous remission. Why
32:00
Speaker 2: do you think those rare cases may hold some of
32:03
Speaker 2: the biggest clues to understanding cancer.
32:07
Speaker 1: Frankly, they only appear rare. Uh they I don't think
32:12
Speaker 1: they were so rare. Before industrialized healthcare came in, people
32:19
Speaker 1: had noticed spontaneous remissions for cancer over a thousand years.
32:24
Speaker 1: You know, they've those fevers and all these other sorts
32:26
Speaker 1: of things. You know that there's nothing new, nothing new
32:29
Speaker 1: about that. And a lot of the time, like I said,
32:32
Speaker 1: a healthy person, we probably all have cancers in us,
32:35
Speaker 1: you know, because the chemicals in the air, you know,
32:39
Speaker 1: and the radiation X rays or carcentergenic you know. And
32:43
Speaker 1: yet you're healthy bodied, you if you're a really really
32:47
Speaker 1: healthy person, Uh, the cancer cells can't really survive. The
32:52
Speaker 1: competition is just too overhall and you stay healthy. And
32:56
Speaker 1: like I say, fevers line for example, a childhood that diesels,
33:00
Speaker 1: mounts and chicken box I had them all. I'm old
33:02
Speaker 1: enough to do that. You know, they're very survivable. In fact,
33:05
Speaker 1: we kids, we loved it. We didn't have to go
33:06
Speaker 1: to school, you know. But the thing, those those things
33:10
Speaker 1: were preparing us, those fever causing diseases. Protect you and
33:16
Speaker 1: educate your body's immune system how to fight cancer. All right, now,
33:21
Speaker 1: what do we do. We don't. We don't let people
33:25
Speaker 1: we try to vaccinate people. We do not let them
33:27
Speaker 1: go through this normal process of development into a healthy
33:31
Speaker 1: into a healthy body. Not only that, we injure people
33:35
Speaker 1: with all the chemicals and all the drugs, all these
33:38
Speaker 1: artificial things that we're putting under ourselves. You know, So
33:42
Speaker 1: everything everything the healthcare industry in the United States is
33:46
Speaker 1: doing to try to prevent or treat cancer is doing
33:50
Speaker 1: just the absolute opposite.
33:53
Speaker 2: Yeah, you're right, they do. And in fact, I was
33:59
Speaker 2: going to ask you touched on it just now that
34:01
Speaker 2: you have an entire section of your book talking about
34:06
Speaker 2: the importance of fever, and most people think of fever
34:09
Speaker 2: as something that should be suppressed. In fact, when when
34:14
Speaker 2: you take your child to the doctor and they get
34:16
Speaker 2: a shot like the measles, mumps and rebella MMR shots
34:21
Speaker 2: or whatever we call it today, they will tell you,
34:24
Speaker 2: if your child gets a fever, just give them tailanof
34:27
Speaker 2: just give them advil. And so they're they're telling us
34:31
Speaker 2: to suppress that. I mean, even if they get the vaccine,
34:34
Speaker 2: we should let them have the fever. But we don't.
34:37
Speaker 2: The doctors are telling you just give them tailanof So
34:43
Speaker 2: what led you to believe that fever deserves more attention
34:47
Speaker 2: in cancer?
34:49
Speaker 1: Well, I mean so literature is there, I mean, uh,
34:53
Speaker 1: funerable area. You know what he was doing with this Knicks,
34:58
Speaker 1: bacterial toxins, the high spike fever. He's all in the
35:01
Speaker 1: spand and as cancer went away in hours, you know,
35:06
Speaker 1: and it's been reproduced around the world. There's there's no
35:11
Speaker 1: mystery here. And it was known for over a thousand
35:15
Speaker 1: years the importance of fever, you know, with things, So
35:20
Speaker 1: it's not new. The healthcare industry is rotten to the core.
35:25
Speaker 2: To listen to the rest, head over to my Patreon today,
35:29
Speaker 2: sign up for the Plus show and get the full experience. Well,
35:33
Speaker 2: I really enjoyed our conversation. Before we wrap up, I
35:36
Speaker 2: just want to thank you for taking the time for
35:38
Speaker 2: joining me today. I really enjoyed our conversation and appreciate
35:42
Speaker 2: you sharing your research and your experience and the story
35:45
Speaker 2: behind the outsider's advantage. Where can people find you and
35:50
Speaker 2: your work that you do. What is your website?
35:53
Speaker 1: It's my name, David Rasnik R D A B I
35:56
Speaker 1: D R A S N I c K forty years
36:02
Speaker 1: worth of information lots of stuff, tick download, lots of videos,
36:06
Speaker 1: lots of books, and tons and tons and tons of
36:10
Speaker 1: probably once you look at I said, I'm never one
36:12
Speaker 1: dot Eric Yes, lunch back.
36:17
Speaker 2: Well, thank you so much for spending the time with
36:20
Speaker 2: me today and for answering all my questions that I have.
36:23
Speaker 2: It's been a fascinating conversation. I'm grateful that you've joined
36:27
Speaker 2: us today. Thank you.
36:30
Speaker 1: Been a pleasure.
36:31
Speaker 2: Thanks guys for listening, and I'll see you on the
36:34
Speaker 2: next one.
36:35
Speaker 1: Huejoca cole.
36:38
Speaker 3: Bye, tell me it soon.
37:00
Speaker 4: Mak a mon. I'm going crazy, old. I know you
37:06
Speaker 4: say you're not afraid to dub it just stomping nothing,
37:13
Speaker 4: just boots. But I'm not that a way. Take them
37:19
Speaker 4: a sweet time. I don't want to be the one
37:22
Speaker 4: sur Crissa. It's uncle, make them out, I don't know,
37:29
Speaker 4: says so I'm walking ye swearing my father.
37:36
Speaker 3: Again. Tell me?
37:39
Speaker 4: Is it to soups? Uncle? Make them all going crazy?
37:45
Speaker 3: O n say shake when you walk into the room.
37:52
Speaker 4: Okay, sings straight. There's no one like you. But how
37:59
Speaker 4: in the chake it's feeling away you walk away from
38:04
Speaker 4: this tonight.
38:06
Speaker 3: I'm with you.
38:08
Speaker 4: Those are my coat break the mold. I don't use
38:12
Speaker 4: they get so, I don't like open me up, tweting
38:16
Speaker 4: my phone into lose a loco making mold. I don't
38:22
Speaker 4: know I'm saying so, I'm ok lean twitting my phone
38:28
Speaker 4: into again so many? Is it too soon? Its cocking
38:38
Speaker 4: crazy crazy crazy, I'm crazy, okay crazy coming crazy crazy
38:49
Speaker 4: over it, accona. I don't mean I'm saying, ok going
38:59
Speaker 4: crazy way. Why are you