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Zombie Cells Fuel Aging Inflammation | Science News
Scientists have uncovered how “zombie” senescent cells fuel chronic inflammation in aging: their malfunctioning mitochondria overproduce acetyl-CoA, loosening DNA to activate inflammatory genes—and simultaneously leak genetic material that triggers immune responses. This dual mechanism creates a persistent low-grade fire of inflammation tied to age-related diseases. But here’s the breakthrough: blocking acetyl-CoA in mice slashed inflammation, improved tissue health, and slowed aging—even with mitochondrial damage still present. It’s a new metabolic target for fighting age-driven decline…
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