Latest / Ageless and Timeless Podcast® / Dr. Suzanne Ferree, Health and Wellness, Longevity, Beauty, Fitness, Relationships

Transcript

Dr. Suzanne Ferree, Health and Wellness, Longevity, Beauty, Fitness, Relationships · Ageless and Timeless Podcast®

0:05
Good morning everyone. This is Michelle Hughs from Ageless and Timeless. Today I
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have a very special guest. I know I always say that, but today
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a guest that I've been wanting to meet on our podcast for a few months,
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introduced to me by another very good former podcast guest named Debbie Barber from
0:26
Vitally and So. Doctor Suzanne Foray is a functional medicine practitioner in Atlanta.
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However, she does do telemedicine consults and her specialty is actually with peptide.
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So I'm going to let Suzanne dive into the peptide subject, but she does
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a lot of other things as well, and she has just written a book.
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I think it was just released on last week, and the book is
0:56
called counter Clockwise. There it is just before we get to you and talking
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about you. Tell me how you why you gave the name of the book
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Counterclockwise. It actually was a patient who named it for me. I was
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explaining he's he's a peptide user, and I was explaining to him about the
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book and what we're trying to accomplish, and that that we've written the book,
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and I said, I'm trying to come up with a really creative name,
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and I think I'm going to call it this, and I had some
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different ideas, and he said, what about counterclockwise? And my publisher looked
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it up and I don't think there were any other books by that name,
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so it perked. It was perfect. It's the concept of trying to turn
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back time without sort of embracing aging, but also not allowing it to drag
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you down. I love that. That's perfect and no one else had it,
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which is pretty amazing too, right, right, able to find a
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name, so good for him. Good that you asked your patients or some
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suggestions, right, or he knew about the book. So let's start with
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you first. So I know you. I want our viewers to know that
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you have had quite a journey to where you are today. So why don't
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you start us from the beginning where you were a allopathic medical doctor practicing with
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you know, fifty patients a day, and then feeling like you weren't making
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a difference and wanting to do more, and then you know, as always
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happens to so many of my guests have said this that they've had an event
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occur, like in your case, it was an injury. Because you're an
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athlete, and you were looking for some answers, and so this is,
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you know what. I don't want to steal your thunder, but this is,
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you know, sort of what led you off to a different path.
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So why don't you dive in and tell us more about about you. You
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have a great memory. You must be taking memory peptides. Well, so
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I was. I've done family medicine. That's what my basic education was in
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was in family medicine. This was and then about ten years ago I had
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some patients that came to me and said, Hey, I've got this bioidentical
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hormone. Will you mind filling the prescription? And I said, oh what?
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And so I filled the prescription and the pharmacist said, you clearly don't
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know what you're doing. Can I send you to a conference? So the
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local, just my local compounder sent me to a conference and it was great.
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He was really sweet and the conference was great. It was called Get
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Out of Line, and it really just told me that there were so many
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other things out there we can do to take care of patients. Because I'd
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see over and over again these patients who were they were definitely sick, but
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they were not getting better. With conventional medicine, and their conventional labs were
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not showing anything wrong. So if I was just to look at their labs,
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I would say, well, you probably have a prozac defficiency and would
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treat them that way. But I wasn't. I didn't know a lot.
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Medical school doesn't teach just a lot of what I now know, and I
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didn't realize how much the gap between bench research and clinical medicine. There's such
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a huge gap there, and there are big mountains for the pharmaceutical companies and
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for the industry in general to climb over in order to get to from the
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clinical the bench practice to laboratory practice into a clinical practice where you're actually treating
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people. I just did a podcast the or an interview the other day with
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the Washington Post, and the man was asking me about a drug that I
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prescribed for anti aging purposes, and he said, well, there's no human
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studies in this drug on using it for anti aging. How can you justify
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using it in humans? And I said, well, there are lots of
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human studies, it's just not for anti aging purposes. We have lots of
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drugs we do that with. So I think that what I'm trying to be
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is one of the doctors that helps to bridge that gap. And because I
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have a lot more time, I only see about six five or six patients
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a day, I actually can go and do a lot of research. So
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if you were a new patient coming to me, I would be able to
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go and do some research, find some things that maybe were outside the box,
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that hadn't been tried, that had been researched but hadn't been tried on
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people yet, or had been used for another reason. So it's a real
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blessing to be able to help There was another layer of patients that got better
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and another layer of patients that got better. Every time I would do some
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more research, we were able to find something else that helped people get better
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and better. So where did you actually go to medical school? And what
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was your degree in? And so I went to Emory and it was in
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family medicine. It was family medicine, that's what you said. Okay,
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So then did you go back to I know I remember you saying you went
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to a for the American Academy of Anti Aging and did some advanced studies.
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What were those studies in and how did they help you? Sure they have
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an amazing fellowship program that's very thorough. It looks at all the hormones,
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gas, ro intestinal health, metabolic health, nutritional health, it's a cardiovascular
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disease. It's a very thorough program, and it's sort of like a fire
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hose to the face, especially if you've never delved into this kind of medicine
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before. It really is so much information. So you spend really twenty four
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hours over a four day period just tackling a topic in heavy rotation, and
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so you're there for ten hours a day for four days straight, and you
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come home with all this information. So I did their basic Fellowship program,
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and then, of course I was still hungry for more, and I went
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back and did their advanced end of chronology, and then I did their peptide
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course. I'm in the middle of their stem cell course, their fellowship program
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for stem cells. There is something to be said for having the education to
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back up your experience and to back up your use. Even though we already
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are using a lot of doing a lot of that in our practice. It's
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nice to have the official document on your wall saying I did actually complete the
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recommended coursework in this particular program. Suzanne, how do you how do you
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know which group, like A four M is very you know, very renowned,
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But how do you how do you did you know which group to choose
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in order to get the advanced education? And how do you know that what
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they're teaching is the efficacious you know what, what do you call the efficacious
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uh result that you want? For me, it was exactly the right thing
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at the time because I was very allopathic at the time, and one of
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the things I liked about afoor M was that they are very evidence based,
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so all of their their teachers are required to use up to date research to
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back up what they're saying. So as opposed to you know, I just
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got back from a conference this past weekend in Miami on stem cells, and
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most of the lecturers did not use There were several that did, but most
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of them did not use references to back up what they were saying. Not
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that there aren't plenty of references for stem cell research or for regenerative therapy,
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but they did not really use a lot of that to back up what they
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were saying. And so I like being able to go to a patient and
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say, look, I have twenty articles to support why I think this would
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be a good choice for you because a lot of the things we do,
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there's again there's not an FDA approval, there's not a you know, sixteen
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articles that say this is exactly you know in this particular disease state. I
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just know because I practice cellular medicine. I know what the what it does
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in the cell, or I have a pretty good idea of what it does
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in the cell. And I know what the disease state, what the problems
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are in the cell, what the cell is experiencing as a result of the
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disease state, or what problem is causing the disease state, So I can
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match those two up in treating. And that's how we use the combination of
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things that we use is because because I know the disease state, I know
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the mechanism of action of the of the drug or the herb or the protein
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or whatever it is that we're using. You know, I'm kind of curious
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to know why is it a win? Was it that cellular medicine became the
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cornerstone of good health that people recognize in your positions that the cell is where
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it all begins and ends in terms of overall health. Well, as I
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mentioned before, I really think that the bench researchers, the ones who practice
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at universities in the research lab, not seeing patients but who are using lab
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rats. I think they have been looking at that for a long time.
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And I think that we've gotten away from that with the pharmaceutical industry to some
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degree, and then as we get around them, you know, that became
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with the insurance companies and the pharmaceutical industry, We really became a very diseased
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disease focused healthcare system. And I think as even as functional medicine practitioners were
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coming out, they were still very disease focused in there. You know,
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they may have used alternative therapies to treat, but it was still very disease
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focused as opposed to, let's just get the cell to do what it needs
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to do and then the disease state's going to go away. So your body
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has this beautiful innate ability to heal itself if we give it what it needs.
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Just put a little tweak here and a little tweak there, and now
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the cells actually able to do what it needs to do, and it has
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all of the capability to heal itself. If we can turn back on some
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of those genes and we can just start a cascade of action we often will
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get the cell to heal itself. Today's podcast is brought to you by International
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you want to let patients know that chacing symptoms is you know, the typical
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way that you know an allopastic medical doctor or a drug advocate, pharmaceutical drug
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advocate, I mean would say is the way to go, But actually you're
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chasing the symptom, you're not solving the problem. So yeah, you might
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have let's say, chronic depression, so you can give them, you know,
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a drug like prozac or whatever. But yet you're not getting at the
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roots cause of what is causing the depression in the first place. So how
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do you get around that? In your practice? I'm sure you have many,
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many moments when you have to defend yourself or how you go about doing
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what you do. Yes, so you know, sometimes something as simple as
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that I can say to that. Sometimes we will use things like prozac as
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a temporary measure to get you through the next phase. But most of the
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time I can get patients better fairly quickly, with with just decreasing their inflammation,
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with working on how their intestinal health is, with changing their diet,
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with improving that mucus layer on the lining of the intestine, with restoring their
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tight junctions, all of those things. If we can get those things working
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again, often I don't have to do a lot of intervention and they work
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pretty quickly. Patients recover. Your body wants to be well, so it's
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a matter of fixing it. Most of the patients, thankfully, who come
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to me come kind of knowing what we do. They know we do hormones,
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they know we do peptides, they know we do you know, all
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the things that we do ozone and ebo and all the things, and so
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they come with an expectation of that sort of So I'm not having a conversation
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with someone and saying, hey, we really need to consider doing bioidentical hormones
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on blah blah blah, because they're already most of them already know that's what
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we do. Do you have a kind of a list of requiring means that
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you put out to before you'll accept a patient? Is are there any prerequisites?
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I think the biggest thing is to and this is just one of the
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things I teach my nurse practitioners and the residents that come through our program,
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is that they need to The patient needs to be engaged as much or more
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than you are. And so if they are not willing to participate in what
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you're doing, they're not the right fit for us. And that's fine.
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And they may not be in a position right now where that's what they want
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to do. Maybe they don't want to change their diet. Maybe they don't
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want to begin a resistance training program. Maybe they don't want to participate.
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If that's the case, that's wonderful, but please give up your space to
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someone else who does want to participate, and we want patients to get better.
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My practice administrator does most of our contacts with people first, so she'll
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engage with them and find out if they're a good fit for us, and
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then she'll run out them through me. We do about a ten minute visit
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or fifteen minute visit to be sure that they're a good fit for us,
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and then that and that were a good fit for them, and that then
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we will schedule them for initial visits. So okay, so let's go back.
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I don't want to forget about your journey. So you you you know
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you did speak about the A four am and how that changed your educational side.
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What about the physical injury that you suffered, and how did that affect
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your journey? So I used to be a competitive runner and I got a
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condition called Trendellenberg where you're you're you're using your hamstrings more than your glutes to
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do the action of running. It's just a it's a common runner's injury and
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then you end up with a high hamstring strain because of that technical form.
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I didn't have a trainer at the time and was not I was doing that
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on my own and I've learned that lesson now, and I did not have
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someone to help guide me through form. And you know, you think running
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is just running, you don't have to worry about what you do, but
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when you're running the distances or when you're running competitively, you really need to
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have someone who's helping you with with that and giving you training and stretching exercises
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before and how do you cross train for other things. So this injury just
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persisted for a while and I missed a couple of big races and finally said,
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I need to switch this up. So I changed what I was doing
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to a more resistance training program and then looking for what could I find,
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I went to one of the conferences and they had a conference. They had
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a breakout session that was about mccun of growth factor, which is an IGF
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derivative that the body makes in response to injury. And this explained if to
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our viewers, Sure, IGF is what the liver makes in respect to the
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release of growth hormone from the brain, and IGF is one of the repair
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hormones that's very involved in not only repair, but helping the cell to choose
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which substrate the most efficient substrate to make energy. So let's say you go
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to the refrigerator because you're hungry. You could either pick a twizzler, you
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could pick a chicken breast and some broccoli, or you could pick a you
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could pick a h what's a good fat, a hummus or some other good
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fat like an avocado. And so the same thing is presented to the cell
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depending on what your diet looks like. And so the cell has the opportunity
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to choose which substrate it uses, and depending on which one it uses,
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as you know, if you had a bag of twizzlers an hour later,
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you're going to be hungry all over again. So and that's the same thing
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with the cell. If it's using straight glucose, you're going to have a
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harder time. You can make energy quickly, but you'll be back to needing
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refueling again. We don't want your body to use protein as a substrate because
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that's burning muscle and all of us need muscle for those beautiful mayakines the chemicals
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that are released from exercising muscle that help to maintain our useful state, right,
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and so the most efficient substrate for a cell to use is actually fat.
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So we I love that concept about the cell. And that's one of
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the things that IGF one does. Helps the cell to be able to choose
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that it's way more efficient in its use of fat to make energy. And
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that just allows the cell and we're talking about every cell in the body to
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allow the cell to do its job, the heart, muscle, the liver,
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the kidneys, the skeletal muscle, et cetera. So very efficient.
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And also, you know, IDF one is the way you measure human grows
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hormone in the blood, right, So you existactly don't. You don't have
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a direct HGH measurement, So that is kind of a back door measure way
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to measure, but it's as accurate as you can get to understand what your
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growth hormone levels are, you got it. It's tough to figure out because
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in women especially, we probably have seven to nine peaks of growth hormone release
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throughout the day, So your peaks are going up and down and going up
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and down all day long, So if I'm checking your level, I don't
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know if we're on a rise, or if we're at the peak, or
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if we're on a decline. I have no idea where we are. And
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IGF one is a little bit more stable. It's still not a perfect test
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of what things are happening with growth hormone, but it's helpful, so we
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do that, we do a combination of others that give us a better idea
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of what your growth hormone levels are. So that actually brings up another thought
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I have is when you do your blood work, your lab tests, what
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kind of tests do you do with people? And I know I'm segueing away,
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but it's very important, I think for people to understand what is it
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that you are looking to find out what, particularly with a new patient,
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So what are some of the most important measurements that you require in your lab
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work. I think it's interesting I've learned doing this kind of a research so
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much more about the very basic complete metabolic panel and complete blood count that I've
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done for years and years. I just didn't know how much they were telling
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me. So when I go back and look at the bench research, the
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laboratory results, research on things like albumin. It's an amazing marker of aging,
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and the reference ranges are so different when you look at it from a
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lab point of view, then you look at it from a clinical point of
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view. And so even when we do, some human studies are showing that
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the normal albumin level is over four and a half. So if I see
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a patient whose albumin level is declining, and I get labs on patients probably
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every three to four months so I can trend how they're doing, I know
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if they're albumin level is under four and a half, we have to look
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at several things. First, we have to look at their liver function.
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We have to look at what their protein intake is in their diet, how
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much resistance exercise they're doing in their life. And of course they're catabolic hormones
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like thyroid and cortisol, and by catabolic I mean break down hormones. So
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I'm looking at their balance between their catabolic and anabolic or growth hormones like estrogen,
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progesterone, testosterone, and their catabolic cortisol TSH to see if they're in
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a more catabolic state, which I would presume if their albumin is low.
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We see that a lot in post infectious syndromes, and in patients who have
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sort of chronic disease states, we'll see their low and I can say,
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look, your albumin's gone from so and so to so and so with the
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treatment that we've just done. And that's just a simple blood test that you
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would always, almost always, every doctor's going to order a complete metabolic panel
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and you'll see that albumin level. So there's several little points like that that
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I did not know as a regular allopathic doctor that I use now to help
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guide patients. So that's not all we do. Of course, we've run
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a large panel of hormones, looking at the ones I just mentioned, plus
22:10
prolactin of course the growth that we mentioned as well, and then we look
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at testosterone, estrogen, both of the estradil and estriol. We look at
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a complete thyroid panel, which most allopathic doctors aren't looking at and we're trying
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to. And then we look at a lot of inflammatory markers. So I'll
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look at a galectin three and ato, HDG is sistat and see, these
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are all just markers helping me know where the patient's overall health is. And
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IGF, I'm sorry, transforming growth factor beta we look at we look at
22:45
their aisle six, so other inflammatory markers, trying to see what's your basic
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inflammatory state and do I need to be addressing that in general or can I
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move forward and begin looking at the intestine. So the first place that I
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would go after that is intestines, and often we'll look at it just a
23:03
basic PCR test, which is looking for specific pieces of DNA looking for bugs,
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but also I'll look for a a intestinal panel that will look for what
23:18
kinds of inflammatory markers and like CALP protecting that would be present, potentially an
23:26
eosinophil X that would be causing some inflammation in the intestines, looking for things
23:30
like all sort of colitis, Crohn's disease. Well, the liver is another,
23:36
the alkaline phosphatase and the you know, the other liver markers are what
23:40
you're going to be really concerned about, particularly as you look at the microbion
23:44
and the GI trapped. It's really what they say, eighty percent of our
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immune system. And I'm I'm very pleased that when you said that, the
23:52
first thing you look at is the intestines or at least the GI trapped.
23:56
So you understand. You know obviously your practice in the medicine that preaches that.
24:02
So what And you didn't mention DHTA and cortisol, but of course those
24:06
are very important the sex hormones, right, and testosterone, I'd say,
24:11
but when you two tystosterone, do you do the whole panel of testosterone the
24:15
free testosterone and you know all you do the whole things? Yeah? You
24:21
know one thing I want to say, Sudan, it is so complex for
24:26
the lay person, and you you are simplifying it. I mean, I
24:32
congratulate you for doing that. You make something complex seem very simple. But
24:36
there's so many names that you just said, even just in the blood work.
24:40
I mean, yes, people know, most people know albumen is protein,
24:44
a measurement of protein. But these other tests that you mentioned, do
24:49
you sit down and explain to patients like what is each test and what are
24:53
we looking for? Like you just did with us, but obviously in a
24:56
very rapid way because we're on a pot. But you know, do you
25:02
hand hold what's a patient to let them know what they're what they're getting.
25:06
So we have the system we've worked out is I Usually we'll see the patient
25:10
and do a very thorough history and exam, and then we'll draw their blood
25:14
ideally on that first day, and then I see them back ten days later.
25:17
Maybe we'll do some small interventions between them that might make some good impacts,
25:22
just so they don't feel like they're being abandoned. And we do some
25:26
sort of impact the first couple of days, and then I'll see them about
25:29
ten days to two weeks later, and we'll go through their labs. I
25:33
usually will have commented on their labs and sent them to them on the portal
25:38
before I see them, so they have a chance to sit down, read,
25:41
digest, come up with some questions. Doesn't always happen that efficiently,
25:45
but that's our goal. And so then they can sit down with me and
25:48
we can go through line by line exactly what happens. It's difficult because I
25:55
don't have the whole picture. Even two weeks later, I don't have the
25:57
whole picture of what's happened. So let's say their CRP is high. But
26:03
in between when I saw them and when they come and see me again,
26:06
they had a some sort of dental thing that happened, or they got up
26:10
a respiratory infection that popped up two days after they saw me. Well,
26:14
probably the body was already ramping up that inflammation when they were in for the
26:18
blood work, and so unless I have that information, So that's how that's
26:22
why we sit down and talk about it, because I can tell you what
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that result means big picture, but without knowing what it means for you,
26:32
I can't tell you exactly what it means. And we have to look at
26:34
all the labs collectively to make a decision about each one fits together like a
26:41
lock and key, and I need to I'm not just doing one test for
26:45
one disease state, and none of them are perfect. So we're putting together
26:51
these incomplete pieces, trying to come up with a whole and then making a
26:55
plan for what our next steps are for optimizing your overall health. So the
27:00
one thing that you said CRP, I want people to know that that stands
27:06
for C reactive protein, and C reactive protein is a measurement of inflammation,
27:10
but it also has a direct measurement to your heart health. Correct, yes,
27:15
yes, So normally you would do your lipids and then your CRP and
27:19
your Homo systein to get and also the size of particle size you get a
27:25
better picture of your health, so you do the all the you know,
27:29
the lipid test itself is pretty complex. It is, so yes, I
27:34
just give you kudos for trying to make this, you know, understandable to
27:40
people when they're just you know, they're trying to take responsibility for their health
27:45
and yet you know, it can be daunting. Yes, it's a lot,
27:48
especially if you are a high achiever and you have high hopes for yourself.
27:55
It can be very frustrating when I say my goal for your blood shirter
27:59
is to have it less than eighty seven because of fasting blood sugar greater than
28:03
eighty seven. There's a study that came out in twenty fourteen that for every
28:07
point above eighty seven that your fasting blood sugar goes, your risk of developing
28:11
diabetes goes up by six percent. So if you're at one hundred, which
28:17
is what everyone in the allopathic community considers abnormal, you're already at fifteen times
28:22
six's. You're already at sixty percent seventy percent increase risk of developing diabetes in
28:29
the next ten years. So there's no need for me to wait. Most
28:33
of my patients don't want me to wait until they're already like go book.
28:36
By the way, surprise you have diabetes. I want to say, your
28:38
blood sugar is starting to trend high. What are things that we can do
28:44
to help reverse that, because that's one of the things we know is a
28:48
marker of overall aging and disease in general. And so I know that your
28:53
cells are stressed if your blood sugar is a little on the high side.
28:57
So how do I help your cells you a better job? And there's lots
29:00
of things we can do to intervene on those things. So you know,
29:04
I keep keep segueing back to your injury into how this changed your life.
29:08
But I also remember you said that you had something else happened because you're heavy
29:15
a weightlifter, right, and say that you had some something else is life
29:21
changing? Was weightlifting that caused you to, you know, move towards final
29:27
medicine. So the weightlifting actually started around the same time. The I think
29:37
the the big thing that you're maybe referring to is that the gyms were all
29:42
closed during the pandemic a few years ago, and so the only gym that
29:48
was available around my area, the gym I went to was closed. The
29:52
only gym that was available was this powerlifting gym, so I went to they
29:57
were open one per at a time in the dark. They would let you
30:02
in the back door and they would be able to train with one person at
30:04
a time. So that's been really fun. And I've this is a,
30:08
like I said, a powerlifting gym, and I've been able to compete with
30:11
them with their team a few times. It's been really fun. Okay,
30:15
so have you had injuries doing the powerlifting? I did? Back in August,
30:21
I was training for the last competition, and just before my competition,
30:26
you know, I think this was a case of I was trying to get
30:30
into the lower weight class. I was more competitive at the lower weight class
30:34
and just on the border of my of that you know, weight class,
30:40
so I wanted to kind of drop down if I could. I think I
30:42
was overtraining, which I actually don't think is overtraining. I think it's under
30:45
nutrition. And so I had probably been cutting my total coloric intake too much
30:53
for the amount of training I was doing in competition prep, and I sustained
31:00
an injury. I have been under some stress from some other things as well,
31:03
so sustained injury that also has done better. We've treated that with some
31:08
ozone, some neural therapy, some peptides, and so that's been really helpful
31:12
and what exactly is neural therapy. It has been so dramatically life changing for
31:21
our practice. It's been sort of the little missing link for us. And
31:26
it is a centuries old procedure that's primarily done in Germany and in South America,
31:33
and they use the old procaine. So originally anesthesia was done with cocaine.
31:38
Procaine was the original derivative of cocaine that did not have the addictive potential
31:47
of cocaine. And procaine has some unique properties that the other anesthetics don't have.
31:53
Number One, it's metabolite is paba, which has some unique way that
32:00
it works in the cell to re establish the membrane potential of the cell.
32:06
It's a fascinating molecule how it works. So cells that are under stressed will
32:10
have a change in their energetic ability to function, so their sodium potassium channels
32:20
will drop and the area will lose blood supply because of that change in polar
32:27
charge. So then we can go in with something like procaine and we can
32:30
re establish that polar charge. So now the cell walls work better. We
32:36
can get nutrients into the cell, we can get toxins out of the cell,
32:39
we can get fuel sources into the cell that the cell needs to do
32:45
its job, and now the cell begins to work better. Often it requires
32:49
several treatments, two or three usually for it to be effective, depending on
32:52
the situation. And where we've seen a lot of benefit is in stress PTS
33:00
D anxiety. We do what we call a therapeutic stellate infiltration and it's been
33:07
dramatically beneficial for several of our patients in reversing and supporting their recovery from all
33:16
kinds of illnesses and injuries. And then how do you combine that with the
33:22
ozone, So you can either use it at the same time or you can
33:25
use it you can do separately because it's available IV the ozone. They work
33:32
in separate ways, but they do a really beautiful symbiosis for this cell.
33:38
So the ozone is converted into the hydrogen peroxide or lipid peroxides, and that's
33:46
one of the ways that it works in the cell is by improving the microbial
33:54
milieu in the cell and in the space where the cell exists. So now
34:00
cells not having to fight off those microbes. It also triggers the NRF two.
34:06
NRF two is one of the proteins that helps you've heard of antioxidants.
34:10
Well, once we get very stressed, our cells become very stressed. We
34:15
have not enough antioxidants to handle all of the reactive oxidants or the oxidative stress
34:21
that our cells have, and so NRF two is the trigger that allows the
34:28
cell to recycle those antioxidants like vitamin C, vitamin E, glutathione so that
34:34
they are available for use again. So that's a great ozone is a great
34:39
combination. So now we have the prokine which is helping with re establishing the
34:45
cellular membrane polarity, and then we have the ozone which is not only removing
34:51
any microbes but also triggering the antioxidant benefit so that your mitochondria are able to
34:58
function optimally with plenty of antioxidants. And it's this beautiful symbiosis. The cell
35:04
is in this beautiful milia and now can do its job better. And you
35:07
do just buy IV or sometimes into the joint, sometimes i am. It
35:13
depends on the patient, depends on what the patient needs. A lot of
35:16
neural therapy can be done just subcutaneously, it's taken up by the cutaneous nerves
35:22
and then that travels along the nerve to the main nerves of the spinal cord
35:29
and the brain. In fact, most people will say that they have sort
35:32
of a light headed, euphoric feeling, a joyful kind of feeling. Some
35:37
people have an emotional release. I know you've probably heard of people holding emotion
35:40
in their scars, and a lot of neural therapy is treating those scars with
35:45
procaine, opening up those channels, releasing the scar so that there can be
35:52
communication through the fascia in that area. That is fascinating. You know.
35:57
I never heard of that before. I know of vosone therapy, but not
36:00
of the neural therapy. And are you one of the few clinics that are
36:05
using that or is that pretty widespread. I think it's not as common as
36:08
it as it could be, but I think there are probably a handful of
36:13
physicians around the country doing it. Yeah, I'm just wondering if there's anyone
36:17
out here in the California. I'll have to ask you that when we get
36:20
offline. So, Suzan, I don't want to lose the most important part
36:27
of this podcast. I want you to talk about your book, and we've
36:30
segue over to that if you don't mind, because you focus on peptides and
36:37
you're one of the few courageous ones that's taken on that subject in a very
36:40
simple, simple way to help delay person understand because honestly, the number of
36:45
peptides, and you know you'll get into that, I'm sure, but just
36:51
the number of peptides that are out there and the different modalities that they address
36:59
is again another daunting subject. So, uh, I started reading your book.
37:04
I had to ask Terry to send me a hard copy because it was
37:07
hard to read it on the PDF. But because I want to take notes.
37:10
It's just such it has so much information. I just, you know,
37:15
want to be able to jot down notes on the on a on a
37:19
page and then come back to you with questions. So if that's all right
37:22
with you, if I could have that hard copy, I really appreciate it
37:25
because it isn't this is a really important book. I just want everyone that's
37:30
watching today to know counterclockwise you need to go. Is it on Amazon already?
37:37
No, it'll be on It'll be on in two more weeks March fourth.
37:42
Okay, So just the beginning that I've you know, I've gone through
37:45
maybe three chapters and I'm like fascinated and I but I also, like I
37:51
said, I know it's going to be a bit confusing to people. So
37:53
let's talk about the peptides. Let's try to take a very simple look,
38:00
like a thirty thousand foot overview of peptides. Why didn't you start from the
38:06
beginning and just give us an overview? It's pretty exciting. So think about
38:12
this that most of the proteins that we eat in our diet are over one
38:17
hundred and fifty amino acids long. So amino acids are the building blocks in
38:22
our bodies. They create protein, and so if a chain of them was
38:28
put together sort of like a string of pearls, they depending on which sequence
38:32
they're put together and how long the chain is, it will do a different
38:38
thing and it will bind to a different receptor inside your cell. So peptides
38:42
are not just drugs that we give people. These are actually things that your
38:45
body makes. So there are hundreds of thousands that we don't even know about
38:51
yet. This science is still evolving. It's very exciting. But since the
38:54
nineteen twenties, we've known about peptides like insulin, like this is a cretan,
39:00
which was the first one that was discovered. And if you all remember
39:05
Pavlov's dog, that laboratory researcher was actually doing research in finding peptides in dog
39:14
intestine, so he was doing research on and he found secretan was one of
39:20
the first ones back then. So it's very exciting and the science sort of
39:27
drug along because there wasn't a really easy way to make them quickly in a
39:31
lab until around two thousand and five, so it was done in a very
39:36
labor intensive process, and or we had to take them from cows or pigs
39:40
or other humans like human insulin for example. Now peptides are everywhere. The
39:45
pharmaceutical companies are aware of them, and because we're able to make them en
39:50
mass, we are actually the pharmaceutical companies are using them. So there's some
39:54
relatively new migraine medicines that are out using that are being used that are peptides.
40:00
There are some diabetes and weight loss drugs that are out and that's where
40:04
most of the research and development with peptides is going right now, is in
40:08
the oncology space and in the metabolic health space. So it's very exciting time
40:14
for us to be doing this kind of research. And there's every day new
40:19
peptides being especially because of artificial intelligence, we can there are brand new peptides
40:23
we can learn about every day. Every time I look on the on Google
40:28
scholar, there's a new one that I didn't know anything about. So it's
40:30
it's an exciting time. And although it could be disappointing and discouraging that the
40:37
FDA has given us a thwart in our in our process, the exciting part
40:43
is that that's not because of any reports of adverse effects or of patients having
40:52
not having good outcomes or efficacy. It's really just it's really just it's not
40:58
because of that. So that's great news, and it just means we have
41:01
to be more creative and keep doing research. So that's what we're doing.
41:05
Yeah, So let's talk about the top can so that people know what you
41:10
used most frequently in your practice. So let's start one by one and talk
41:15
about each one so that people understand what it does and what kind of results
41:22
you've had in using them for years. Now. Okay, So one of
41:25
the reasons I wanted to write the book was because I felt like there was
41:30
a dosing difference between men and women, and so I wanted to have a
41:34
book that really addressed the difference in women, because these have been studied for
41:39
years or used for years in male primarily bodybuilders, and I knew that there
41:45
was a difference in women, and I wanted to be able to produce to
41:49
give that information and to be able to provide that. So what do I
41:54
commonly use? Almost always, depending everyone's different. So to say what do
41:59
I use in every single patient, that's a tough one, but I would
42:01
say almost always, I'm going to use something like a growth hormone secretagog and
42:07
there's lots of them that are out there. I don't usually use growth hormone
42:12
directly because it can raise blood sugar and it can raise cortisol, and I
42:15
don't know many of us that need a higher cortisol. So I like the
42:21
growth hormone secreitagogues because they don't raise blood sugar or cortisol. Typically, all
42:25
of this, of course, is done under the supervision of a trained physician
42:30
who knows how to treat this disease state. And this is for my patients.
42:35
A general idea of what I do with my patients and so I'm going
42:38
to use a growth hormone secretagogue. And part of the reason for that is
42:43
is that patients, again this is at the cellular level. I want the
42:46
cells to be able to choose the proper substrate for them to use for energy
42:52
and to be able to be very efficient at creating energy, at making mitochondria.
42:57
Mitochondria those energy generators in every sell And that happens when you use the
43:02
growth hormone. Secrete to Gods helps to build muscle, which is one of
43:07
the most important issues for people, particularly when they're doing weight loss like Gozempeic
43:13
or Manjaro or Govi. You know, their potential is to yeah, the
43:20
lower their insulin level, the lose weight, but they also lose muscle.
43:22
So the secrete of gods what you're talking about, help you to counteract that.
43:28
Correct, You're right, that's excellent. So what they actually do is
43:31
they improve your recovery after injury so that you're able to get back to sport
43:38
more quickly. So the research doesn't really support so much. It's interesting to
43:44
me that it's banned by Water because there's not a lot of research supporting that
43:49
it actually improves your strength improves your speed, improves your muscle. But what
43:55
it does do is allows you to get back to sport quickly. Because you're
44:00
able to do that, then you're able to build muscle better, whereas if
44:06
you were not using them, you would take a longer rest between training sessions
44:09
and it would be harder for you to gain strength or muscle or speed.
44:15
Was ipra morell and one of the ones that you like in that category?
44:21
It's one of the ones we liked in that category. Yes, and what
44:23
else? What are the other one I do have that I do like that
44:27
one? In my female patients, they tend to not get so much hand
44:32
swelling, which is one of the potential side effects. They tend to not
44:36
get. The hand swelling is the big one that women will report. And
44:43
also, I mall in tends to move the bowels a little bit so they
44:46
get that nice resolution of their constipation. Yep, that's very important. So
44:52
okay, so that's a good one to jot down everybody. Ip of morele
44:55
And I've used it and I really like it a lot, and I ran
45:00
out of it, So I'm just I'm glad we're talking about it because it's
45:02
reminding me to go get another prescription of that. Okay, what about be
45:08
more difficult to get because the FDA has told the compounding pharmacies they are no
45:13
longer allowed to compound it. I do think, however, that there is
45:16
a pharmaceutical company that owns it and will be coming out probably in the next
45:22
couple of years with some version of it. Pharmaceutical. I thought, though,
45:25
you could get it from a research laboratory. Even if you can't get
45:29
it from a compound pharmacy like this, Life is a good research lab right,
45:36
So I don't recommend that patients do that because I don't know what They
45:40
have not been tested. In fact, if you get a vial from one
45:44
of those places, it will say for research purposes only, not for human
45:47
consumption. They have not been tested for the toxin. So the way that
45:52
peptides are made requires a resin at the sort of This is a simple way
45:59
to explain it, but there's a resin at the bottom that's sort of sticky,
46:01
and then they put the amino acids in one at a time to build
46:06
that string of pearls. Then they have to chop off that resin. Well,
46:08
they don't completely chop off that resin off the bottom, then it can
46:13
be liver toxic. Again, that's an oversimplification of the process, but if
46:19
they're not tested for that, you may find that there's a problem with it,
46:22
and that's what we're seeing. If there are reports of toxicity from peptides,
46:27
it's usually from coming from a research lab and not from a compounding pharmacy.
46:34
So that's where we get concerned. That's really good point. I'm so
46:37
glad we talked about that because I don't think everyone understood that, and you
46:42
know they So if Amrellan's off the market for a while, so what would
46:45
be the replacement that would be next best that is available. So there'll be
46:51
three that I know of available in the next couple of months as things are
46:53
transitioning over the Sermarillan will be around, Tesla morellan will be around, and
47:00
there's a new new ish one called massimarillin m aci morelen very similar effects.
47:07
The massamarillin has some interesting research, particularly in fatty liver disease, which is
47:15
where the liver becomes infiltrated with fat, usually due to diet and sedentary lifestyle,
47:21
but can be due to genetics or genetic variants, and so patients will
47:25
not be able to use their liver as efficiently because of the lack of this
47:30
or because of the presence of this, and the massimarillin has some interesting research
47:36
there. It also has some interesting research in icee you patients with maintaining the
47:45
diaphragm muscle integrity. So as patients go on a ventilator, their diaphragm becomes
47:52
weaker, just like if your bise if you stop doing bicep curls, your
47:54
bicep would become flaccid or weaker. And so the patients we're put on this
48:00
growth hormone secreteagogue, we're able to maintain the muscle density of their diaphragm even
48:07
in the face of ventilator youth. So that was pretty interesting. That's great.
48:13
Okay, So that's the secretey togogue. I mean the you know,
48:17
well, I guess you call those secreteagogs, right, yeah, okay,
48:21
So now we move on to your next choice in the next category. So
48:25
it depends on the patient and almost always if there's any sort of inflammatory state.
48:30
So if we're talking about a very healthy patient who has who is trying
48:37
to work on their aging processes, and we know that aging is related to
48:42
cellular health in general, mitochondrial health, et cetera. And so if we
48:45
can work on those things, we can improve the health of the self.
48:49
We can decrease the inflammatory state of the body in general, and then work
48:52
on the cellular health. We're golden. So one of the things that we
48:57
do is addressing the intestinal health. And there's a couple of peptides I really
49:01
like for this and they're available orally, which is really nice, and I
49:05
think that they will probably still be available after this FDA change, And so
49:10
BPC one five seven, which people are familiar with. I think one of
49:14
the pieces this is a naturally occurring peptide that comes from the stomach that its
49:19
original design I presume from God was to help to treat or prevent stomach ulcers
49:27
from occurring. But the interesting research is in things like tendinitis, in joint
49:31
arthropathy, in other pain syndromes that it can be very helpful in healing wounds,
49:38
et cetera. Most of that research is done in animals, but there
49:42
is an interesting study done by doctor Lee in intra articular injections in the knee
49:47
and the benefits of that are something like eighty percent in patients who were treated
49:52
with interarticular knee injections in his lab. Again, just one human study,
49:58
but very compelling evidence. I think it's important for people to know too that
50:02
the ooral BPC one five seven is better targeted for the intestines, as you
50:09
said, and that the injectable is better targeted at the area of the wound.
50:15
So if you were injecting subcutaneously, inject right close to or into where
50:21
like you did with the knee, into the area of trauma, is that
50:27
what you would tell patients to exactly, And I would tell my patients to
50:32
that of course, because the ooral is available. One of the things I
50:35
think that's a clinical pearl that I want people to remember is if your problem
50:38
is in your stomach, you actually need to open the capsule and take the
50:43
BPC by mouth with the capsule open, because if you're taking it with the
50:47
capsule intact, it's going to bypass the stomach and won't even start to work
50:52
until it gets to the smaller listine. There are lots of new delivery methods
50:57
that are coming out, some nanoparticles and some other delivery methods that will be
51:00
very effective. But keeping in mind right now, if you're treating a stomach
51:06
or esophagial problem. There's some interesting literature around using it for dental issues,
51:14
and so opening the capsule and pouring it into your mouth and swallowing it with
51:20
the capsule open. Unless you're treating something below this, below the stomach,
51:23
you really want to do that. It doesn't taste great, but hold your
51:28
notice. Yeah, it's so. I was just going to say that would
51:30
be right before you said that, because I just had some gentle procedure and
51:36
my doctor and Beverly Hill, said take the BPC, open the capsule and
51:40
brush with it right on your gums because that's going to help to heal that
51:45
area that had the trauma the surgery. So I haven't done it yet,
51:50
but i've been, you know. And now that you're telling me to take
51:52
it even intestingly take it out of the capsule, is even that's even really
51:59
better? Because I've been just swallowing it thinking it was going right to the
52:04
stomach. But you're saying go to the stomach but not to the colon.
52:07
Yes, So take it out and just hold your nose and take it.
52:12
Okay, good, And then if you want to treat small intestine and lower.
52:16
You can use the capsule whole, but just realize if you're taking it
52:21
by mouth, it's not going to get to your stomach or your esophagus when
52:23
you take it that way. The other one I really love, I think
52:27
is important is the lazatide, and that also is in phase three trials for
52:31
chrome I mean, sorry for Celiac disease. So very exciting, but we
52:36
need to keep in mind it's mechanism of action is repairing that tight junction,
52:39
that space between the intestinal epithelial cells that protects the body from the invaders,
52:46
both allergens and microbes. And so if we can maintain that tight junction with
52:53
this peptide called eighteen one thousand and one, if we can maintain that tight
52:59
junction, then we can some really good benefit. The other things that break
53:01
down type junctions are infection, So anytime that you're having intestinal symptoms with an
53:07
infection. For example, our most recent pandemic, a lot of my patients
53:10
had intestinal symptoms with their viral infection, and so we will use this for
53:17
them. The other thing that breaks down tight junctions is cortisol or stress hormone,
53:22
So in your patients that are particularly stressed. We want to put them
53:25
consider protecting their tight junctions and their overall health of their body, because almost
53:31
all health begins in the gut, and if we can keep the inflammation there
53:36
at a minimum, then we can decrease inflammation in your whole body. And
53:39
for those who don't understand what the ucastal lining, there's a name called leaki
53:44
guts, which is a little more graphic, but it does explain what Suzanne
53:50
just described to us. So that is a key element of your overall health
53:55
is to prevent leaki guts. As Suzanne, they just signaled me that we
54:00
only have a couple of minutes left, and I know I'm cutting us short.
54:05
This is frustrating for me because there's so much more I want our viewers
54:08
to know. So I'm going to invite you back. We'll give it a
54:12
few months. That's to let people digest no pun intended talked about. But
54:19
also we want to get your book out there and see you get that traction
54:22
on the book and have some people read it and so forth. So I
54:25
know you've offered a very generous gift to our viewers, So to help them
54:30
on that journey, could you just give us a quick explanation of what you'd
54:34
like our viewers to do. For just a nice incentive from you, sure,
54:39
We're giving a free PDF of the first chapter of the book. It's
54:45
if you just go to Vine as in grape Vine vinemedical dot com forward slash
54:51
counterclockwise, you'll get the first chapter of the book, and when the book
54:54
comes out, we'll send you an email telling you that the book is now
54:59
available on Amazon. And I believe it's Barnes and Noble and in airports.
55:05
I love that. And you know, believe me, everyone, when you
55:07
read that first chapter, you're going to be frustrated that you can't go right
55:10
to the rest of it. But that will come in time, as soon
55:14
as Suzanne's publishers are ready. So okay, well, I just want to
55:17
thank you so so much, and I'm so happy that I'm sorry that we
55:22
didn't get to meet at a four m But I know you're going to be
55:24
talk lecturing next year, right in December. Yes, So what's your lecture?
55:29
The title of your talk? I'm lecturing this month on all Roads of
55:36
Health lead to the start with the guts. Oh good. The one in
55:39
the fall will be on bioregular or peptides. Okay, good well, you
55:44
and I are both you have that in common, which we didn't talk about
55:46
today, but with doctor Bill Lawrence and our involvement in his clinical trials.
55:54
So that's a whole other subject. That's why we need to have you back.
55:58
So I do want to say again, thank you very much for taking
56:00
the time. I know how busy you are, and I am going to
56:04
follow up with you on that that new peptide that you just mentioned. I'd
56:07
like to get some some avenue to obtaining that. Okay, okay, so
56:15
very little. Thank you so much, Michelle. It's going to treat Okay,
56:17
take care, Sudan. I like bye.