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WHAT'S THE DEAL WITH CELIAC DISEASE? · Your Daily Dose

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  Speaker 1: You know, if you've gone shopping lately, you notice there's
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  Speaker 1: a big push for gluten free this and gluten free that,
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  Speaker 1: because the whole world thinks they have Silliac's disease and
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  Speaker 1: silly X disease is an autoimmune problem that needs to
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  Speaker 1: be diagnosed. But a lot of people think they have that,
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  Speaker 1: But could it be they just have an allergy to gluten.
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  Speaker 1: And I just want to make sure we understand that
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  Speaker 1: there are enzymes available to help digest gluten. That seems
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  Speaker 1: to be a problem, So we're going to go into
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  Speaker 1: that today.
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  Speaker 2: Stay tuned, hi.
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  Speaker 3: And welcome to Health Talk with Doctor Lynn.
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  Speaker 4: Your daily dose.
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  Speaker 1: The information you hear on the show is informational. It's
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  Speaker 1: not meant to diagnose or treat any disease. Any kind
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  Speaker 1: of complete health conditions must be checked out and discussed
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  Speaker 1: with your primary care physicians. Get ready for the good
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  Speaker 1: news of the day. God bless you, folks, and here
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  Speaker 1: is doctor Lynn. Our daily devotional today comes from the
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  Speaker 1: Book of Revelation, Chapter nineteen. Expect to encounter adversity in
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  Speaker 1: your life, remembering that you live in a deeply fallen world.
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  Speaker 1: Stop trying to find a way that circumvents difficulties. The
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  Speaker 1: main problem with an easy life is that it masks
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  Speaker 1: your need for me. When you became a Christian, I
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  Speaker 1: infused my very life into you, empowering you to live
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  Speaker 1: on a supernatural plane by depending on me. Anticipate coming
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  Speaker 1: face to face with impossibilities, situations totally beyond your ability
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  Speaker 1: to handle. This awareness of your inadequacy is not something
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  Speaker 1: you should child I to evade. It's precisely where I
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  Speaker 1: want you, the best place to encounter me in my
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  Speaker 1: glory and power. When you see armies of problems marching
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  Speaker 1: towards you.
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  Speaker 2: Cry out to me.
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  Speaker 1: Allow me to fight for you. Watch me working on
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  Speaker 1: your behalf as you rest in the shadow of my
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  Speaker 1: almighty presence.
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  Speaker 2: Stay tuned point.
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  Speaker 1: The number to reach me off air seven to two,
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  Speaker 1: seven nine one, six, four to two. New studies suggests
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  Speaker 1: a potential new cause of Siliac's disease. A common autoimmune
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  Speaker 1: disease may not be caused by an overactive immune system,
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  Speaker 1: so what has been the gold standard for these years
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  Speaker 1: may not be true. Your immune system may signal that
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  Speaker 1: Ciliac's disease is coming long before you ever feel sick,
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  Speaker 1: possibly even before you eat a single bite of gluten.
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  Speaker 1: Siliac's disease has long been attributed to an overactive immune
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  Speaker 1: response to gluten. However, a study recently published in the
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  Speaker 1: journal Immunology and Cell Biology suggests there's a reduction in
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  Speaker 1: immune activity that starts much earlier and runs much deeper.
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  Speaker 1: The research focused on the type of immune cell called
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  Speaker 1: a CD four helper T cell. These white blood cells
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  Speaker 1: are to immune cells what sergeants are to soldiers. They
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  Speaker 1: give orders, help coordinate the body's immune response, fight infections,
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  Speaker 1: support antibody production. The surprise wasn't that these cells were overactive.
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  Speaker 1: They were actually weaker, producing less of a key immune
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  Speaker 1: signal called interluken iiO, dividing more slowly, and less likely
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  Speaker 1: to survive. This pattern held regardless of sex and recently
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  Speaker 1: someone was diagnosed, or how long they've been gluten free.
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  Speaker 1: That suggests that changes in the gut may not be
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  Speaker 1: caused only by active inflammation or recent glutenant exposures, said
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  Speaker 1: doctor Jason Cornblit, a Board certified Gas storenerrologist who was
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  Speaker 1: not involved in this particular study. This may reflect a
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  Speaker 1: built in immune tendency, like influenced by genetics to detect
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  Speaker 1: these differences, researchers use the new technology called criton to
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  Speaker 1: cell timer and a method that briefly activates immune cells
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  Speaker 1: then removes all signals to observe how the cells behave
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  Speaker 1: on their own. Our essay is a bit like whinding
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  Speaker 1: up a toy and letting it go and see how
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  Speaker 1: long it runs and what trips it performed, said study
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  Speaker 1: author doctor Vanessa Bryant in a statement. What that whining
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  Speaker 1: and releasing revealed is a property that researchers called immune momentum,
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  Speaker 1: a new scientific concept describing how long immune cells stay
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  Speaker 1: active after their initial trigger has stopped. They found that
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  Speaker 1: CD four helper cells are producing less of a key
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  Speaker 1: immune signal called interlukent, dividing more slowly and less likely
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  Speaker 1: to survive. Gluten is still necessary to activate the disease,
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  Speaker 1: Cornplit said, but the immune system may already be wired
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  Speaker 1: to respond abnormally. That may help explain why Ciliac's disease
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  Speaker 1: clusters and families, and why it overlaps with other immune diseases.
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  Speaker 1: From a gastonrology standpoint, he said, this study is important
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  Speaker 1: because it supports the idea that itself Blix disease is
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  Speaker 1: not just a reaction to glute in the gut may
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  Speaker 1: also involve a deeper baseline difference in how the system
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  Speaker 1: was programmed. While the study focused on Siliac's disease, the
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  Speaker 1: researchers believe similar immune patterns might exist in other autoimmune conditions,
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  Speaker 1: which affect about five percent of the population. Study author
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  Speaker 1: Jason tied Dinn believes that new testing model will eventually
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  Speaker 1: uncover hidden patterns across the range of autommune conditions, which
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  Speaker 1: affect again roughly five percent of the population. Although these
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  Speaker 1: helper cells aren't always the main focus in immune research,
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  Speaker 1: they may hold important clues as to why the disease
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  Speaker 1: develops in some people and not in others. The team
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  Speaker 1: is now investigating whether similar immune patterns exist in other
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  Speaker 1: autommune conditions now earlier in life. These differences appear, the
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  Speaker 1: implications for early detection are significant, though experts urge caution
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  Speaker 1: on how quickly this could translate into clinical practice. If
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  Speaker 1: future studies confirm that these immune patterns appear before symptoms
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  Speaker 1: or intestinal damage, testing immune cell behavior could identify high
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  Speaker 1: risk individuals far earlier than is currently possible. This, he added,
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  Speaker 1: could be especially useful for first degree relatives of people
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  Speaker 1: with Siliac's disease, children with Siliacs associated genes, or patients
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  Speaker 1: with other autoimmune diseases. Earlier detection could allow closer monitoring,
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  Speaker 1: earlier antibody testing, nutritional assessment, and faster diagnosis whence disease begins. However,
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  Speaker 1: we would need larger long term studies following at risk
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  Speaker 1: people over time before using this clinically. Now, I want
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  Speaker 1: to tell you about a digestive enzyme called cybe zymes
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  Speaker 1: cybs zymbs by Nutritional Frontiers, has the capability of actually
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  Speaker 1: helping to digest gluten in the system. So again, want
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  Speaker 1: to be cautious. Have been diagnosed, Still seems that something
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  Speaker 1: happened if I consume gluten. Let's try adding a cybzime
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  Speaker 1: prior to every meal and watch what happens. You can
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  Speaker 1: find that at nutritional frontiers dot com. Here's a little
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  Speaker 1: bit on them. Hey everybody, once again, thanks so much
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  Speaker 1: for tuning in today and listening.
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  Speaker 3: All you have to do is go to nutritionalfrontiers dot com.
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  Speaker 3: and don't forget the numbers to reach me directly seven
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  Speaker 3: two seven win six four two one five. God bless
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  Speaker 3: you folks, talk to you soon.
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  Speaker 1: All right, that's what we got for you today. Please
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  Speaker 1: share it with others. God bless see you