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Reduce Inflammation with Science-Backed Natural Solutions with Samuel Shepherd - Part 2 | Holistic Pharmacy Podcast · Pharmacy Podcast Network

• 0:05
  Speaker 1: You are listening to the global network of podcasters dedicated
• 0:09
  Speaker 1: to the pharmacy profession. Welcome to the Pharmacy podcast Network.
• 0:27
  Speaker 2: Hello, and welcome to the Holistic Pharmacy Podcast. I'm your host,
• 0:33
  Speaker 2: Marina books Of and I'm a functional medicine pharmacist in
• 0:36
  Speaker 2: New York. I'm also a clinical herbalist and an integrative
• 0:40
  Speaker 2: health coach specializing in mindfulness and empowerment. I'm on a
• 0:45
  Speaker 2: mission to transform the way we approach healthcare by bridging
• 0:49
  Speaker 2: the wisdom of traditional medicine with modern practice, and in
• 0:53
  Speaker 2: this podcast, I will be highlighting guests who do just that.
• 0:58
  Speaker 2: I'm constantly inspired by them, and I know you will
• 1:01
  Speaker 2: be as well. Hello. Hello, We are back this week
• 1:04
  Speaker 2: with part two of doctor Samuel Shepherd's episode. If you
• 1:08
  Speaker 2: haven't checked out part one, it's the episode right before
• 1:12
  Speaker 2: this one, so check it out wherever you listen to podcasts.
• 1:15
  Speaker 3: Enjoy the show.
• 1:19
  Speaker 4: I begin to see these research papers come out of
• 1:23
  Speaker 4: the NIH in direct support of everything that I had found.
• 1:28
  Speaker 4: That was the most remarkable moment of my entire life.
• 1:34
  Speaker 4: And I now know what my purpose was why I
• 1:38
  Speaker 4: didn't die back in two thousand and six, And now
• 1:44
  Speaker 4: everyone I don't have to be here to tell this
• 1:48
  Speaker 4: story anymore. All you have to do is Google or
• 1:51
  Speaker 4: search as does Anton and NIH for the National Institute
• 1:58
  Speaker 4: of Health and whatever disease you want to look at, arthritis, cancer,
• 2:05
  Speaker 4: type two, diabetes, Parkinson's, and all of the research shows
• 2:12
  Speaker 4: the impact of the astes aanthon on those diseases.
• 2:18
  Speaker 3: I love it.
• 2:19
  Speaker 5: I have a couple of more quick questions to clarify.
• 2:23
  Speaker 5: So one, is you mentioned all these wonderful disease states
• 2:27
  Speaker 5: and organ systems that this can positively affect, And it's
• 2:32
  Speaker 5: working at the level of inflammation as an antioxidant and
• 2:35
  Speaker 5: helping your bodies slowly declining indogynist antioxidants right, and helping
• 2:42
  Speaker 5: to replenish their activity. Now, is there a difference in
• 2:47
  Speaker 5: dosage for different indications that you have found? Is there
• 2:51
  Speaker 5: anybody that should not be taking it? Can you give
• 2:54
  Speaker 5: us just like a couple of quick answers to that?
• 2:56
  Speaker 5: Or is everything on the website?
• 2:58
  Speaker 4: Everything's pretty much on the website. But let me clarify
• 3:01
  Speaker 4: some things. Dose is weight dependent and time so the
• 3:09
  Speaker 4: astesanthen is readily absorbed by the fat cells the lipids,
• 3:17
  Speaker 4: so and it has a half life of about thirty
• 3:20
  Speaker 4: six hours. So if you take it and you saturate
• 3:24
  Speaker 4: yourself in thirty six hours, you're going to have half
• 3:27
  Speaker 4: of it remaining in your body. In thirty six more hours,
• 3:31
  Speaker 4: half of that, you're not going to have a quarter left.
• 3:33
  Speaker 4: So there's a half life inventory that you can hold
• 3:39
  Speaker 4: in your body now. So we look at the disease state.
• 3:45
  Speaker 4: If somebody is stage four cancer and they got two
• 3:47
  Speaker 4: to four months, I want to hit them very very
• 3:50
  Speaker 4: hard with a very high dose of astes anthon Because
• 3:54
  Speaker 4: there's two methods of absorption. One is the trojan horse,
• 3:58
  Speaker 4: where it's chemically absorbed across the small bowel with the
• 4:02
  Speaker 4: glucosidic ascisantine. The other one is called a concentration gradient,
• 4:07
  Speaker 4: where if I increase the concentration in the small vow
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  Speaker 4: by brute force, it diffuses through all cell membranes and
• 4:16
  Speaker 4: gets into the tissue very very quickly. We see this
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  Speaker 4: with type two diabetics with insulin. Sometimes sugar will accumulate
• 4:27
  Speaker 4: because it can't get into the cell anymore. The receptors
• 4:34
  Speaker 4: are not opening to allow sugar to flow from the
• 4:36
  Speaker 4: bloodstream into the cell. Well, what happens is the body
• 4:40
  Speaker 4: says just keep increasing my blood sugar levels until I
• 4:44
  Speaker 4: can force it through the cell membrane by brute force.
• 4:47
  Speaker 4: And by that time you're six hundred blood sugars to
• 4:51
  Speaker 4: get enough sugar into your cells to keep your cells alive.
• 4:55
  Speaker 4: So that's diabetes. You can't rely on concentration grade, but
• 5:00
  Speaker 4: with the astaxanthin and the glucosidic form, it readily takes off.
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  Speaker 4: So we look at saturating every cell in your body.
• 5:10
  Speaker 4: Every cell in your body is represented by your body weight.
• 5:16
  Speaker 4: The more cells you have, the more you weigh, so
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  Speaker 4: we look at body weight. That's the importance there. And
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  Speaker 4: then if you have to get saturated in a week
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  Speaker 4: for two weeks, will hit you with a very high
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  Speaker 4: dose to really get every cell saturated over a very
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  Speaker 4: short period of time. People who are taking it as
• 5:38
  Speaker 4: a preventative, we drop them down to twenty five or
• 5:42
  Speaker 4: fifty milligrams a day because they have three or four
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  Speaker 4: months to accumulate this inventory of astesantin in all of
• 5:49
  Speaker 4: their cells. So time is not as important to somebody
• 5:53
  Speaker 4: who's not sick than it is to somebody who has
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  Speaker 4: two to four months to live. So that's the things
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  Speaker 4: that our staff looks at our nurses look at is
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  Speaker 4: what is the condition and what is the dose that
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  Speaker 4: we have to we have to hit you with.
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  Speaker 5: So if somebody were interested in looking into taking ast
• 6:16
  Speaker 5: zampen or recommending it for their patients, what is the
• 6:19
  Speaker 5: method do they do? You have to be a practitioner
• 6:22
  Speaker 5: to order this? Can you order it as a patient?
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  Speaker 5: And how can you get a consultation to figure out
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  Speaker 5: the dose if you're not a practitioner.
• 6:31
  Speaker 4: Yeah, you can call in anytime and the number is
• 6:34
  Speaker 4: eight three four seven zero nineteen thirteen. That's eight oh
• 6:39
  Speaker 4: three four seven zero nineteen thirteen and someone will will
• 6:45
  Speaker 4: There's no charge. The whole idea was that this was
• 6:48
  Speaker 4: to be set up primarily as a service function and
• 6:53
  Speaker 4: secondarily to sell the product. If you can get your
• 6:57
  Speaker 4: HSCRP reduced by any other method, feel free to have
• 7:03
  Speaker 4: at it. You know. The whole objective here is people
• 7:08
  Speaker 4: have got to be aware of their diet, their nutrition.
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  Speaker 4: For example, the one thing that freaks out everybody is
• 7:18
  Speaker 4: fruit is a poison. It's probably going to freak you out.
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  Speaker 4: Fructose is a poison and it always has been. Fructose
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  Speaker 4: is the primary cause of high cholesterol. It's not animal
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  Speaker 4: fats never has been. They lied to us in the
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  Speaker 4: food pyramid. Bobby Kennedy uncovered it. I've been promoting it.
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  Speaker 4: I taught biochemistry for three years and I told people
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  Speaker 4: fructose high fructose corn syrup was introduced in nineteen sixty six,
• 7:47
  Speaker 4: and in the early nineteen seventies we saw this huge
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  Speaker 4: increase in cancer and obesity and all these other diseases.
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  Speaker 4: It's fructose. Fructose cannot be used by a single cell
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  Speaker 4: in your body, not one. Glucose sugar is used by
• 8:05
  Speaker 4: every cell in your body. It's the primary energy in
• 8:08
  Speaker 4: the conversion of ADP into ATP that allows us to
• 8:12
  Speaker 4: carry on this conversation. That's where we get the electrical
• 8:14
  Speaker 4: activity from. Fructose is a poison. And when I contacted
• 8:20
  Speaker 4: the NIH and asked them, they confirmed that fructose is
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  Speaker 4: a poison. But that's where we get sweetness from. That's
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  Speaker 4: our primary desire for eating candy is that taste of fructose.
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  Speaker 4: Fructose will kill you. I eat no fruit, none. I
• 8:45
  Speaker 4: haven't for probably. I might have cheated once in a while.
• 8:49
  Speaker 4: I guess periodically, but it's not a main staple at
• 8:54
  Speaker 4: all in anything that I do. So I eat, I'm
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  Speaker 4: giving you an example, every morning for the last forty years,
• 9:01
  Speaker 4: I get up and I have bacon and eggs. Every
• 9:04
  Speaker 4: morning for forty years, my cholesterol is one sixty. My
• 9:09
  Speaker 4: doctor says, I can't believe that you don't have high cholesterol.
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  Speaker 4: I said, you don't understand. Cholesterol doesn't come from animal fats.
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  Speaker 4: It comes from fructose, and we have the entire chemistry.
• 9:21
  Speaker 4: We know how it is converted into triglycerides. They used
• 9:25
  Speaker 4: to call it the cops disease because cops in their
• 9:27
  Speaker 4: bloodwork always had a hard time with their triglyceride levels
• 9:31
  Speaker 4: because they spent too much time in donut shops. That
• 9:33
  Speaker 4: was always the you know the joke. So once the
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  Speaker 4: fructose is converted into triglycerides, and the triglycerides in about
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  Speaker 4: four hundred n nano grams per liter, the liver converts
• 9:50
  Speaker 4: that triglyride into LDL cholesterol, the bad cholesterol. So that's
• 9:57
  Speaker 4: where cholesterol comes from. The when people begin to cut
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  Speaker 4: out their sugar intake, they're fructose because sugar is fifty
• 10:06
  Speaker 4: percent fructose. Sugar only gets its sweetness most of its
• 10:12
  Speaker 4: sweetness from fructose. So the difference between honey and table
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  Speaker 4: sugar table sugar is chemically attached. Get a glucose molecule
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  Speaker 4: and a fructose molecule, and they're chemically bound by an oxygen.
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  Speaker 4: So if I had to look in, I would find
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  Speaker 4: these molecules called sucrose. But fifty percent of its glucose
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  Speaker 4: fifty percent of its fructose. In honey, which everyone thinks
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  Speaker 4: is safer, actually percentage wise has a much higher concentration
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  Speaker 4: of fructose. But they're not. It's not chemically attached. Bees
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  Speaker 4: produce glucose independently as a separate molecule and fructose, so
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  Speaker 4: they're all floating around as independent molecules. But in table sugar,
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  Speaker 4: it processed sugars. We chemically attach them, so it's a
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  Speaker 4: dumbbell shaped molecule. But in honey you actually have more fructose.
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  Speaker 4: It's actually more inflammatory. Honey is more inflammatory than table sugar.
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  Speaker 4: And that's hard for people now to come to grips
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  Speaker 4: with because that's totally counter to what we've been told.
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  Speaker 4: I was asked to calculate, for example, in an apple.
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  Speaker 4: An apple now is poisonous because the fruit of the
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  Speaker 4: apple has large amounts of fructose. It's hybridized, not genetically modified.
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  Speaker 4: It's just hybridized, genetically hybridized. But the antioxidants of an
• 11:44
  Speaker 4: apple are found in the skin and the seeds of
• 11:48
  Speaker 4: the apple. So I said and calculated what size apple
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  Speaker 4: based on the fructose content of the flesh of the
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  Speaker 4: apple versus the skin and the seeds. How big of
• 12:00
  Speaker 4: an apple can I eat? So that I've been totally
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  Speaker 4: in balanced with the antioxidant properties of the apple versus
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  Speaker 4: the fruitcose, so I can still enjoy an apple. And
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  Speaker 4: it turns out to be a little less than an
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  Speaker 4: inchin diameter. No more big fuji apples, no more macintosh,
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  Speaker 4: none of that. It should be a crab apple about
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  Speaker 4: that big around is as big. About one inchin diameter
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  Speaker 4: is as big an apple where you're where it is
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  Speaker 4: not inflammatory and you may get the antioxidant benefits from it.
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  Speaker 4: It used to be an apple a day would keep
• 12:40
  Speaker 4: the doctor away. Now it's an apple a day, we'll
• 12:44
  Speaker 4: bring the doctor to your door. It's a whole different
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  Speaker 4: mess that we've got ourselves in because there's money, like
• 12:52
  Speaker 4: an orange juice. Orange juice is probably the worst any
• 12:55
  Speaker 4: juice because they'll have very high levels of fructose. But
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  Speaker 4: oranges and orange juice and dates and plums, all of
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  Speaker 4: these fruits. Anything that has over four grams of fructose
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  Speaker 4: per hundred grams of fruit should be avoided. So where
• 13:17
  Speaker 4: the fruits that fall out are blueberries, blackberries, those are
• 13:22
  Speaker 4: good fruits. There's enough fructose in those that we can
• 13:25
  Speaker 4: process it because we have to have some cholesterol. We
• 13:28
  Speaker 4: have to have some LDL some HDL because that's the
• 13:33
  Speaker 4: healing component. When we get a lesion from the high
• 13:38
  Speaker 4: concentration of free radicals forms a lesion or a sore
• 13:42
  Speaker 4: on the inside of an artery. Say you're a orda.
• 13:47
  Speaker 4: The first line of repair is for the white blood
• 13:50
  Speaker 4: cells to go in there and fix that wounds and
• 13:53
  Speaker 4: it will form a scab. Well, that scalb is LDL cholesterol.
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  Speaker 4: So when they go in and look at what is
• 13:59
  Speaker 4: causing this plug the scab to form and close off
• 14:03
  Speaker 4: the artery, they see, well, it's cholesterol. It's all cholesterol.
• 14:07
  Speaker 4: That's just the repair mechanism. You're not dealing with why
• 14:11
  Speaker 4: it's there in the first place. Why are the platelets there,
• 14:15
  Speaker 4: Why are the macrophases there, Why is the cholesterol there?
• 14:18
  Speaker 4: It's because you have a lesion caused because you're inflammatory
• 14:22
  Speaker 4: disease state and if you don't get that under control,
• 14:25
  Speaker 4: you're going to die. It's very simple.
• 14:30
  Speaker 5: Wow, doctor Sheppard, you are sure a wealth of information
• 14:34
  Speaker 5: and can really break down all the biochemical pathways and
• 14:39
  Speaker 5: what you're talking about is so important and so nuanced,
• 14:43
  Speaker 5: and that's why the science on it is so controversial,
• 14:46
  Speaker 5: and there's so many different diets out there and so
• 14:48
  Speaker 5: many different viewpoints. And you make a very good case
• 14:52
  Speaker 5: for fructo's. That's something that's been known in like the
• 14:55
  Speaker 5: holistic natural community that this especially when it's an added
• 15:00
  Speaker 5: form in foods.
• 15:01
  Speaker 3: And not in the natural form. You know, there's a
• 15:04
  Speaker 3: lot of factors to consider.
• 15:06
  Speaker 5: So I'll say this, the added sugars and artificial flavors
• 15:12
  Speaker 5: and all of the processed things that turn healthy foods
• 15:16
  Speaker 5: into junk is definitely a no no.
• 15:18
  Speaker 3: I think we can all agree on that.
• 15:20
  Speaker 5: And then from the natural foods, there's definitely some things
• 15:24
  Speaker 5: that are more healthy because of their sugar content or
• 15:26
  Speaker 5: fiber content, or how they support the microbiome and other things.
• 15:30
  Speaker 5: So I really really appreciate you coming on and sharing
• 15:35
  Speaker 5: all your wisdom and about this great product that you invented.
• 15:39
  Speaker 5: And since we're coming to the end of the show here.
• 15:42
  Speaker 5: I want to invite you to do a rapid fire
• 15:44
  Speaker 5: around with me. Okay, all right, So the first question
• 15:48
  Speaker 5: is a little bit on the longer side.
• 15:50
  Speaker 3: But the rest will definitely be quick.
• 15:52
  Speaker 5: So the first question I want to ask you is
• 15:54
  Speaker 5: to reflect on your journey that you spoke about in
• 15:57
  Speaker 5: the beginning, as you having the education and the background
• 16:01
  Speaker 5: in science, and then adding on the divine component, which
• 16:05
  Speaker 5: I did not even know there was such a degree,
• 16:08
  Speaker 5: even though I took theology classes in my university, which
• 16:11
  Speaker 5: was a Catholic university.
• 16:13
  Speaker 3: So there's that intersection.
• 16:15
  Speaker 5: And then you working with plastics initially, right and like
• 16:19
  Speaker 5: cleaning up this sludge, and then you having this sledge
• 16:22
  Speaker 5: in your blood and coming out on the other side
• 16:25
  Speaker 5: and coming to grips with your own fragility essentially, and
• 16:30
  Speaker 5: still having so much more to contribute. So my question is,
• 16:36
  Speaker 5: how did you initially get excited about your work with plastics,
• 16:41
  Speaker 5: and now how did that change with your mission and
• 16:43
  Speaker 5: what's next for your company and to expand this knowledge
• 16:47
  Speaker 5: and awareness of how to cure these diseases.
• 16:50
  Speaker 4: Yeah, there was I was going to write a book
• 16:53
  Speaker 4: once that the human body is nothing more than plastic
• 16:56
  Speaker 4: and soap, bubbles and proteins, for example, or plastics. They
• 17:02
  Speaker 4: are polymers, and that chemistry is what creates our proteins,
• 17:08
  Speaker 4: our enzymes, our dna. Our DNA is a form of
• 17:13
  Speaker 4: plastic or polymers. It's anything that's macro molecular is called
• 17:21
  Speaker 4: a polymer. So the way we would initiate to make,
• 17:27
  Speaker 4: for example, polystyrene, I have a styrene molecule and a
• 17:30
  Speaker 4: styrene molecule. How do you get them to stick together? Well,
• 17:34
  Speaker 4: we added free radicals. We added an initiator, and we
• 17:39
  Speaker 4: used peroxides, so it would open up the double bond
• 17:44
  Speaker 4: and allow the two styrenes to stick together. Now the
• 17:50
  Speaker 4: next styrene molecule would stick to it. So I only
• 17:53
  Speaker 4: had to initiate the reaction. And that's where my free radical.
• 17:57
  Speaker 4: I had entire courses in free radical chemistry, so that's
• 18:00
  Speaker 4: how I knew how free radicals were utilized in the
• 18:05
  Speaker 4: chemical The petrochemical industry extremely important. They're extremely reactive. And
• 18:10
  Speaker 4: then when I got into the biochemistry side, and I going,
• 18:13
  Speaker 4: oh my golly, here's the same free radicals that are
• 18:18
  Speaker 4: involved in cytokine development or in pathway communication cellular communication.
• 18:25
  Speaker 4: They're the same free radicals, they're that reactive and that
• 18:31
  Speaker 4: is what was causing the inflammatory side of this. So
• 18:36
  Speaker 4: some of the courses, the biochemistry courses, and I taught,
• 18:40
  Speaker 4: like I said, taught biochemistry for three years. It is.
• 18:45
  Speaker 4: It was remarkable how an unknowing on my part where
• 18:50
  Speaker 4: all this was going to lead. But what I learned
• 18:53
  Speaker 4: from the polymer industry that chemistry became crucial in the
• 18:58
  Speaker 4: organic chemistry side and then became very important in the
• 19:04
  Speaker 4: biochemical side of what is really going on with human disease.
• 19:10
  Speaker 4: So all this I'm compiling in an hour. Keep in mind,
• 19:13
  Speaker 4: this is fifty years of working through this stuff, trying
• 19:18
  Speaker 4: to figure it out, and not. When I got sick,
• 19:23
  Speaker 4: my motivation fear left me. Nobody's getting out of here alive.
• 19:28
  Speaker 4: We're all going to die. But I died. My life
• 19:35
  Speaker 4: essentially ended, and all the fear of dying left me.
• 19:41
  Speaker 4: There's nothing I fear anymore. I don't fear bankruptcy, I
• 19:46
  Speaker 4: don't fear dying. I don't fear being run over by
• 19:48
  Speaker 4: a mac cruck, I don't fear being shot. I don't
• 19:51
  Speaker 4: fear anything anymore. And once that fear left me, my
• 19:59
  Speaker 4: whole world opened up. All the guardrails came off, and philosophically,
• 20:07
  Speaker 4: I had to come to terms with that, what does
• 20:09
  Speaker 4: that really mean? So I spent a lot of time
• 20:16
  Speaker 4: with understanding the Stoics, Marcus Aurelius, Zeno, Epictetus, Epicurus, Socrates.
• 20:27
  Speaker 4: I wanted to know what did they know before Christ?
• 20:30
  Speaker 4: What did they know? And it turns out because they
• 20:33
  Speaker 4: were professing the same stuff, love your neighbor, justice systems,
• 20:40
  Speaker 4: but they didn't know about the Son of God. They
• 20:42
  Speaker 4: didn't know that. So why did Paul, who was a
• 20:48
  Speaker 4: Jewish guy when during Christ, where did he go? First?
• 20:52
  Speaker 4: He went to Greece. He went to the Stoics because
• 20:55
  Speaker 4: they already knew it. They just didn't have the person
• 20:58
  Speaker 4: to believe in. They believed in nature and man. It
• 21:03
  Speaker 4: took off because for four hundred years, Socrates and Epictetus
• 21:08
  Speaker 4: and Epicurus and Marcus Aurelius, they were all going down
• 21:12
  Speaker 4: this same human existence, how to live your life, but
• 21:16
  Speaker 4: they were basing it on nature. And Paul goes to
• 21:22
  Speaker 4: Greece in Ephesus and Corinth, where the Stoics were, because
• 21:27
  Speaker 4: they were easy to convince. They just they already had
• 21:30
  Speaker 4: the basis. They didn't have the person.
• 21:37
  Speaker 5: Right, Yeah, I think I need to have you back
• 21:40
  Speaker 5: for a whole other episode our weeken talk philosophy. So
• 21:44
  Speaker 5: what's next where they ask to the xanthein and its
• 21:49
  Speaker 5: role and prevention and cure of diseases such as cancer.
• 21:53
  Speaker 4: We were pushing the envelope a little bit, not so
• 21:57
  Speaker 4: much in changing the chemical structure, but we focus primary
• 22:00
  Speaker 4: on the human industry. Now we have animal products, we
• 22:04
  Speaker 4: can stop lactic acid build up, for example, in horses
• 22:08
  Speaker 4: horse racing, that's all that's the limiting factor. We find
• 22:11
  Speaker 4: that when horses race, it's the last few hundred yards.
• 22:16
  Speaker 4: How much lactic acid did that animal build up? Well,
• 22:19
  Speaker 4: we give it to horses and it stops the lactic
• 22:24
  Speaker 4: acid build up. So now the horses are really pushing
• 22:29
  Speaker 4: it very very quickly right now when they don't have
• 22:32
  Speaker 4: lactic acid build up. It stops nosebleeds in racing horses.
• 22:36
  Speaker 4: We give it to dogs with the same diseases cancer arthritis,
• 22:43
  Speaker 4: and it stops. It stops the dermatitis, it stops the
• 22:45
  Speaker 4: skin issues. It's all inflammatory and that was the real key.
• 22:52
  Speaker 4: But now we've got to push it into the animal world.
• 22:55
  Speaker 4: So dogs, cats, horses, that those are the three animals
• 23:01
  Speaker 4: right now that we're focused on.
• 23:04
  Speaker 3: Amazing. So two more questions.
• 23:06
  Speaker 5: One is what's the number one advice you have for
• 23:09
  Speaker 5: people that want to improve their quality of life right now?
• 23:13
  Speaker 4: Measure their HSCRP number one, see where they are, set
• 23:17
  Speaker 4: their baseline. Love it out where they are because we
• 23:20
  Speaker 4: can now treat to that. That's the primary primary indicator.
• 23:27
  Speaker 4: There's said rates. There's some other more expensive blood tests
• 23:29
  Speaker 4: you that you can run, but CRP is sixty dollars
• 23:33
  Speaker 4: and keep it under three milligrams per lead and you
• 23:37
  Speaker 4: are now in control. If you can't get it under
• 23:40
  Speaker 4: three miligrams per leader, take Velasta. It'll knock it within
• 23:43
  Speaker 4: about ten to fourteen days. It'll knock it down.
• 23:48
  Speaker 3: Love it okay?
• 23:49
  Speaker 5: And then finally, what's your fun fact about yourself or
• 23:54
  Speaker 5: hobby that maybe people don't know about.
• 23:57
  Speaker 4: Right now, I'm developing what's ultra capacitors. These replace lithium
• 24:02
  Speaker 4: iron phosphate batteries. They are one tenth the weight and
• 24:07
  Speaker 4: carry the same power. As a matter of fact, I'm
• 24:10
  Speaker 4: leaving this podcast and I'm going to go meet with
• 24:13
  Speaker 4: some folks from California where we can now build these supercapacitors.
• 24:19
  Speaker 4: That can run your house if you lose power for
• 24:22
  Speaker 4: up to thirty days, and they're very very small. No
• 24:26
  Speaker 4: more gasoline power generators, no more natural gas diesel generators.
• 24:32
  Speaker 4: We can now charge these capacitors and they can be
• 24:35
  Speaker 4: part of the structure of your house. But these are supercapacitors.
• 24:39
  Speaker 4: We're putting them in drones right now to reduce the
• 24:42
  Speaker 4: weight in military and civilian drone right now they use
• 24:46
  Speaker 4: the real heavy lithium iron phosphate battery, and now that's
• 24:51
  Speaker 4: about forty percent of the weight of a drone. We're
• 24:54
  Speaker 4: down under four percent of the weight for the same power.
• 24:59
  Speaker 3: Wow.
• 25:00
  Speaker 5: Well, we definitely have to have you back on to
• 25:02
  Speaker 5: talk about that as well. I'm really interested in sustainable
• 25:06
  Speaker 5: and regenerative energy production, so I don't know how that
• 25:09
  Speaker 5: all fits, but I'm sure I can have a conversation.
• 25:12
  Speaker 3: And I'm so so honored that you are on here.
• 25:16
  Speaker 5: You're doing such amazing work, such great innovation, and just
• 25:20
  Speaker 5: being a frontier in a number of fields it sounds like.
• 25:24
  Speaker 5: So I would love for you to tell the audience
• 25:26
  Speaker 5: how they can get in touch with you and learn
• 25:28
  Speaker 5: more about your work.
• 25:30
  Speaker 4: Have them call the ATO three four seven zero nineteen
• 25:36
  Speaker 4: thirteen and I do. It's free consulting, So if somebody
• 25:40
  Speaker 4: is really in need of having then they'll they'll put
• 25:44
  Speaker 4: you on and I'll call you back. They'll put you
• 25:48
  Speaker 4: on a schedule or on a callback list, and then
• 25:52
  Speaker 4: I call back and I try to spend as much
• 25:55
  Speaker 4: time as I can with each person that calls in.
• 25:59
  Speaker 4: Sometimes it's third minutes, sometimes it's two hours. But these
• 26:03
  Speaker 4: people are going through the same fears that I went through,
• 26:10
  Speaker 4: and it changes who you are as a human being
• 26:16
  Speaker 4: when you realize that this is all going to end
• 26:20
  Speaker 4: here on this earth. We are all going to die.
• 26:25
  Speaker 4: That's an absolute. Now all we you don't know when.
• 26:31
  Speaker 4: So all we have control over is the quality of
• 26:35
  Speaker 4: our life, not the quantity. And that's a struggle for people.
• 26:43
  Speaker 4: People want to live longer, but they don't have control
• 26:47
  Speaker 4: over that. You don't want to live longer. You want
• 26:51
  Speaker 4: to live happy. You want to live with a very
• 26:54
  Speaker 4: good quality and experience life. And that was I think
• 27:04
  Speaker 4: that's where my blinders came off, was when I realized
• 27:10
  Speaker 4: I was out of control. I no longer controlled my life.
• 27:16
  Speaker 4: And I mean, we can get into the philosophical discussion
• 27:21
  Speaker 4: of that, but it's every cancer patient goes through this,
• 27:26
  Speaker 4: What does this mean? Why is it happening to me?
• 27:29
  Speaker 4: Does this mean I'm going to die? Does this mean
• 27:32
  Speaker 4: I'm going to have a terrible life? Is going to
• 27:35
  Speaker 4: be painful? Is? And that terminal type thinking is mind
• 27:42
  Speaker 4: shattering and emotionally, it is very difficult for some people to.
• 27:50
  Speaker 6: Deal with and they've got to be able to work
• 27:54
  Speaker 6: through that because all they have is the quality of
• 27:58
  Speaker 6: their life, and nutrition is a huge part of that.
• 28:03
  Speaker 4: Don't be destroying yourself.
• 28:08
  Speaker 5: Yeah, there's a lot of factor's stacked against us collectively unfortunately,
• 28:14
  Speaker 5: but if you don't have your health, you don't have anything.
• 28:16
  Speaker 3: So it's number one.
• 28:18
  Speaker 4: That's right, that's it. We start dying from the day
• 28:20
  Speaker 4: we're born, and it's all about the quality of our life.
• 28:26
  Speaker 4: And hopefully you know in my mind because my faith
• 28:33
  Speaker 4: and what my I was, I was a trained evolutionist.
• 28:38
  Speaker 4: I was trained in science. But the more science I
• 28:42
  Speaker 4: begin to understand at the most minute detail, the more
• 28:49
  Speaker 4: more of a realist I came that this is not random.
• 28:57
  Speaker 4: This is absolutely by does because we had to have space,
• 29:02
  Speaker 4: time and energy at the same time, because without matter,
• 29:07
  Speaker 4: we have no place to put it. If there's no space.
• 29:10
  Speaker 4: You can't evolve in a serial type of an existence.
• 29:16
  Speaker 4: It had to be created instantaneously. And when I saw
• 29:19
  Speaker 4: it in the cilia or on the tail of a
• 29:22
  Speaker 4: sperm cell, there's nine amino acids in the stationary part
• 29:27
  Speaker 4: and in the rotor part of the tail of a
• 29:29
  Speaker 4: sperm cell. It follows an electron. So in these nine
• 29:33
  Speaker 4: proteins around, there's an electron that is fired and the
• 29:40
  Speaker 4: rotor inside cracks. The magnetic field of that electron chases it,
• 29:45
  Speaker 4: and that's what causes the tail of a sperm cell
• 29:48
  Speaker 4: to spin at one hundred thousand revolutions per minute, and
• 29:52
  Speaker 4: that's what drives the sperm cell. If I remove one
• 29:57
  Speaker 4: of those nine proteins, stops. So all nine of those
• 30:03
  Speaker 4: proteins had to be formed instantaneously at the same time.
• 30:09
  Speaker 4: You can't form one and then wait four million years
• 30:12
  Speaker 4: for the next one to form, and the next one
• 30:15
  Speaker 4: and the next one. There will be no life. So
• 30:21
  Speaker 4: what there's no process in the evolutionary chemistry that explains
• 30:26
  Speaker 4: how all nine of those nine would flash into existence
• 30:32
  Speaker 4: at the same moment. Nothing to explain it. So the
• 30:38
  Speaker 4: deeper the knowledge of science, it leads to this moment
• 30:46
  Speaker 4: Eureka moment was you know how great art tho. You
• 30:51
  Speaker 4: start to look around and you start to think science
• 30:55
  Speaker 4: is just a subset of creation. The very first ever
• 31:01
  Speaker 4: conducted and it's where Sir Francis Bacon developed a scientific
• 31:04
  Speaker 4: method from was the Book of Job, when Satan and
• 31:08
  Speaker 4: God created an experiment, and unfortunately Job was the was
• 31:17
  Speaker 4: the object of the experiment, but he developed a problem,
• 31:24
  Speaker 4: developed a hypothesis that Satan thought that he could convert
• 31:29
  Speaker 4: Job away from God, and God said, well, let's try it.
• 31:32
  Speaker 4: Let's do an experiment. You could down and torment Job
• 31:35
  Speaker 4: all you want. Can't kill him, you take everything he has.
• 31:38
  Speaker 4: So the devil goes down, takes everything, takes his family
• 31:42
  Speaker 4: as kids, everything, and Job still doesn't deviate from God.
• 31:49
  Speaker 4: Satan goes back to God and God says, well, he
• 31:53
  Speaker 4: didn't flip Job introduces another experiment, so he goes back
• 31:58
  Speaker 4: and retests, that's exactly what the scientific method requires. So
• 32:05
  Speaker 4: Joe then gives him sores and blisters and takes him
• 32:09
  Speaker 4: pretty close to death, gets tormented by his men. He
• 32:15
  Speaker 4: still doesn't change. Sir Francis Bacon, who was a Christian,
• 32:22
  Speaker 4: got the scientific method from the Book of Job. And
• 32:28
  Speaker 4: science doesn't want to recognize that it all came from
• 32:33
  Speaker 4: the word of God. Interesting dilemma. But the more knowledge
• 32:39
  Speaker 4: we acquire, the more the creation becomes very very clear.
• 32:49
  Speaker 5: Yeah, I love the picture you have in the background
• 32:53
  Speaker 5: also of Einstein and the quantum physics meeting Newtonian physics.
• 32:59
  Speaker 5: Also in do is a lot of interesting questions and
• 33:03
  Speaker 5: what you stated earlier about the precursor of monotheistic religions
• 33:07
  Speaker 5: being following and observing the patterns of nature correctly. And
• 33:12
  Speaker 5: I always think science is so arrogant because it's kind
• 33:15
  Speaker 5: of like the new kid on the block, and the
• 33:19
  Speaker 5: method is great, right, and you get to test and retest,
• 33:22
  Speaker 5: and you have to continue having an open mind for
• 33:25
  Speaker 5: new information to come in. And when you accept something
• 33:29
  Speaker 5: as unshakable foundational fact, sometimes it's so hard right to
• 33:35
  Speaker 5: accept new information and be open to new ways of
• 33:38
  Speaker 5: doing things. And that's what we see in the scientific
• 33:40
  Speaker 5: and the medical community and you.
• 33:42
  Speaker 4: But we have somebody existence, But we have some Charlatans who,
• 33:47
  Speaker 4: for example, there's never been an experiment performed to ever
• 33:51
  Speaker 4: show evolution, not one. It's always it's only speculative from
• 33:56
  Speaker 4: fossil records or whatever. But that's speculation. Just see an
• 34:00
  Speaker 4: animal and they see one similar. Okay, if someone looked
• 34:06
  Speaker 4: at me and looked at you, they would say, oh
• 34:09
  Speaker 4: my golly, they are very close. They look they have
• 34:11
  Speaker 4: a head, they have two arms, two feet. But we're
• 34:14
  Speaker 4: not related. We're different. Now, I wrote a paper linking
• 34:21
  Speaker 4: Newtonian and relativistic in the proving of what is at
• 34:26
  Speaker 4: the center of a black hole? Is it a singularity?
• 34:28
  Speaker 4: Because the singularity falls apart. And what I had shown
• 34:32
  Speaker 4: is that the physics points to a black hole is
• 34:37
  Speaker 4: a spinning cooroid, a spinning gyroscope. There's nothing at the
• 34:42
  Speaker 4: center of a black hole. The event horizon is spinning
• 34:46
  Speaker 4: at the speed of light, and as it moves out,
• 34:50
  Speaker 4: it spins faster, but the maximum speeds the speed of light.
• 34:53
  Speaker 4: And if it's moving in that angle, we can't see
• 34:56
  Speaker 4: past it. But there's nothing at the center of a
• 34:59
  Speaker 4: black hole. If you entered a bible star entered a
• 35:02
  Speaker 4: black hole, it would be accelerated and turned from matter
• 35:06
  Speaker 4: into energy under e equals mc square, and at that
• 35:12
  Speaker 4: point it becomes plasma spinning at the speed of light,
• 35:16
  Speaker 4: it becomes pure energy. There's a whole other story and
• 35:20
  Speaker 4: there's some papers there, but I'm going to have to schedudle. Also, Marina,
• 35:26
  Speaker 4: I've enjoyed this. Thank you so much.
• 35:30
  Speaker 5: Thank you so much, doctor Shepherd. There is a very
• 35:33
  Speaker 5: interesting show I listened to by Adam Apollo and he
• 35:38
  Speaker 5: talks about how the protons are a representation of a
• 35:43
  Speaker 5: black hole that have the elections spinning around us. So
• 35:47
  Speaker 5: there's a whole lot to this micro and macro conversation
• 35:51
  Speaker 5: and the meaning of life. And it's at the end
• 35:54
  Speaker 5: of the day, still a miracle, like you said, that
• 35:56
  Speaker 5: we are all here and we get to have this experience.
• 36:00
  Speaker 3: Thank you so much again for coming on. And I
• 36:02
  Speaker 3: think I'll probably.
• 36:02
  Speaker 5: Split this into two episodes, so I'll let you know
• 36:05
  Speaker 5: when this airs.
• 36:06
  Speaker 3: And thank you to see you and follow your work.
• 36:09
  Speaker 4: Thank you, great day. Thank you so much, Marina, Take care,
• 36:13
  Speaker 4: bye moha bye bye.
• 36:14
  Speaker 2: Thank you for tuning in to another episode of the
• 36:17
  Speaker 2: Holistic Pharmacy Podcast and I truly hope you enjoyed the show.
• 36:22
  Speaker 2: If you learned something useful in this episode, please consider
• 36:25
  Speaker 2: giving the show a five star rating and a sincere
• 36:28
  Speaker 2: review so that more people can find it or share
• 36:31
  Speaker 2: it with a friend directly to get in touch with me.
• 36:34
  Speaker 2: You can find me on social media by looking up
• 36:37
  Speaker 2: my name Marina books Of or email me at Marina
• 36:41
  Speaker 2: at rawfork dot com. Take good care and see you soon.