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TREATING AUTISM EFFECTIVELY??? · Your Daily Dose

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  Speaker 1: Everybody. It is may going into a Memorial Day weekend.
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  Speaker 1: Hopefully have some good plans. Today. We're to talk a
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  Speaker 1: little bit about autism, some new information that came out
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  Speaker 1: about autism, and we'll dive into this just a bit
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  Speaker 1: so they can get some basic information. So stay tended, hi,
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  Speaker 1: and welcome to Health Talk with Dr Lynn. Your daily dose.
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  Speaker 1: The information you hear on the show is informational. It's
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  Speaker 1: not meant to diagnose or treat any disease. Any kind
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  Speaker 1: of complete health conditions must be checked out and discussed
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  Speaker 1: with your primary care physicians. Get ready for the good
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  Speaker 1: news of the day. God bless you, folks, and here
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  Speaker 1: is Doctor Lynn. Our daily devotional today comes from the
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  Speaker 1: Book of One Peter, Chapter five. When things don't go
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  Speaker 1: as you would like, accept the situation immediately. If you
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  Speaker 1: indulge in feelings of regret, they can easily spill over
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  Speaker 1: into a line everything. Remember that I am sovereign over
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  Speaker 1: your circumstances, and humble yourself under my mighty hand. Rejoice
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  Speaker 1: in what I'm doing in your life, even though it's
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  Speaker 1: beyond your understanding. I am the Way, the Truth, and
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  Speaker 1: the life in me. You have everything you need both
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  Speaker 1: for this life or for the life yet to come.
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  Speaker 1: Don't let the impact of the world shatter your thinking
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  Speaker 1: or draw you away from focusing on me. The ultimate
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  Speaker 1: challenge is to keep fixing your eyes on me, no
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  Speaker 1: matter what's going on around you. When I am central
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  Speaker 1: in your thinking, you're able to review circumstances from my perspective. Ooh,
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  Speaker 1: good stuff, Stay tuned m HM seven two seven, nine
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  Speaker 1: one six four two one five. For decades, autism has
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  Speaker 1: been understood as a condition of brain wiring. Whoever, a
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  Speaker 1: growing body of research points to more fundamental problems. The
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  Speaker 1: brain may simply not be getting enough energy to develop
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  Speaker 1: the way it should. Brain is not getting enough energy.
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  Speaker 1: Up to eighty percent of children with autism show signs
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  Speaker 1: of mitochondrial dysfunction. The little power packs and every cell
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  Speaker 1: in our body, specifically in the brain, aren't making enough
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  Speaker 1: energy to really support. In mitochondrial dysfunction, the mitochondria, the
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  Speaker 1: power plants inside your cells, cannot produce enough energy for
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  Speaker 1: the body, which can create a significant problem for a
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  Speaker 1: child's development. The link between mitochondrial disfunction and autism was
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  Speaker 1: first proposed in nineteen eighty five, when clinicians reported seeing
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  Speaker 1: four autistic patients who also had lactic acidosis built of
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  Speaker 1: galactate in the body, which occurs when it cannot efficiently
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  Speaker 1: create energy, suggesting that mitochondria health may have been impaired.
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  Speaker 1: Since nineteen eighty five, bigger and more elaborate studies have
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  Speaker 1: only shown that this problem may be more prevalent in
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  Speaker 1: autism than previously thought. The brain expends twenty percent of
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  Speaker 1: the body's total energy, making it uniquely vulnerable when mitochondriactivity
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  Speaker 1: is impaired. Animal studies have shown that mice with dysfunctional
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  Speaker 1: mitochondria develop autism like symptoms with or without apparent brain changes.
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  Speaker 1: It's still unclear if mitochondrial dysfunction is a cause or
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  Speaker 1: a feature of autism. Many of the genetic and environmental
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  Speaker 1: factors that contribute to autism also impair mitochondrial health. Genetically,
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  Speaker 1: genes that drive mitochondrial dysfunction have been directly linked to autism.
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  Speaker 1: At the environmental level, exposure to early antibiotics, infections, air pollution,
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  Speaker 1: nutritional deficiencies, autoimmune diseases, and I'm going to put in
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  Speaker 1: their vaccines, all of which can raise the risk of
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  Speaker 1: developing both conditions. According to neurologists Rich fry. The mitochondria
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  Speaker 1: very very sensitive to environmental changes, which is why the
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  Speaker 1: vaccine issue has been big. His research suggests that some
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  Speaker 1: of the stress can start pre natally and remain after birth.
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  Speaker 1: Children with autism also have higher levels of oxidative stress.
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  Speaker 1: Glued tothion, which neutralizes oxidative stress, needs energy from the
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  Speaker 1: mitochondria just to be made, so if the mitochondria aren't working,
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  Speaker 1: you're going to make less glutithione and not be able
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  Speaker 1: to control that oxidative stress. Information is another common feature.
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  Speaker 1: Inflammation is another common feature of autism, and mitochondrial dysfunction
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  Speaker 1: can drive that too. Anti inflammatory cells require mitochondria to
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  Speaker 1: be activated, whereas inflammatory cells don't. Among children with autism,
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  Speaker 1: about five percent of classic mitochondrial disease caused by genetic changes.
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  Speaker 1: Most children with autism, however, have mitochondrialis function not traceable
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  Speaker 1: to a gene. Children of autism and mitochondri dysfunction often
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  Speaker 1: show condural related symptoms such as fatigue, developmental delay, muscle weakness,
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  Speaker 1: and nerve pain. One unique factor is that the mitochondrial
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  Speaker 1: disfunctions often link to regression type autism, where children appear
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  Speaker 1: to be developing typically but in early childhood start regressing
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  Speaker 1: and losing their skills. This is in line with medical
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  Speaker 1: understanding of mitochondrial dysfunction. Most patients may not show symptoms
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  Speaker 1: for years before they suddenly manifest The most comprehensive research
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  Speaker 1: comes from Robert Burrier in colleagues who analyze blood samples
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  Speaker 1: from more than a thousand young children comparing metabolic markers
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  Speaker 1: to those in autism to those without. Their study, also
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  Speaker 1: known as the Children's Autism Metabolome Project, identified seven different
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  Speaker 1: metabolic profiles and children with autism, all of which showed
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  Speaker 1: that normal pathways that may be implicated in mitochondrial dysfunction.
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  Speaker 1: Supplements are the most common treatments for mitochondrial dysfunction, in
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  Speaker 1: part because the condition affects every person differently, and supplements
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  Speaker 1: provide a broader treatment when targeting drugs are often unavailable.
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  Speaker 1: There is no magic pill, even for genetically confirmed mitochondrial disease.
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  Speaker 1: Wily have two FDA treatments, so we have very little
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  Speaker 1: definitive treatment for these patients, said doctor Francis Kendall. Common
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  Speaker 1: treatment is a mitochondrial cocktail. The contents vary depending on
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  Speaker 1: the child's individual needs, but common ingredients include coins, m
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  Speaker 1: Q ten, levocarnatine, and the alphalopoic acid. All three of
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  Speaker 1: these compounds are naturally produced in the body and play
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  Speaker 1: an important role in normal mitochondrial function. One study administered
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  Speaker 1: these three supplements and found that children with autism generally
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  Speaker 1: improved at least one metabolic health marker. Clinical trials of
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  Speaker 1: levocarnatine and amino acid derivative used to treat carnatine deficiencies
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  Speaker 1: in co Q ten individually, who showed improvements in behavior
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  Speaker 1: and communication. A single case study showed that evocarnatine supplementations
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  Speaker 1: stopped regression in a four year old boy. Other nutrients
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  Speaker 1: included in the cocktails, including the B group vitamins that
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  Speaker 1: are directly involved in energy production in the mitochondria, while
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  Speaker 1: B six helps prevent oxidative stress and supports energy production.
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  Speaker 1: Clinical trials in which B six was supplemented with magnesium
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  Speaker 1: showed improvements in behavior and language. Children with autism often
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  Speaker 1: have low levels of vitamin B nine and B twelve.
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  Speaker 1: These two nutrients help to maintain mitochondrial DNA creatine, which
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  Speaker 1: improves muscle functions. Cellular energies sometimes also included keutogenic diets
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  Speaker 1: have shown promise. While established clinical practice exist to diagnose
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  Speaker 1: mitochondrial dysfunction, they are not routinely used in clinics determine
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  Speaker 1: whether it's playing a role. Clinical research into autism diagnostic
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  Speaker 1: biomorkers is both expensive and time consuming. Doctor Burrier Hope
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  Speaker 1: holds a PhD in biochemistry and it's the principal investigator
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  Speaker 1: in the Children's Autism Metabalone project. However, this research needs
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  Speaker 1: to be verified. It's a little unfortunate. Biomarker research is
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  Speaker 1: currently great academic research. However, from a commercial perspective, diagnostics
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  Speaker 1: have little value. If there's no targeted therapeutics, they don't
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  Speaker 1: have a drug for it. The mitochondri dysfunction is a
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  Speaker 1: fairly niche area of research for I Hope, so it'll
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  Speaker 1: be growing interest in the future for better treatments for
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  Speaker 1: children and family. You want more information on that, you
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  Speaker 1: want to put together a protocol, give a call seven
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  Speaker 1: two seven nine one six four two one five great information.
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  Speaker 1: Please share this If you have any friends that are
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  Speaker 1: expecting or have a child that may be there is
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  Speaker 1: a fear factor of autism, or they have another child
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  Speaker 1: with autism and they have another child now so again
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  Speaker 1: we can check that out and put together a protocol